Future Perspectives on Pathogenesis of Lupus Nephritis: Facts, Problems, and Potential Causal Therapy Modalities

Autor: Rekvig, Ole P., Thiyagarajan, Dhivya, Pedersen, Hege L., Horvei, Kjersti D., Seredkina, Natalya

Publikováno v: In The American Journal of Pathology November 2016 186(11):2772-2782

Impact of the Tumor Necrosis Factor Receptor-Associated Protein 1 (Trap1) on Renal DNaseI Shutdown and on Progression of Murine and Human Lupus Nephritis

Autor: Fismen, Silje, Thiyagarajan, Dhivya, Seredkina, Natalya, Nielsen, Henrik, Jacobsen, Søren, Elung-Jensen, Thomas, Kamper, Anne-Lise, Johansen, Steinar D., Mortensen, Elin S., Rekvig, Ole P.

Publikováno v: In The American Journal of Pathology March 2013 182(3):688-700

Lupus nephritis: low urinary DNase I levels reflect loss of renal DNase I and may be utilized as a biomarker of disease progression

Autor: Pedersen, Hege Lynum, Horvei, Kjersti Daae, Thiyagarajan, Dhivya, Norby, Gudrun, Seredkina, Natalya, Moroni, Gabriella, Eilertsen, Gro Østli, Holdaas, Hallvard, Strøm, Erik Heyerdahl, Bakland, Gunnstein, Meroni, Pier Luigi, Rekvig, Ole Petter

Source at https://doi.org/10.1002/cjp2.99. Licensed CC BY-NC-ND 4.0. Renal DNase I is lost in advanced stages of lupus nephritis. Here, we determined if loss of renal DNase I reflects a concurrent loss of urinary DNase I, and whether absence of urina

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=dedup_wf_001::d38d6a71a96ee582f3f458d8b804fd03
https://hdl.handle.net/10037/14016

Lupus nephritis progression in FcγRIIB-/-yaa mice is associated with early development of glomerular electron dense deposits and loss of renal DNase I in severe disease.

Autor: Horvei, Kjersti Daae, Pedersen, Hege Lynum, Fismen, Silje, Thiyagarajan, Dhivya, Schneider, Andrea, Rekvig, Ole Petter, Winkler, Thomas, Seredkina, Natalya

Publikováno v: Horvei, K. D., Pedersen, H. L., Fismen, S., Thiyagarajan, D., Schneider, A., Rekvig, O. P. ... & Seredkina, N. (2017) Lupus nephritis progression in FcγRIIB-/-yaa mice is associated with early development of glomerular electron dense deposits and loss of renal DNase I in severe disease. PLoS ONE 12(11): e0188863.

Externí odkaz: https://hdl.handle.net/10037/12181

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