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pro vyhledávání: '"Seila Fernandez-Fernandez"'
Autor:
Seila Fernandez-Fernandez, Veronica Bobo-Jimenez, Raquel Requejo-Aguilar, Silvia Gonzalez-Fernandez, Monica Resch, Monica Carabias-Carrasco, Joaquim Ros, Angeles Almeida, Juan P. Bolaños
Publikováno v:
Redox Biology, Vol 19, Iss , Pp 52-61 (2018)
Loss of brain glutathione has been associated with cognitive decline and neuronal death during aging and neurodegenerative diseases. However, whether decreased glutathione precedes or follows neuronal dysfunction has not been unambiguously elucidated
Externí odkaz:
https://doaj.org/article/1a2bfe24eb2e4c2aa575d43ec958f2d2
Autor:
Juan P. Bolaños, Monica Carabias-Carrasco, Angeles Almeida, Silvia Gonzalez-Fernandez, Monica Resch, Joaquim Ros, Seila Fernandez-Fernandez, Raquel Requejo-Aguilar, Veronica Bobo-Jimenez
Publikováno v:
Repositorio Abierto de la UdL
Universitad de Lleida
Redox Biology, Vol 19, Iss, Pp 52-61 (2018)
Digital.CSIC. Repositorio Institucional del CSIC
instname
Recercat. Dipósit de la Recerca de Catalunya
Redox Biology
Universitad de Lleida
Redox Biology, Vol 19, Iss, Pp 52-61 (2018)
Digital.CSIC. Repositorio Institucional del CSIC
instname
Recercat. Dipósit de la Recerca de Catalunya
Redox Biology
Loss of brain glutathione has been associated with cognitive decline and neuronal death during aging and neurodegenerative diseases. However, whether decreased glutathione precedes or follows neuronal dysfunction has not been unambiguously elucidated
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f877997b5ac2102a2d526f9f98fb08a7
http://hdl.handle.net/10459.1/64860
http://hdl.handle.net/10459.1/64860
Publikováno v:
Biochemical Journal. 443:3-11
Oxidative and nitrosative stress underlie the pathogenesis of a broad range of human diseases, in particular neurodegenerative disorders. Within the brain, neurons are the cells most vulnerable to excess reactive oxygen and nitrogen species; their su
Autor:
Angeles Almeida, Seila Fernandez-Fernandez, Elena Chiricozzi, Juan P. Bolaños, Vincenza Nardicchi, Gianfrancesco Goracci
Publikováno v:
Journal of Neurochemistry. 112:1574-1583
J. Neurochem. (2010) 112, 1574–1583. Abstract Phospholipases A2 (PLA2) participate in neuronal death signalling pathways because of their ability to release lipid mediators, although the contribution of each isoform and mechanism of neurotoxicity a
Autor:
Juan P. Bolaños, Maria Delgado-Esteban, Angeles Almeida, Angel Herrero-Mendez, Seila Fernandez-Fernandez
Publikováno v:
Biochimica et Biophysica Acta (BBA) - Bioenergetics. 1777:789-793
Besides its essential role at regulating neural functions through cyclic GMP, nitric oxide is emerging as an endogenous physiological modulator of energy conservation for the brain. Thus, nitric oxide inhibits cytochrome c oxidase activity in neurone
Publikováno v:
Biochemical Society Transactions. 35:1224-1227
NO (nitric oxide) participates in a considerable number of physiological functions. At the biochemical level, most of its actions can be ascribed to its ability to bind, and activate, soluble guanylate cyclase. However, mounting evidence now strongly
Publikováno v:
Brain Energy Metabolism ISBN: 9781493910588
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::ce53a1e9688b274282120412a1283a31
https://doi.org/10.1007/978-1-4939-1059-5_8
https://doi.org/10.1007/978-1-4939-1059-5_8
γ-Glutamylcysteine detoxifies reactive oxygen species by acting as glutathione peroxidase-1 cofactor
Autor:
Juan P. Bolaños, Seila Fernandez-Fernandez, Veronica Bobo-Jimenez, Angeles Almeida, Juan Sastre, Ruben Quintana-Cabrera, Javier Escobar
Publikováno v:
Nature Communications
Digital.CSIC. Repositorio Institucional del CSIC
instname
Digital.CSIC. Repositorio Institucional del CSIC
instname
This work is licensed under a Creative Commons Attribution-NonCommercial-No Derivative Works 3.0 Unported License.
Reactive oxygen species regulate redox-signaling processes, but in excess they can cause cell damage, hence underlying the aetiolo
Reactive oxygen species regulate redox-signaling processes, but in excess they can cause cell damage, hence underlying the aetiolo
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::149d18bac5c06c36be1aa9cbd9e78a7a
http://hdl.handle.net/10261/116054
http://hdl.handle.net/10261/116054