Zobrazeno 1 - 10
of 29
pro vyhledávání: '"Sedef Tunaoğlu"'
Publikováno v:
Turkish Journal of Thoracic and Cardiovascular Surgery. 31:145-148
Treprostinil was approved by the United States Food and Drug Administration for use in the treatment of pulmonary arterial hypertension in 2002. Intravenous or subcutaneous treprostinil is used in pulmonary arterial hypertension patients in the funct
Autor:
Tugba Bedir Demirdag, Kivilcim Gucuyener, A. Sebnem Soysal, Sezen Guntekin Ergun, Zeynep Ozturk, Mehmet Ali Ergun, Sedef Tunaoğlu
Publikováno v:
Cardiology in the Young. :1-5
Studies have demonstrated an association between CHD and neurodevelopmental delay. This delay is associated with many factors like reduced blood flow and oxygen, cardiac catheterisations, and genetic factors. Apo E gene polymorphism is one of these g
Autor:
SABİRE GÖKALP, FATMA SEDEF TUNAOĞLU, FATMA CANBEYLİ, SEMİHA TERLEMEZ, VİLDAN ATASAYAN, LATİFE ARZU ARAL, SERDAR KULA, AYŞE DENİZ OĞUZ
Publikováno v:
Turkish Journal of Medical Sciences. 52:1900-1905
Autor:
Pelin Asfuroglu, Deniz Oğuz, Tugba Sismanlar Eyuboglu, Ayse Tana Aslan, Zeynep Reyhan Onay, Sedef Tunaoğlu, Tugba Ramasli Gursoy, Semiha Tokgoz, Serdar Kula, Fatma Incedere
Publikováno v:
Paediatric bronchology.
Objective: Pulmonary hypertension (PH) is a rare condition associated with diverse cardiac, pulmonary, and systemic diseases in childhood. We aimed to detect the frequency of PH due to pulmonary diseases and clinical features of them in a pediatric p
Autor:
Ayse Tana Aslan, Oznur Boyunaga, Cigdem Oztunali, Şişmanlar Eyüboğlu T, Sedef Tunaoğlu, Serdar Kula, Betül Emine Derinkuyu, Ayşe Deniz Oğuz
Publikováno v:
Volume: 47, Issue: 5 1384-1392
Turkish Journal of Medical Sciences
Turkish Journal of Medical Sciences
Background/aim: Airway compression (AC) by vascular structures is an important complication of congenital heart disease (CHD) that often goes unrecognized. It is not easy to identify whether CHD patients require additional invasive examinations or no
Autor:
Hatice Pasaoglu, Gokcen Oz-Tuncer, Sedef Tunaoğlu, Serdar Kula, Deniz Oğuz, Ayhan Pektaş, Erman Cilsal, Rana Olguntürk, Ozge Tugce Pasaoglu
Publikováno v:
Cardiology in the Young. 27:255-260
ObjectiveThe present study aims to identify the role of inflammatory markers such as C-reactive protein, interleukin-6, and fractalkine in CHD-associated pulmonary hypertension in children.MethodsThis is a prospective review of 37 children with CHD-r
Autor:
Fatma Rana Olguntürk, Ayhan Pektaş, Serdar Kula, Fatma Sedef Tunaoğlu, Ayşe Deniz Oğuz, Erman Cilsal
Publikováno v:
Volume: 47, Issue: 6 1854-1860
Turkish Journal of Medical Sciences
Turkish Journal of Medical Sciences
Background/aim: Endothelial dysfunction, tissue damage, inflammation, and microthrombosis are involved in the pathogenesis of pulmonary hypertension (PH), which may be present as a complication of congenital heart diseases. This study aims to identif
Autor:
Berna Şaylan, Sedef Tunaoğlu, Rana Olguntürk, Deniz Oğuz, Serdar Kula, Ayhan Çevik, Ayhan Pektaş
Publikováno v:
Congenital Heart Disease. 8:527-534
Objective The aim of this study is to determine the relationship between the cardiac catheterization findings and pulsed-wave (PW) Doppler and Doppler tissue imaging (DTI) in pulmonary arterial hypertension patients with congenital heart disease with
Autor:
Ayse Tana Aslan, Deniz Oğuz, Oznur Boyunaga, Serdar Kula, Betül Emine Derinkuyu, Tugba Sismanlar, Sedef Tunaoğlu
Introduction: Congenital heart diseases may cause respiratory symptoms due to increased or decreased pulmonary flow. Method: Congenital heart disease patients with persistent respiratory symptoms were reviewed. Results: 251 patients with congenital h
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::88c791651d3bdc83654f8e194ef36f9b
https://avesis.gazi.edu.tr/publication/details/4aa80831-70b8-4c17-b548-4be27d444622/oai
https://avesis.gazi.edu.tr/publication/details/4aa80831-70b8-4c17-b548-4be27d444622/oai
Publikováno v:
International Journal of Angiology. 11:83-85
Turner Syndrome is a genetic disease with a frequency of 1 in every 1500–2500 live-born female babies. In this report, a 2 years 10 months old patient who had the genotype of 46,XOi (Xq) will be introduced, although her phenotype and cardiovascular