Zobrazeno 1 - 10
of 16
pro vyhledávání: '"Sebastian Prager"'
Autor:
Felix Brehmer, Ivo Bendix, Sebastian Prager, Yohan van de Looij, Barbara S Reinboth, Julia Zimmermanns, Gerald W Schlager, Daniela Brait, Marco Sifringer, Stefanie Endesfelder, Stéphane Sizonenko, Carina Mallard, Christoph Bührer, Ursula Felderhoff-Mueser, Bettina Gerstner
Publikováno v:
PLoS ONE, Vol 7, Iss 11, p e49023 (2012)
Intrauterine infection and inflammation are major reasons for preterm birth. The switch from placenta-mediated to lung-mediated oxygen supply during birth is associated with a sudden rise of tissue oxygen tension that amounts to relative hyperoxia in
Externí odkaz:
https://doaj.org/article/89b94f4db86a4ba0a396168cfd235611
Autor:
Gunther Wennemuth, Pawel Mach, Tom Moore, Rainer Kimmig, Alexandra Gellhaus, Sebastian Prager, Angela Köninger, Bernhard B. Singer
Problem CEACAM1 and CEACAM6 belong to the carcinoembryonic antigen (CEA) family and may play an immune-modulatory role during pregnancy. The aim of the study was to determine the blood serum levels of soluble CEACAM1 and CEACAM6 over the course of pr
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::681f17f77ea351684dddb75d738c0c5e
https://www.scopus.com/inward/record.url?partnerID=HzOxMe3b&origin=inward&scp=85020257554
https://www.scopus.com/inward/record.url?partnerID=HzOxMe3b&origin=inward&scp=85020257554
Autor:
Frederik Bertling, Ivo Bendix, Ursula Felderhoff-Mueser, Matthias Keller, Karla Drommelschmidt, Sebastian Prager, H.G. Wisniewski
Inflammation is an important factor contributing to developmental brain injury in preterm infants. Although tumor necrosis factor-inducible gene 6 protein (TSG-6) has immunomodulatory effects in several inflammatory conditions of adult animals, nothi
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4b025f700eb7ac3aa87ba4c0cef16936
https://www.ncbi.nlm.nih.gov/pubmed/26953231
https://www.ncbi.nlm.nih.gov/pubmed/26953231
Publikováno v:
Monatsschrift Kinderheilkunde. 153:264-272
Beim Silver-Russell-Syndrom (SRS) handelt es sich um ein meist sporadisch auftretendes Kleinwuchssyndrom mit charakteristischen Dysmorphien. Bei etwa 7–10% der Patienten liegt eine materne uniparentale Disomie von Chromosom 7 (matUPD7) vor. Auch me
Autor:
Jürgen Tomiuk, Sebastian Prager, E. Meyer, Caitriona Obermann, Hartmut A. Wollmann, Thomas Eggermann
Publikováno v:
Molecular Genetics and Metabolism. 82:246-250
Silver-Russell syndrome (SRS) is a heterogeneous syndrome with evidence for a substantial role of genetic factors in its etiology. Apart from other specific clinical features, severe intrauterine and postnatal growth retardation are the dominant char
Autor:
Sebastian Prager, Katja Eggermann, Hartmut A. Wollmann, Miriam Mavany, Susanne Mergenthaler, Michael B. Ranke, Thomas Eggermann
Publikováno v:
Genetic Testing. 7:259-263
Silver-Russell syndrome (SRS) is a syndrome of severe pre- and postnatal growth retardation and typical dysmorphic features. Rare chromosomal aberrations have been reported in SRS; among these are two balanced translocations involving 17q24-q25. Rece
Autor:
Frederik Bertling, Matthias Keller, Ursula Felderhoff-Mueser, Süleyman Ergün, Ivo Bendix, Bernhard B. Singer, Gerald W. Schlager, Burcin Ceylan, Sebastian Prager
Publikováno v:
Developmental neuroscience. 35(2-3)
CEACAM1 is the founder molecule of the family of ‘carcinoembryonic antigen-related cell adhesion molecules' and part of the immunoglobulin superfamily. Due to its role as a coreceptor to many other receptors (e.g. Toll-like receptor 2, Toll-like re
Autor:
Stefanie Endesfelder, Gerald W. Schlager, Ivo Bendix, C Börner, Marco Sifringer, Matthias Keller, S Holifanjaniaina, Alexander Kalb, C von Haefen, Sebastian Prager, Etienne Jacotot, Ursula Felderhoff-Mueser
Publikováno v:
Cell Death and Disease
Cell Death and Disease, Nature Publishing Group, 2012, 3, pp.e250. ⟨10.1038/cddis.2011.133⟩
Cell Death and Disease, 2012, 3, pp.e250. ⟨10.1038/cddis.2011.133⟩
Cell Death & Disease
Cell Death and Disease, Nature Publishing Group, 2012, 3, pp.e250. ⟨10.1038/cddis.2011.133⟩
Cell Death and Disease, 2012, 3, pp.e250. ⟨10.1038/cddis.2011.133⟩
Cell Death & Disease
International audience; Within the last decade, it became clear that oxygen contributes to the pathogenesis of neonatal brain damage, leading to neurocognitive impairment of prematurely born infants in later life. Recently, we have identified a criti
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::656166635252c4e9b69d12408033d43a
http://hdl.handle.net/10044/1/18028
http://hdl.handle.net/10044/1/18028
Autor:
Daniela Brait, Stéphane Sizonenko, Yohan van de Looij, Felix Brehmer, Bettina Gerstner, Barbara S. Reinboth, Gerald W. Schlager, Ivo Bendix, Marco Sifringer, Julia Zimmermanns, Stefanie Endesfelder, Carina Mallard, Ursula Felderhoff-Mueser, Sebastian Prager, Christoph Bührer
Publikováno v:
PLoS ONE, Vol 7, Iss 11, p e49023 (2012)
PLOS ONE, Vol. 7, No 11 (2012) P. e49023
PLoS ONE
PLOS ONE, Vol. 7, No 11 (2012) P. e49023
PLoS ONE
Intrauterine infection and inflammation are major reasons for preterm birth. The switch from placenta-mediated to lung-mediated oxygen supply during birth is associated with a sudden rise of tissue oxygen tension that amounts to relative hyperoxia in
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::72e341f3817f1f94fec65f1a57294745
https://duepublico.uni-due.de/servlets/DocumentServlet?id=47078
https://duepublico.uni-due.de/servlets/DocumentServlet?id=47078