Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Sean Post"'
Autor:
Pedro Aguilar Garrido, Maria Velasco Estevez, Miguel Ángel Navarro Aguadero, María Hernández Sánchez, Prerna Malaney, Marisa Hornbaker, Xiaorui Zhang, Marta Ibañez Navarro, Alejandra Ortiz-Ruiz, Álvaro Otero-Sobrino, Diego Megías, Manuel Pérez, Jesús Gómez, Gadea Mata, Orlando Dominguez, Osvaldo Grana, Eduardo Caleiras, Marta Isasa, Paloma Jimena de Andrés, Sandra Rodriguez, Raúl Torres, Oleksandra Sirozh, Vanesa Lafarga, Joaquín Martínez-Lopez, Sean Post, Miguel Gallardo
Publikováno v:
HemaSphere, Vol 7, p e8819188 (2023)
Externí odkaz:
https://doaj.org/article/31825dcb3d8a4b67a90a4ac36218cf8e
Autor:
Marisa Hornbaker, Miguel Gallardo, Xiaorui Zhang, Huaxian Ma, Peter Hu, Stephen Kornblau, Carlos Bueso-Ramos, Sean Post
Publikováno v:
Journal of Clinical and Translational Science, Vol 1, Pp 58-58 (2017)
OBJECTIVES/SPECIFIC AIMS: Acute myeloid leukemia (AML) is a devastating hematologic malignancy wherein 20% of circulating white blood cells harboring markers of immature stem cells in conjunction with positive myeloperoxidase staining and blast-appea
Externí odkaz:
https://doaj.org/article/0c1158a4594e4fca9aa607a9f1e905d1
Autor:
Bofei Wang, Christopher Ly, Fatima Zahra Jelloul, Enes Dasdemir, Guilin Tang, Sonali Jindal, Yulong Chen, Sreyashi Basu, Poonam Desai, Pamella Borges, Preethi Gunaratne, Natalia Baran, Qing Deng, Dapeng Hao, Sean Post, Michael Green Green, Marina Konopleva, Andy Futreal, Padmanee Sharma, Hussein A. Abbas
Publikováno v:
Cancer Research. 83:5927-5927
Acute Myeloid Leukemia (AML) is a heterogenous disease characterized by immature blasts at different states of differentiation resulting in marked intra-tumor heterogeneity (ITH). Compared to patients with diploid cytogenetics, deletions in chromosom
Autor:
Ivo Veletic, Taghi Manshouri, Ping Li, C. Cameron Yin, Sean Post, Srdan Verstovsek, Zeev Estrov
Publikováno v:
Clinical Lymphoma Myeloma and Leukemia. 21:S232
Publikováno v:
Journal of Genetic Syndromes & Gene Therapy. 3
Publikováno v:
Blood. 118:1490-1490
Abstract 1490 Background: The tumor suppressor p53 is frequently mutated in human cancer, including acute myeloid leukemia (AML). In AML, p53 mutations have been associated with poor risk cytogenetics (i.e. complex karyotype, −5/−7). However, the