Zobrazeno 1 - 10
of 46
pro vyhledávání: '"Scott M, Gianino"'
Supplementary Figure 4 from Interpreting Mammalian Target of Rapamycin and Cell Growth Inhibition in a Genetically Engineered Mouse Model of Nf1-Deficient Astrocytes
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8e0d34766bd47a770273e971a9293824
https://doi.org/10.1158/1535-7163.22496658
https://doi.org/10.1158/1535-7163.22496658
Supplementary Figure 2 from Interpreting Mammalian Target of Rapamycin and Cell Growth Inhibition in a Genetically Engineered Mouse Model of Nf1-Deficient Astrocytes
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6ee6ea9ef13d4090ffc5014804cd23da
https://doi.org/10.1158/1535-7163.22496667
https://doi.org/10.1158/1535-7163.22496667
Supplementary Figure 6 from Interpreting Mammalian Target of Rapamycin and Cell Growth Inhibition in a Genetically Engineered Mouse Model of Nf1-Deficient Astrocytes
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a6db59498270b60a154d482ec53fccc8
https://doi.org/10.1158/1535-7163.22496652.v1
https://doi.org/10.1158/1535-7163.22496652.v1
Supplementary Figure Legends from Interpreting Mammalian Target of Rapamycin and Cell Growth Inhibition in a Genetically Engineered Mouse Model of Nf1-Deficient Astrocytes
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::052e05ac73230dcc55aaefe9c45c4721
https://doi.org/10.1158/1535-7163.22496649.v1
https://doi.org/10.1158/1535-7163.22496649.v1
The identification of mammalian target of rapamycin (mTOR) as a major mediator of neurofibromatosis-1 (NF1) tumor growth has led to the initiation of clinical trials using rapamycin analogs. Previous studies from our laboratory have shown that durabl
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::afa8cd26a0477449079fd41c1f040177
https://doi.org/10.1158/1535-7163.c.6535353
https://doi.org/10.1158/1535-7163.c.6535353
Supplementary Figure 1 from Interpreting Mammalian Target of Rapamycin and Cell Growth Inhibition in a Genetically Engineered Mouse Model of Nf1-Deficient Astrocytes
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7c6a4a0ba7564379f6cca61ebfe42ca2
https://doi.org/10.1158/1535-7163.22496670
https://doi.org/10.1158/1535-7163.22496670
Supplementary Figure 3 from Interpreting Mammalian Target of Rapamycin and Cell Growth Inhibition in a Genetically Engineered Mouse Model of Nf1-Deficient Astrocytes
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2781815fa6b0ff60e1606c8f2530b201
https://doi.org/10.1158/1535-7163.22496664
https://doi.org/10.1158/1535-7163.22496664
Supplementary Material and Methods from Interpreting Mammalian Target of Rapamycin and Cell Growth Inhibition in a Genetically Engineered Mouse Model of Nf1-Deficient Astrocytes
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a758c7937385ed93891383ebd9e06190
https://doi.org/10.1158/1535-7163.22496646
https://doi.org/10.1158/1535-7163.22496646
Autor:
Joshua B. Rubin, David H. Gutmann, David Piwnica-Worms, Joel R. Garbow, Patricia Goldhoff, Erin Jackson, Scott M. Gianino, Nicole M. Warrington
Supplementary Figure Legends 1-4 from Cyclic AMP Suppression Is Sufficient to Induce Gliomagenesis in a Mouse Model of Neurofibromatosis-1
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d0007d9449c8338f863aa270f53acd20
https://doi.org/10.1158/0008-5472.22385339.v1
https://doi.org/10.1158/0008-5472.22385339.v1
Autor:
Joshua B. Rubin, David H. Gutmann, David Piwnica-Worms, Joel R. Garbow, Patricia Goldhoff, Erin Jackson, Scott M. Gianino, Nicole M. Warrington
Supplementary Figure 2 from Cyclic AMP Suppression Is Sufficient to Induce Gliomagenesis in a Mouse Model of Neurofibromatosis-1
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6cb98d41d9ae47279b1cf7e201521597
https://doi.org/10.1158/0008-5472.22385348
https://doi.org/10.1158/0008-5472.22385348