Výsledky vyhledávání - "Schneider, UC"

Template-based target point identification for surgical occlusion of the middle meningeal artery during chronic subdural haematoma evacuation

Autor: Wessels, L, Brunk, I, Rusakowa, E, Schneider, UC, Vajkoczy, P, Hecht, N

Publikováno v: 73. Jahrestagung der Deutschen Gesellschaft für Neurochirurgie (DGNC), Joint Meeting mit der Griechischen Gesellschaft für Neurochirurgie; 20220529-20220601; Köln; DOCP200 /20220525/

Objective: Burr hole evacuation of chronic subdural hematoma (cSDH) carries a relevant risk of recurrence. Recent evidence has suggested that endovascular middle meningeal artery (MMA) occlusion may help reduce this risk. However, endovascular MMA oc

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Complete or partial parent artery sacrifice – effect of vessel occlusion strategies on complete obliteration of complex aneurysms

Autor: Wessels, L, Hecht, N, Faust, K, Schneider, UC, Czabanka, M, Vajkoczy, P

Publikováno v: 72. Jahrestagung der Deutschen Gesellschaft für Neurochirurgie (DGNC), Joint Meeting mit der Polnischen Gesellschaft für Neurochirurgie; 20210606-20210609; sine loco [digital]; DOCP161 /20210604/

Objective: A small number of complex intracranial aneurysms is not amenable to direct clipping strategies or endovascular treatment. In these patients, parent artery sacrifice and bypass revascularization for aneurysm occlusion is an option. There ar

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Comparison of treatment efficacy and outcome in patients with refractory trigeminal neuralgia and neurological co-morbidity – CyberKnife radiosurgery vs. radiofrequency thermocoagulation

Autor: Löbel, F, Senger, C, Temaj, E, Kluge, AK, Acker, G, Schneider, UC, Onken, J, Klene, W, Kord, M, Budach, V, Vajkoczy, P, Dengler, N

Objective: Treatment of symptomatic trigeminal neuralgia (TN) in patients with neurological co-morbidites, e.g., multiple sclerosis remains challenging. Single-fraction Cyberknife radiosurgery (CK) and retrogasserian thermocoagulation (TC) are possib

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Resolution of cerebral inflammation following subarachnoid hemorrhage in vivo

Autor: Patsouris, V, Blecharz-Lang, K, Schneider, UC, Vajkoczy, P

Publikováno v: 68. Jahrestagung der Deutschen Gesellschaft für Neurochirurgie (DGNC), 7. Joint Meeting mit der Society of British Neurological Surgeons (SBNS); 20170514-20170517; Magdeburg; DOCMO.20.07 /20170609/

Objective: Cerebral inflammation and neuronal cell death play an important role in the development of brain injury after SAH. In previous studies we have demonstrated an array of SAH-induced inflammatory processes in the injured brain including: micr

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Minocycline-mediated neuroprotection by counteracting microglia accumulation following subarachnoid hemorrhage

Autor: Patsouris, V, Blecharz, K, Schneider, UC, Vajkoczy, P

Publikováno v: 67. Jahrestagung der Deutschen Gesellschaft für Neurochirurgie (DGNC), 1. Joint Meeting mit der Koreanischen Gesellschaft für Neurochirurgie (KNS); 20160612-20160615; Frankfurt am Main; DOCDI.02.09 /20160608/

Objective: Neuroprotective treatment strategies aiming at interfering with either inflammation or cell death indicate the importance of these mechanisms in the development of brain injury after SAH. Our previous study showed microglia accumulation in

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Implantation of a new Vagus Nerve Stimulation (VNS) Therapy® generator, AspireSR® – Considerations and recommendations during implantation and replacement surgery – Comparison to a traditional system

Autor: Schneider, UC, Bohlmann, K, Vajkoczy, P, Straub, HB

Publikováno v: 66. Jahrestagung der Deutschen Gesellschaft für Neurochirurgie (DGNC); 20150607-20150610; Karlsruhe; DOCMI.05.02 /20150602/

Objective: The most widely used neuro-stimulation treatment for drug-resistant epilepsy is Vagus Nerve Stimulation (VNS) Therapy®. Ictal tachycardia can be an indicator of a seizure, and if monitored, can be used to trigger an additional on-demand s

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Combination of a Supratentorial Venous Anomaly and Infratentorial Developmental Venous Anomalies Mimicking AV-Malformation: A Case Report

Autor: Schneider Uc, Marc A. Brockmann, Johann Scharf, Marcel Seiz, Woitzik J

Publikováno v: Zentralblatt für Neurochirurgie - Central European Neurosurgery. 68:217-219

Developmental venous anomalies (DVAs) are considered to be congenital anatomical variants of venous drainage. In most cases unspecific symptoms lead to their diagnosis. We describe a 35-year-old patient presenting with headache and dizziness. MRI was

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https://doi.org/10.1055/s-2007-985854

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Activation of brain residential microglia is responsible for delayed neuronal apoptosis after experimental subarachnoid hemorrhage

Autor: Schneider, UC, Radon, AM, Magrini, S, Atangana, EN, Heppner, F, Vajkoczy, P

Publikováno v: 64. Jahrestagung der Deutschen Gesellschaft für Neurochirurgie (DGNC); 20130526-20130529; Düsseldorf; DOCMO.09.05 /20130521/

Objective: Inflammatory mechanisms have become an interesting target for analysis of early and delayed brain injury after subarachnoid hemorrhage (SAH). Our group could demonstrate a microglia activation in the central nervous system (CNS) after SAH,

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Aneurysmal subarachnoid hemorrhage induces pro-inflammatory properties within the cerebro-spinal fluid

Autor: Schneider, UC, Hakiy, N, Schiffler, J, Horn, P, Vajkoczy, P

Publikováno v: 63. Jahrestagung der Deutschen Gesellschaft für Neurochirurgie (DGNC), Joint Meeting mit der Japanischen Gesellschaft für Neurochirurgie (JNS); 20120613-20120616; Leipzig; DOCDO.01.04 /20120604/

Objective: Inflammatory mechanisms have become an interesting target for early and delayed brain injury after acute subarachnoid hemorrhage (SAH). To functionally characterize pro-inflammatory and vasoconstrictive properties of cerebrospinal fluid af

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Inflammatory cells in the brain parenchyma after subarachnoid hemorrhage (SAH) are brain residential microglia cells and do not derive from the periphery

Autor: Schneider, UC, Elke, A, Brandenburg, S, Müller, A, Vajkoczy, P

Objective: Inflammatory mechanisms in early and delayed brain injury after SAH have become an interesting research target. In previous studies we could show an intraparenchymal inflammatory (Iba-1-positive cells) response after SAH. This cellular inf

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