Zobrazeno 1 - 10
of 73
pro vyhledávání: '"Satoshi Tateishi"'
Autor:
Yanzhe Gao, Elizabeth Mutter-Rottmayer, Alicia M. Greenwalt, Dennis Goldfarb, Feng Yan, Yang Yang, Raquel C. Martinez-Chacin, Kenneth H. Pearce, Satoshi Tateishi, Michael B. Major, Cyrus Vaziri
Publikováno v:
Nature Communications, Vol 15, Iss 1, Pp 1-5 (2024)
Externí odkaz:
https://doaj.org/article/38abafd32f7e4839a15ea58ce38cbe75
Autor:
Yutaka Takaoka, Mika Ohta, Satoshi Tateishi, Aki Sugano, Eiji Nakano, Kenji Miura, Takashi Suzuki, Chikako Nishigori
Publikováno v:
Biomedicines, Vol 9, Iss 3, p 249 (2021)
Xeroderma pigmentosum complementation group D (XPD) is a UV-sensitive syndrome and a rare incurable genetic disease which is caused by the genetic mutation of the excision repair cross-complementation group 2 gene (ERCC2). Patients who harbor only XP
Externí odkaz:
https://doaj.org/article/d519288b3295445da882cc5d8cfd204d
Autor:
Yanzhe Gao, Elizabeth Mutter-Rottmayer, Alicia M. Greenwalt, Dennis Goldfarb, Feng Yan, Yang Yang, Raquel C. Martinez-Chacin, Kenneth H. Pearce, Satoshi Tateishi, Michael B. Major, Cyrus Vaziri
Publikováno v:
Nature Communications, Vol 7, Iss 1, Pp 1-14 (2016)
Abstract Trans-lesion synthesis (TLS) is an important DNA-damage tolerance mechanism that permits ongoing DNA synthesis in cells harbouring damaged genomes. The E3 ubiquitin ligase RAD18 activates TLS by promoting recruitment of Y-family DNA polymera
Externí odkaz:
https://doaj.org/article/0e5d0907f6a14928a23a27cda5802f8e
Publikováno v:
Journal of Orthopaedic Science.
It is unclear whether hip and pelvic mobility in the sagittal plane are associated with hip function in FAIS. This study aimed to determine whether hip-pelvis-lumbar mobility is associated with preoperative hip function and postoperative outcomes in
Autor:
Chikako Senju, Yuka Nakazawa, Taichi Oso, Mayuko Shimada, Kana Kato, Michiko Matsuse, Mariko Tsujimoto, Taro Masaki, Yasushi Miyazaki, Satoshi Fukushima, Satoshi Tateishi, Atsushi Utani, Hiroyuki Murota, Katsumi Tanaka, Norisato Mitsutake, Shinichi Moriwaki, Chikako Nishigori, Tomoo Ogi
Publikováno v:
Proceedings of the National Academy of Sciences of the United States of America; 7/4/2023, Vol. 120 Issue 27, p1-12, 24p
Autor:
Megumi Sasatani, Yanbin Xu, Hidehiko Kawai, Lili Cao, Satoshi Tateishi, Tsutomu Shimura, Jianxiang Li, Daisuke Iizuka, Asao Noda, Kanya Hamasaki, Yoichiro Kusunoki, Kenji Kamiya
Publikováno v:
PLoS ONE, Vol 10, Iss 2, p e0117845 (2015)
The ubiquitin ligase RAD18 is involved in post replication repair pathways via its recruitment to stalled replication forks, and its role in the ubiquitylation of proliferating cell nuclear antigen (PCNA). Recently, it has been reported that RAD18 is
Externí odkaz:
https://doaj.org/article/b92572082da74c93908a5a8a82687781
Autor:
Satona Murakami, Soshi Uchida, Yasuhiro Yamamoto, Jahorn, Nobuyuki Watanabe, Hidetsugu Ohara, Satoshi Tateishi, Taiki Kojima
Publikováno v:
Journal of Orthopaedic Science. 24:447-451
This study aimed to develop a Japanese version of the international PROMs "Vail Hip Score (Vail10)" and to establish its reliability, validity, and responsiveness with COSMIN check-list.The study was conducted from March 2016 to October 2017 and incl
Autor:
Siddhi Desai, Qisheng Gu, Di Wu, Lisle E. Mose, Joel S. Parker, Jitong Lou, Brandon A. Price, Satoshi Tateishi, Brian Dong Chen, Yuri Fedoriw, Cyrus Vaziri, Yang Yang, Yuheng Qiu
Publikováno v:
Nar Cancer
The E3 ubiquitin ligase Rad18 promotes a damage-tolerant and error-prone mode of DNA replication termed trans-lesion synthesis that is pathologically activated in cancer. However, the impact of vertebrate Rad18 on cancer genomes is not known. To dete
Autor:
Satoshi Tateishi, Kawsar Mustofa, Chie Tateishi, Mami Chirifu, Yuki Tanoue, Tatsuya Shimasaki, Teruya Nakamura
Publikováno v:
Journal of biochemistry. 170(1)
The E3 ubiquitin ligase RAD18 mono-ubiquitinates PCNA to promote bypass of replication fork-stalling DNA lesions. On the other hand, RAD18 also contributes to DNA double-strand break (DSB) repair. RAD18 is recruited to ionizing radiation (IR)-induced
Publikováno v:
Environmental and molecular mutagenesisREFERENCES. 61(7)
Checkpoint kinase 2 (human CHEK2; murine Chk2) is a critical mediator of the DNA damage response and has established roles in DNA double strand break (DSB)-induced apoptosis and cell cycle arrest. DSBs may be invoked directly by ionizing radiation bu