Zobrazeno 1 - 10
of 10
pro vyhledávání: '"Satja Mulej Bratec"'
Social reappraisal of emotions is linked with the social presence effect in the default mode network
Publikováno v:
Frontiers in Psychiatry, Vol 14 (2023)
IntroductionSocial reappraisal, during which one person deliberately tries to regulate another’s emotions, is a powerful cognitive form of social emotion regulation, crucial for both daily life and psychotherapy. The neural underpinnings of social
Externí odkaz:
https://doaj.org/article/02f4bff42290485a8dea76f847797553
Autor:
Teresa Bertram, Daniel Hoffmann Ayala, Maria Huber, Felix Brandl, Georg Starke, Christian Sorg, Satja Mulej Bratec
Publikováno v:
Frontiers in Psychiatry, Vol 13 (2023)
IntroductionThreat processing, enabled by threat circuits, is supported by a remarkably conserved neural architecture across mammals. Threatening stimuli relevant for most species include the threat of being attacked by a predator or an aggressive co
Externí odkaz:
https://doaj.org/article/7871c83ce603451d9dfe3b651aae8126
Autor:
Felix Brandl, Benedikt Weise, Satja Mulej Bratec, Nazia Jassim, Daniel Hoffmann Ayala, Teresa Bertram, Markus Ploner, Christian Sorg
Publikováno v:
Neuropsychopharmacology. 47:1071-1080
Major depressive disorder (MDD), anxiety disorders (ANX), and chronic pain (CP) are closely-related disorders with both high degrees of comorbidity among them and shared risk factors. Considering this multi-level overlap, but also the distinct phenot
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::bc36a465418c40bcd00860c34c074b7d
https://doi.org/10.1038/s41386-022-01271-y
https://doi.org/10.1038/s41386-022-01271-y
Autor:
Teresa Bertram, Daniel Hoffmann Ayala, Maria Huber, Felix Brandl, Georg Starke, Christian Sorg, Satja Mulej Bratec
IntroductionThreat processing, enabled by threat circuits, is supported by a remarkably conserved neural architecture across mammals. Threatening stimuli relevant for most species include the threat of being attacked by a predator or an aggressive co
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9efd5b23c912a2f6f2f4110c748539c1
https://mediatum.ub.tum.de/doc/1696450/document.pdf
https://mediatum.ub.tum.de/doc/1696450/document.pdf
Your presence soothes me: a neural process model of aversive emotion regulation via social buffering
Publikováno v:
Social Cognitive and Affective Neuroscience
The reduction of aversive emotions by a conspecific’s presence—called social buffering—is a universal phenomenon in the mammalian world and a powerful form of human social emotion regulation. Animal and human studies on neural pathways underlyi
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f2aad7a3cc4d9d6e9b907bd7a6033f92
https://doi.org/10.1093/scan/nsaa068
https://doi.org/10.1093/scan/nsaa068
Autor:
Valentin Riedl, Satja Mulej Bratec, Felix Brandl, Chun Meng, Xiyao Xie, Afra M. Wohlschläger, Christian Sorg
Publikováno v:
Cerebral Cortex. 28:3082-3094
Cognitive emotion regulation (CER) enables humans to flexibly modulate their emotions. While local theories of CER neurobiology suggest interactions between specialized local brain circuits underlying CER, e.g., in subparts of amygdala and medial pre
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::23ab1930422ec79923319cdefc179553
https://doi.org/10.1093/cercor/bhx178
https://doi.org/10.1093/cercor/bhx178
Autor:
Satja Mulej Bratec, Afra M. Wohlschläger, Xiyao Xie, Valentin Riedl, Yijun Wang, Christian Sorg, Leonhard Schilbach, Claus Zimmer
Publikováno v:
NeuroImage. 147:650-657
Cognitive emotion regulation (CER) is a critical human ability to face aversive emotional stimuli in a flexible way, via recruitment of specific prefrontal brain circuits. Animal research reveals a central role of ventral striatum in emotional behavi
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9bbebb2bb6836d72bee390aa8579424b
https://doi.org/10.1016/j.neuroimage.2016.12.078
https://doi.org/10.1016/j.neuroimage.2016.12.078
Autor:
Anselm Doll, Leonhard Schilbach, Xiyao Xie, Christian Sorg, Gabriele Schmid, Valentin Riedl, Afra M. Wohlschläger, Satja Mulej Bratec, Claus Zimmer
Publikováno v:
NeuroImage. 123:138-148
Cognitive emotion regulation is a powerful way of modulating emotional responses. However, despite the vital role of emotions in learning, it is unknown whether the effect of cognitive emotion regulation also extends to the modulation of learning. Co
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::90536d1569ece3dbb31b43ca2693168d
https://doi.org/10.1016/j.neuroimage.2015.08.038
https://doi.org/10.1016/j.neuroimage.2015.08.038
Autor:
Chun Meng, Claus Zimmer, Leonhard Schilbach, Satja Mulej Bratec, Gabriele Schmid, Reinhard Pekrun, Afra M. Wohlschläger, Christian Sorg, Kathrin Finke, Hans Förstl, Anselm Doll, Xiyao Xie, Valentin Riedl
Publikováno v:
NeuroImage. 134
Socially-induced cognitive emotion regulation (Social-Reg) is crucial for emotional well-being and social functioning; however, its brain mechanisms remain poorly understood. Given that both social cognition and cognitive emotion regulation engage ke
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::33e5be4ffd95561dbfd80959e1950064
https://pubmed.ncbi.nlm.nih.gov/27095057
https://pubmed.ncbi.nlm.nih.gov/27095057
Autor:
Kuangyu Shi, Stefan Förster, Martin Scherr, Valentin Riedl, Chun Meng, Masoud Tahmasian, Alexander Drzezga, Janine Diehl-Schmid, Timo Grimmer, Igor Yakushev, Markus Schwaiger, Christian Sorg, Lorenzo Pasquini, Satja Mulej Bratec
Publikováno v:
Neurology. 84(19)
Based on the hippocampus disconnection hypothesis in Alzheimer disease (AD), which postulates that uncoupling from cortical inputs contributes to disinhibition-like changes in hippocampus activity, we suggested that in patients with AD, the more the
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8788991d71a81bce7dc4c692f24e378f
https://pubmed.ncbi.nlm.nih.gov/25878180
https://pubmed.ncbi.nlm.nih.gov/25878180