Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Sarah Y. Weißenberg"'
Autor:
Annika Wiedemann, Marie Lettau, Sarah Y. Weißenberg, Ana-Luisa Stefanski, Eva-Vanessa Schrezenmeier, Hector Rincon-Arevalo, Karin Reiter, Tobias Alexander, Falk Hiepe, Andreia C. Lino, Thomas Dörner
Publikováno v:
Frontiers in Immunology, Vol 12 (2021)
B- and T-lymphocyte attenuator (BTLA/CD272) is an inhibitory checkpoint molecule expressed on T and B cells. Prior studies reported defective function of BTLA by T cells in patients with systemic lupus erythematosus (SLE), whereas nothing is known ab
Externí odkaz:
https://doaj.org/article/0d325ee477fd4493886422919bf70fff
Autor:
Sarah Y. Weißenberg, Franziska Szelinski, Eva Schrezenmeier, Ana-Luisa Stefanski, Annika Wiedemann, Hector Rincon-Arevalo, Anna Welle, Annemarie Jungmann, Karl Nordström, Jörn Walter, Juliana Imgenberg-Kreuz, Gunnel Nordmark, Lars Rönnblom, Prathyusha Bachali, Michelle D. Catalina, Amrie C. Grammer, Peter E. Lipsky, Andreia C. Lino, Thomas Dörner
Publikováno v:
Frontiers in Immunology, Vol 10 (2019)
Autoimmune diseases (AID) such as systemic lupus erythematosus (SLE), primary Sjögren's syndrome (pSS), and rheumatoid arthritis (RA) are chronic inflammatory diseases in which abnormalities of B cell function play a central role. Although it is wid
Externí odkaz:
https://doaj.org/article/d9ed2fdab4744e97827ac0c5501dd134
Autor:
Arman Aue, Franziska Szelinski, Sarah Y. Weißenberg, Thomas Rose, Andreia C. Lino, Annika Wiedemann, Thomas Dorner
Publikováno v:
Annals of the Rheumatic Diseases. 79:4-5
Background:Systemic lupus erythematosus (SLE) is characterized by two pathogenic key signatures, type I interferon (IFN) (1.) and B-cell abnormalities (2.). How these signatures are interrelated is not known. Type I-II IFN trigger activation of Janus
Autor:
Franziska Szelinski, Thomas Rose, Sarah Y. Weißenberg, Annika Wiedemann, Arman Aue, Thomas Dörner, Andreia C. Lino
Publikováno v:
Rheumatology (Oxford, England). 59(11)
Objectives SLE is characterized by two pathogenic key signatures, type I IFN and B-cell abnormalities. How these signatures are interrelated is not known. Type I-II IFN trigger activation of Janus kinase (JAK) – signal transducer and activator of t
Publikováno v:
FRIDAY, 15 JUNE 2018.
Background Previous studies demonstrated impaired B cell receptor (BCR) response in patients with systemic lupus erythematosus (SLE). Besides diminished cytokine production upon TLR9 stimulation1 it was found that phosphorylation of Syk2 and downstre