Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Sarah L Sokol-Borrelli"'
Publikováno v:
PLoS Pathogens, Vol 16, Iss 6, p e1008528 (2020)
Toxoplasma gondii and Hammondia hammondi are closely-related coccidian intracellular parasites that differ in their ability to cause disease in animal and (likely) humans. The role of the host response in these phenotypic differences is not known and
Externí odkaz:
https://doaj.org/article/de95e4da367c46b1bcc6cd4fffaa3dd8
Autor:
Sarah L. Sokol-Borrelli, Sarah M. Reilly, Michael J. Holmes, Stephanie B. Orchanian, Mackenzie D. Massmann, Katherine G. Sharp, Leah F. Cabo, Hisham S. Alrubaye, Bruno Martorelli Di Genova, Melissa B. Lodoen, William J. Sullivan, Jon P. Boyle
Publikováno v:
Nature Communications, Vol 14, Iss 1, Pp 1-20 (2023)
Abstract Identification of regulators of Toxoplasma gondii bradyzoite development and cyst formation is the most direct way to address the importance of parasite development in long-term persistence and reactivation of this parasite. Here we show tha
Externí odkaz:
https://doaj.org/article/308cbfa9402143bbbb45da037f0e7ce1
Publikováno v:
Frontiers in Cellular and Infection Microbiology, Vol 10 (2020)
Frontiers in Cellular and Infection Microbiology
Frontiers in Cellular and Infection Microbiology
Stage conversion is a critical life cycle feature for several Apicomplexan parasites as the ability to switch between life forms is critical for replication, dissemination, pathogenesis and ultimately, transmission to a new host. In order for these d
Publikováno v:
Int J Parasitol
Toxoplasma gondii is remarkably unique in its ability to successfully infect vertebrate hosts from multiple phyla and can successfully infect most cells within these organisms. The infection outcome in each of these species is determined by the compl
Publikováno v:
PLoS Pathogens
PLoS Pathogens, Vol 16, Iss 6, p e1008528 (2020)
PLoS Pathogens, Vol 16, Iss 6, p e1008528 (2020)
Toxoplasma gondii and Hammondia hammondi are closely-related coccidian intracellular parasites that differ in their ability to cause disease in animal and (likely) humans. The role of the host response in these phenotypic differences is not known and