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pro vyhledávání: '"Sarah E Baxley"'
Autor:
Stephanie L Easter, Elizabeth H Mitchell, Sarah E Baxley, Renee Desmond, Andra R Frost, Rosa Serra
Publikováno v:
PLoS ONE, Vol 9, Iss 11, p e113247 (2014)
Wnt5a is a non-canonical signaling Wnt that has been implicated in tumor suppression. We previously showed that loss of Wnt5a in MMTV-PyVmT tumors resulted in a switch in tumor phenotype resulting in tumors with increased basal phenotype and high Wnt
Externí odkaz:
https://doaj.org/article/4f0823c706c2480cb21e6a84f132c239
Publikováno v:
Gynecology and Pelvic Medicine. 3:35-35
Publikováno v:
Biology of Reproduction. 85:907-915
Wingless-related MMTV integration site 5A (Wnt5a )i s a noncanonical signaling WNT that is expressed in every stage of mouse mammary gland development except lactation. Using slow release pellets containing WNT5A as well as Wnt5a-null tissue, we prev
Autor:
Sarah E. Baxley, Rosa Serra
Publikováno v:
Current Drug Targets. 11:1089-1102
Transforming Growth Factor beta (TGFbeta) signaling influences most aspects of cellular function in addition to playing a major role in organ development, remodeling, and repair. Given the wide range of effects induced by TGFbeta, it is not surprisin
Publikováno v:
Biology of reproduction. 85(5)
Wingless-related MMTV integration site 5A (Wnt5a) is a noncanonical signaling WNT that is expressed in every stage of mouse mammary gland development except lactation. Using slow release pellets containing WNT5A as well as Wnt5a-null tissue, we previ
Publikováno v:
Breast Cancer Research : BCR
The tumour-suppressive effects of transforming growth factor-beta (TGF-beta) are well documented; however, the mechanistic basis of these effects is not fully understood. Previously, we showed that a non-canonical member of the Wingless-related prote
Autor:
Rosa Serra, Sarah E. Baxley, Andra R. Frost, Elizabeth H. Mitchell, Stephanie L. Easter, Renee A. Desmond
Publikováno v:
PLoS ONE, Vol 9, Iss 11, p e113247 (2014)
PLoS ONE
PLoS ONE
Wnt5a is a non-canonical signaling Wnt that has been implicated in tumor suppression. We previously showed that loss of Wnt5a in MMTV-PyVmT tumors resulted in a switch in tumor phenotype resulting in tumors with increased basal phenotype and high Wnt