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pro vyhledávání: '"Sarah, Bellouze"'
Autor:
Diana Toli, Dorothée Buttigieg, Stéphane Blanchard, Thomas Lemonnier, Boris Lamotte d’Incamps, Sarah Bellouze, Gilbert Baillat, Delphine Bohl, Georg Haase
Publikováno v:
Neurobiology of Disease, Vol 82, Iss , Pp 269-280 (2015)
Amyotrophic lateral sclerosis (ALS) is a severe and incurable neurodegenerative disease. Human motor neurons generated from induced pluripotent stem cells (iPSc) offer new perspectives for disease modeling and drug testing in ALS. In standard iPSc-de
Externí odkaz:
https://doaj.org/article/127b0c1de7534dae9c254c3dc12a0a5d
Autor:
Sarah Bellouze
Publikováno v:
Questions de santé publique. :1-8
Plus d’un milliard de personnes, soit environ 15 % de la population mondiale [1], présentent une forme ou une autre de handicap. Selon l’enquête Handicap-Santé 2008 de l’INSEE, à l’échelle nationale, 11,5 millions de personnes sont en pe
Autor:
Jean-Michel Vallat, Sébastien Schaller, Sarah Bellouze, Arnaud Jacquier, Stéphane Mathis, Laurence Richard, Michael K. E. Schäfer, Georg Haase
Publikováno v:
Brain Pathology. 27:459-471
Motor neuron diseases such as amyotrophic lateral sclerosis (ALS) are now recognized as multi-system disorders also involving various non-motor neuronal cell types. The precise extent and mechanistic basis of non-motor neuron damage in human ALS and
Autor:
Dorothée Buttigieg, Stéphane Blanchard, Diana Toli, Delphine Bohl, Georg Haase, Gilbert Baillat, Sarah Bellouze, Thomas Lemonnier, Boris Lamotte d'Incamps
Publikováno v:
Neurobiology of Disease, Vol 82, Iss, Pp 269-280 (2015)
Neurobiology of Disease
Neurobiology of Disease, Elsevier, 2015, 82, pp.269-280. ⟨10.1016/j.nbd.2015.06.011⟩
Neurobiology of Disease, 2015, 82, pp.269-280. ⟨10.1016/j.nbd.2015.06.011⟩
Neurobiology of Disease
Neurobiology of Disease, Elsevier, 2015, 82, pp.269-280. ⟨10.1016/j.nbd.2015.06.011⟩
Neurobiology of Disease, 2015, 82, pp.269-280. ⟨10.1016/j.nbd.2015.06.011⟩
International audience; Amyotrophic lateral sclerosis (ALS) is a severe and incurable neurodegenerative disease. Human motor neurons generated from induced pluripotent stem cells (iPSc) offer new perspectives for disease modeling and drug testing in
Autor:
Dorothée Buttigieg, Gilbert Baillat, Catherine Rabouille, Georg Haase, Michael K. E. Schäfer, Sarah Bellouze
Publikováno v:
Human Molecular Genetics, 23(22), 5961-75. Oxford University Press
Golgi fragmentation is an early hallmark of many neurodegenerative diseases but its pathophysiological relevance and molecular mechanisms are unclear. We here demonstrate severe and progressive Golgi fragmentation in motor neurons of progressive moto
Autor:
Michael K, Schäfer, Sarah, Bellouze, Arnaud, Jacquier, Sébastien, Schaller, Laurence, Richard, Stéphane, Mathis, Jean-Michel, Vallat, Georg, Haase
Publikováno v:
Brain Pathol
Motor neuron diseases such as amyotrophic lateral sclerosis (ALS) are now recognized as multi‐system disorders also involving various non‐motor neuronal cell types. The precise extent and mechanistic basis of non‐motor neuron damage in human AL
Autor:
Georg Haase, Catherine Rabouille, Dorothée Buttigieg, Gilbert Baillat, Pierre de la Grange, Sarah Bellouze
Publikováno v:
Molecular Neurodegeneration
Molecular Neurodegeneration, BioMed Central, 2016, 11, pp.43. ⟨10.1186/s13024-016-0111-6⟩
Molecular Neurodegeneration, 2016, 11, pp.43. ⟨10.1186/s13024-016-0111-6⟩
Molecular Neurodegeneration, 11(1). BioMed Central
Molecular Neurodegeneration [E], 11(1). BioMed Central
Molecular Neurodegeneration, BioMed Central, 2016, 11, pp.43. ⟨10.1186/s13024-016-0111-6⟩
Molecular Neurodegeneration, 2016, 11, pp.43. ⟨10.1186/s13024-016-0111-6⟩
Molecular Neurodegeneration, 11(1). BioMed Central
Molecular Neurodegeneration [E], 11(1). BioMed Central
Background Pathological Golgi fragmentation represents a constant pre-clinical feature of many neurodegenerative diseases including amyotrophic lateral sclerosis (ALS) but its molecular mechanisms remain hitherto unclear. Results Here, we show that t
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c22dc7d9000feb9f1f2d15617ffabf7e
https://hal-amu.archives-ouvertes.fr/hal-01463801
https://hal-amu.archives-ouvertes.fr/hal-01463801
Autor:
Amapola Autillo-Touati, Helene Vacher, Bénédicte Dargent, Sarah Bellouze, Magali Musarella, Ghislaine Caillol
Publikováno v:
European Journal of Neuroscience. 36:2400-2408
Postnatal formation of the neuromuscular synapse requires complex interactions among nerve terminal, muscle fibres and terminal Schwann cells. In motor endplate disease (med) mice, neuromuscular transmission is severely impaired without alteration of
Publikováno v:
Human Molecular Genetics. 18:2127-2139
Three neurodegenerative diseases affecting upper and/or lower motor neurons have been associated with loss of ALS2/Alsin function: juvenile amyotrophic lateral sclerosis, primary lateral sclerosis and infantile-onset ascending hereditary spastic para
Autor:
Ghislaine, Caillol, Hélène, Vacher, Magali, Musarella, Sarah, Bellouze, Bénédicte, Dargent, Amapola, Autillo-Touati
Publikováno v:
The European journal of neuroscience. 36(4)
Postnatal formation of the neuromuscular synapse requires complex interactions among nerve terminal, muscle fibres and terminal Schwann cells. In motor endplate disease (med) mice, neuromuscular transmission is severely impaired without alteration of