Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Sara Moreno-Estar"'
Autor:
Diego A. Peraza, Lucía Benito-Salamanca, Sara Moreno-Estar, Esperanza Alonso, José R. López-López, M. Teresa Pérez-Garcia, Pilar Cidad
Publikováno v:
Frontiers in Physiology, Vol 15 (2024)
IntroductionCoronary artery disease (CAD) is the foremost single cause of mortality and disability globally. Patients with type 2 diabetes (T2DM) have a higher incidence of CAD, and poorer prognosis. The low-grade inflammation associated to T2DM cont
Externí odkaz:
https://doaj.org/article/ad3ca193969c450d8a4bdd0cc6067603
Autor:
Marycarmen Arevalo-Martinez, Pilar Cidad, Sara Moreno-Estar, Mirella Fernández, Sebastian Albinsson, Irene Cózar-Castellano, José R. López-López, M. Teresa Pérez-Garcia
Publikováno v:
Molecular Metabolism, Vol 53, Iss , Pp 101306- (2021)
Objectives: Restenosis after vessel angioplasty due to dedifferentiation of the vascular smooth muscle cells (VSMCs) limits the success of surgical treatment of vascular occlusions. Type 2 diabetes (T2DM) has a major impact on restenosis, with patien
Externí odkaz:
https://doaj.org/article/e00a9e75cdf44eabb6c98499dacf5b54
Autor:
F. Javier Arias, Mercedes Santos, M. Teresa Pérez-García, Pilar Cidad, Sofía Serrano, Marycarmen Arévalo-Martínez, Sara Moreno-Estar, José R. López-López
Publikováno v:
Digital.CSIC. Repositorio Institucional del CSIC
instname
instname
Coronary artery disease (CAD) is the most common cardiovascular disorder. Vascular surgery strategies for coronary revascularization (either percutaneous or open) show a high rate of failure because of restenosis of the vessel, due to phenotypic swit
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::efb3baae241743045d2a7d1330f6fa85
https://doi.org/10.1016/j.actbio.2020.07.053
https://doi.org/10.1016/j.actbio.2020.07.053
Autor:
José R. López-López, Pilar Cidad, Mirella Fernández, Sebastian Albinsson, Sara Moreno-Estar, M. Teresa Pérez-García, Irene Cózar-Castellano, Marycarmen Arévalo-Martínez
Publikováno v:
Digital.CSIC. Repositorio Institucional del CSIC
instname
UVaDOC. Repositorio Documental de la Universidad de Valladolid
Molecular Metabolism, Vol 53, Iss, Pp 101306-(2021)
Molecular Metabolism
instname
UVaDOC. Repositorio Documental de la Universidad de Valladolid
Molecular Metabolism, Vol 53, Iss, Pp 101306-(2021)
Molecular Metabolism
Producción Científica
Objectives: Restenosis after vessel angioplasty due to dedifferentiation of the vascular smooth muscle cells (VSMCs) limits the success of surgical treatment of vascular occlusions. Type 2 diabetes (T2DM) has a major impa
Objectives: Restenosis after vessel angioplasty due to dedifferentiation of the vascular smooth muscle cells (VSMCs) limits the success of surgical treatment of vascular occlusions. Type 2 diabetes (T2DM) has a major impa
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7cbcc347fd83fc9d342ee5c3f4b295be
http://hdl.handle.net/10261/260813
http://hdl.handle.net/10261/260813
Autor:
Julia Serna, Sara Moreno-Estar, Diego A. Peraza, Dino Gobelli, Maria Simarro, José R. López López, Pilar Cidad, Miguel A. de la Fuente, M. Teresa Perez-Garcia
Publikováno v:
Biophysical Journal. 121:385a-386a
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::8d0734f9c3e1abc8b98215f69e683d21
https://doi.org/10.1016/j.bpj.2021.11.821
https://doi.org/10.1016/j.bpj.2021.11.821
Autor:
Pilar Cidad, Mirella Fernández, Sara Moreno-Estar, M. Teresa Pérez-García, Miguel Angel de la Fuente, José R. López-López, Nadia García-Mateo, Julia Serna, Marycarmen Arévalo-Martínez, María Simarro, Karl Swärd
Publikováno v:
UVaDOC. Repositorio Documental de la Universidad de Valladolid
instname
Arterioscler Thromb Vasc Biol
Digital.CSIC. Repositorio Institucional del CSIC
instname
Arterioscler Thromb Vasc Biol
Digital.CSIC. Repositorio Institucional del CSIC
[Objective]: We have previously described that changes in the expression of Kv channels associate to phenotypic modulation (PM), so that Kv1.3/Kv1.5 ratio is a landmark of vascular smooth muscle cells phenotype. Moreover, we demonstrated that the Kv1
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::bfc9ff8ee5dda5e54c4f9a96b24c77fd
https://doi.org/10.1161/atvbaha.119.313492
https://doi.org/10.1161/atvbaha.119.313492