Zobrazeno 1 - 10
of 43
pro vyhledávání: '"Sara De Palma"'
Autor:
Daniele Capitanio, Manuela Moriggi, Enrica Torretta, Pietro Barbacini, Sara De Palma, Agnese Viganò, Hanns Lochmüller, Francesco Muntoni, Alessandra Ferlini, Marina Mora, Cecilia Gelfi
Publikováno v:
Journal of Cachexia, Sarcopenia and Muscle, Vol 11, Iss 2, Pp 547-563 (2020)
Abstract Background Duchenne muscular dystrophy (DMD) and Becker muscular dystrophy (BMD) are characterized by muscle wasting leading to loss of ambulation in the first or third decade, respectively. In DMD, the lack of dystrophin hampers connections
Externí odkaz:
https://doaj.org/article/28b1bdd4361d472ab808673911eccfe0
Autor:
Daniele Capitanio, Manuela Moriggi, Sara De Palma, Dario Bizzotto, Sibilla Molon, Enrica Torretta, Chiara Fania, Paolo Bonaldo, Cecilia Gelfi, Paola Braghetta
Publikováno v:
Frontiers in Molecular Neuroscience, Vol 10 (2017)
Collagen VI is an extracellular matrix (ECM) protein playing a key role in skeletal muscles and whose deficiency leads to connective tissue diseases in humans and in animal models. However, most studies have been focused on skeletal muscle features.
Externí odkaz:
https://doaj.org/article/d4c3c9aa7b554f0d90f72f904691b6d7
Autor:
Roberta Palorini, Giuseppina Votta, Yuri Pirola, Humberto De Vitto, Sara De Palma, Cristina Airoldi, Michele Vasso, Francesca Ricciardiello, Pietro Paolo Lombardi, Claudia Cirulli, Raffaella Rizzi, Francesco Nicotra, Karsten Hiller, Cecilia Gelfi, Lilia Alberghina, Ferdinando Chiaradonna
Publikováno v:
PLoS Genetics, Vol 12, Iss 3, p e1005931 (2016)
Cancer cells often rely on glycolysis to obtain energy and support anabolic growth. Several studies showed that glycolytic cells are susceptible to cell death when subjected to low glucose availability or to lack of glucose. However, some cancer cell
Externí odkaz:
https://doaj.org/article/1dffd9bb091d49a68263f73cbf38e7da
Autor:
Sara De Palma, Roberta Leone, Paolo Grumati, Michele Vasso, Roman Polishchuk, Daniele Capitanio, Paola Braghetta, Paolo Bernardi, Paolo Bonaldo, Cecilia Gelfi
Publikováno v:
PLoS ONE, Vol 8, Iss 2, p e56716 (2013)
This study identifies metabolic and protein phenotypic alterations in gastrocnemius, tibialis anterior and diaphragm muscles of Col6a1(-/-) mice, a model of human collagen VI myopathies. All three muscles of Col6a1(-/-) mice show some common changes
Externí odkaz:
https://doaj.org/article/06f11c51d48c481d9176ab93c19dd84c
Autor:
Roberta Leone a, Paola Giussani b, Sara De Palma c, Chiara Fania a, Daniele Capitanio a, d, Michele Vasso c, Loredana Brioschi b, Laura Riboni b, Paola Viani b, Cecilia Gelfi *a, c
Publikováno v:
Molecular bioSystems
11 (2015): 1612–1621. doi:10.1039/c4mb00725e
info:cnr-pdr/source/autori:Roberta Leone a, Paola Giussani b, Sara De Palma c, Chiara Fania a, Daniele Capitanio a,d, Michele Vasso c, Loredana Brioschi b, Laura Riboni b, Paola Viani b and Cecilia Gelfi *a,c,d/titolo:Proteomic analysis of human glioblastoma cell lines differently resistant to a nitric oxide releasing agent/doi:10.1039%2Fc4mb00725e/rivista:Molecular bioSystems (Print)/anno:2015/pagina_da:1612/pagina_a:1621/intervallo_pagine:1612–1621/volume:11
11 (2015): 1612–1621. doi:10.1039/c4mb00725e
info:cnr-pdr/source/autori:Roberta Leone a, Paola Giussani b, Sara De Palma c, Chiara Fania a, Daniele Capitanio a,d, Michele Vasso c, Loredana Brioschi b, Laura Riboni b, Paola Viani b and Cecilia Gelfi *a,c,d/titolo:Proteomic analysis of human glioblastoma cell lines differently resistant to a nitric oxide releasing agent/doi:10.1039%2Fc4mb00725e/rivista:Molecular bioSystems (Print)/anno:2015/pagina_da:1612/pagina_a:1621/intervallo_pagine:1612–1621/volume:11
Glioblastoma multiforme is the most aggressive astrocytoma characterized by the development of resistant cells to various cytotoxic stimuli. Nitric oxide (NO) is able to overcome tumor resistance in PTEN mutated rat C6 glioma cells due to its ability
Autor:
Cecilia Gelfi, Michele Vasso, Patrizia Procacci, Sara De Palma, Daniele Capitanio, Enrica Torretta, Francesco Paolo Cammarata, Chiara Fania, Valerio Magnaghi
Publikováno v:
Proteomics (Weinh., Internet) 16 (2016): 645–656. doi:10.1002/pmic.201500395
info:cnr-pdr/source/autori:Capitanio D.; Vasso M.; De Palma S.; Fania C.; Torretta E.; Cammarata F.P.; Magnaghi V.; Procacci P.; Gelfi C./titolo:Specific protein changes contribute to the differential muscle mass loss during ageing/doi:10.1002%2Fpmic.201500395/rivista:Proteomics (Weinh., Internet)/anno:2016/pagina_da:645/pagina_a:656/intervallo_pagine:645–656/volume:16
info:cnr-pdr/source/autori:Capitanio D.; Vasso M.; De Palma S.; Fania C.; Torretta E.; Cammarata F.P.; Magnaghi V.; Procacci P.; Gelfi C./titolo:Specific protein changes contribute to the differential muscle mass loss during ageing/doi:10.1002%2Fpmic.201500395/rivista:Proteomics (Weinh., Internet)/anno:2016/pagina_da:645/pagina_a:656/intervallo_pagine:645–656/volume:16
In the skeletal muscle, the ageing process is characterized by a loss of muscle mass and strength, coupled with a decline of mitochondrial function and a decrease of satellite cells. This profile is more pronounced in hindlimb than in forelimb muscle
Autor:
Paolo Cerretelli, Marilena Ripamonti, Shajna Begum, Cecilia Gelfi, Sara De Palma, Robin Wait, Agnese Viganò
Publikováno v:
Proteomics (Weinh., Print) 6 (2006): 321–340.
info:cnr-pdr/source/autori:Gelfi C. 1, Viganò A. 1 2, De Palma S. 1 2, Ripamonti M .1, Begum S. 3, Cerretelli P.2, Wait R. 3/titolo:2-D protein maps of rat gastrocnemius and soleus muscles: A tool for muscle plasticity assessment/doi:/rivista:Proteomics (Weinh., Print)/anno:2006/pagina_da:321/pagina_a:340/intervallo_pagine:321–340/volume:6
info:cnr-pdr/source/autori:Gelfi C. 1, Viganò A. 1 2, De Palma S. 1 2, Ripamonti M .1, Begum S. 3, Cerretelli P.2, Wait R. 3/titolo:2-D protein maps of rat gastrocnemius and soleus muscles: A tool for muscle plasticity assessment/doi:/rivista:Proteomics (Weinh., Print)/anno:2006/pagina_da:321/pagina_a:340/intervallo_pagine:321–340/volume:6
Functional characterization of muscle fibers relies on ATPase activity and on differential measurements of metabolic proteins, including mitochondrial and glycolytic enzymes, glucose, lactate and lactic acid transporters, calcium cycling proteins and
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c7f2ae094dd200b1c371aed9569e19c6
https://doi.org/10.1002/pmic.200501337
https://doi.org/10.1002/pmic.200501337
Autor:
Michele Vasso, Karsten Hiller, Francesco Nicotra, Sara De Palma, Raffaella Rizzi, Roberta Palorini, Lilia Alberghina, Claudia Cirulli, Pietro Paolo Lombardi, Giuseppina Votta, Ferdinando Chiaradonna, Francesca Ricciardiello, Humberto De Vitto, Cecilia Gelfi, Yuri Pirola, Cristina Airoldi
Publikováno v:
PLOS genetics 12 (2016). doi:10.1371/journal.pgen.1005931
info:cnr-pdr/source/autori:Palorini R.; Votta G.; Pirola Y.; De Vitto H.; De Palma S.; Airoldi C.; Vasso M.; Ricciardiello F.; Lombardi P.P.; Cirulli C.; Rizzi R.; Nicotra F.; Hiller K.; Gelfi C.; Alberghina L.; Chiaradonna F./titolo:Protein Kinase A Activation Promotes Cancer Cell Resistance to Glucose Starvation and Anoikis/doi:10.1371%2Fjournal.pgen.1005931/rivista:PLOS genetics/anno:2016/pagina_da:/pagina_a:/intervallo_pagine:/volume:12
PLoS Genetics, Vol 12, Iss 3, p e1005931 (2016)
PLoS genetics, 12(3), 1005931. United States (2016).
PLoS Genetics
info:cnr-pdr/source/autori:Palorini R.; Votta G.; Pirola Y.; De Vitto H.; De Palma S.; Airoldi C.; Vasso M.; Ricciardiello F.; Lombardi P.P.; Cirulli C.; Rizzi R.; Nicotra F.; Hiller K.; Gelfi C.; Alberghina L.; Chiaradonna F./titolo:Protein Kinase A Activation Promotes Cancer Cell Resistance to Glucose Starvation and Anoikis/doi:10.1371%2Fjournal.pgen.1005931/rivista:PLOS genetics/anno:2016/pagina_da:/pagina_a:/intervallo_pagine:/volume:12
PLoS Genetics, Vol 12, Iss 3, p e1005931 (2016)
PLoS genetics, 12(3), 1005931. United States (2016).
PLoS Genetics
Cancer cells often rely on glycolysis to obtain energy and support anabolic growth. Several studies showed that glycolytic cells are susceptible to cell death when subjected to low glucose availability or to lack of glucose. However, some cancer cell
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e48005efae1bf4456e9cf945b4cb0be6
http://www.cnr.it/prodotto/i/354677
http://www.cnr.it/prodotto/i/354677
Autor:
Cecilia Gelfi, Sara De Palma, Serena Zacchigna, Michele Vasso, Manuela Moriggi, Giorgio Friso, Mauro Sturnega, Antero Macedo, Mauro Giacca
Publikováno v:
Human gene therapy 23 (2012): 146–157. doi:10.1089/hum.2011.157
info:cnr-pdr/source/autori:Antero Macedo; Manuela Moriggi; Michele Vasso; Sara De Palma; Mauro Sturnega; Giorgio Friso; Cecilia Gelfi; Mauro Giacca; Serena Zacchigna/titolo:Enhanced Athletic Performance on Multisite AAV-IGF1 Gene Transfer Coincides with Massive Modification of the Muscle Proteome./doi:10.1089%2Fhum.2011.157/rivista:Human gene therapy/anno:2012/pagina_da:146/pagina_a:157/intervallo_pagine:146–157/volume:23
Human Gene Therapy
info:cnr-pdr/source/autori:Antero Macedo; Manuela Moriggi; Michele Vasso; Sara De Palma; Mauro Sturnega; Giorgio Friso; Cecilia Gelfi; Mauro Giacca; Serena Zacchigna/titolo:Enhanced Athletic Performance on Multisite AAV-IGF1 Gene Transfer Coincides with Massive Modification of the Muscle Proteome./doi:10.1089%2Fhum.2011.157/rivista:Human gene therapy/anno:2012/pagina_da:146/pagina_a:157/intervallo_pagine:146–157/volume:23
Human Gene Therapy
Progress in gene therapy has hinted at the potential misuse of gene transfer in sports to achieve better athletic performance, while escaping from traditional doping detection methods. Suitable animal models are therefore required in order to better
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ffccf1609ade3d3fd48a9ec348f74778
http://online.liebertpub.com/doi/abs/10.1089/hum.2011.157
http://online.liebertpub.com/doi/abs/10.1089/hum.2011.157
Autor:
Sara De Palma 1, 2, Daniele Capitanio1 3, Michele Vasso 2, Paola Braghetta 4, Chiara Scotton 5, Paolo Bonaldo 4, Hanns Lochmüller 6, Francesco Muntoni 7, Alessandra Ferlini 5, Cecilia Gelfi 1, 3
Publikováno v:
Journal of proteome research
3 (2014): 11–5022. doi:10.1021/pr500675e
info:cnr-pdr/source/autori:Sara De Palma 1,2, Daniele Capitanio1 3, Michele Vasso 2, Paola Braghetta 4, Chiara Scotton 5, Paolo Bonaldo 4, Hanns Lochmüller 6, Francesco Muntoni 7, Alessandra Ferlini 5 and Cecilia Gelfi 1,2,3/titolo:Muscle proteomics reveals novel insights into the pathophysiological mechanisms of collagen VI myopathies./doi:10.1021%2Fpr500675e/rivista:Journal of proteome research (Print)/anno:2014/pagina_da:11/pagina_a:5022/intervallo_pagine:11–5022/volume:3
3 (2014): 11–5022. doi:10.1021/pr500675e
info:cnr-pdr/source/autori:Sara De Palma 1,2, Daniele Capitanio1 3, Michele Vasso 2, Paola Braghetta 4, Chiara Scotton 5, Paolo Bonaldo 4, Hanns Lochmüller 6, Francesco Muntoni 7, Alessandra Ferlini 5 and Cecilia Gelfi 1,2,3/titolo:Muscle proteomics reveals novel insights into the pathophysiological mechanisms of collagen VI myopathies./doi:10.1021%2Fpr500675e/rivista:Journal of proteome research (Print)/anno:2014/pagina_da:11/pagina_a:5022/intervallo_pagine:11–5022/volume:3
Mutations in the collagen VI genes cause the Ullrich congenital muscular dystrophy (UCMD), with severe phenotype, and Bethlem myopathy (BM) with mild to moderate phenotype. Both, UCMD and BM patients show dystrophic features with degeneration/regener
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::269aeadc7891500b8ad69657e5cb93d0
http://hdl.handle.net/11392/2119613
http://hdl.handle.net/11392/2119613