Zobrazeno 1 - 9
of 9
pro vyhledávání: '"Sara, Pruneddu"'
Autor:
Fangyi Xie, Dawn Marie R. Davis, Farah Baban, Emma F. Johnson, Regan N. Theiler, Austin Todd, Sara Pruneddu, Jenny E. Murase, Julia-Tatjana Maul, Christina M. Ambros-Rudolph, Julia S. Lehman
Publikováno v:
Journal of the American Academy of Dermatology.
Autor:
Antonio Galleu, Claudio Fozza, Maria Pina Simula, Salvatore Contini, Patrizia Virdis, Giovanna Corda, Simonetta Pardini, Francesca Cottoni, Sara Pruneddu, Antonio Angeloni, Simona Ceccarelli, Maurizio Longinotti
Publikováno v:
Neoplasia: An International Journal for Oncology Research, Vol 14, Iss 6, Pp 487-494 (2012)
It is widely accepted that a deranged immune system plays a key role in the onset and evolution of classic Kaposi sarcoma (CKS). Nevertheless, the usage of the T-cell receptor (TCR) β-variable (BV) chain repertoire expressed by peripheral blood lymp
Externí odkaz:
https://doaj.org/article/e2babf670e834d54a945fe6388276b2d
Autor:
Maurizio Roberto Longinotti, Antonio Angeloni, Simonetta Pardini, Patrizia Virdis, Simona Ceccarelli, Francesca Cottoni, Giovanna Corda, Sara Pruneddu, Maria Pina Simula, Antonio Galleu, Salvatore Contini, Claudio Fozza
Publikováno v:
Neoplasia: An International Journal for Oncology Research, Vol 14, Iss 6, Pp 487-494 (2012)
It is widely accepted that a deranged immune system plays a key role in the onset and evolution of classic Kaposi sarcoma (CKS). Nevertheless, the usage of the T-cell receptor (TCR) β-variable (BV) chain repertoire expressed by peripheral blood lymp
Autor:
Sami Abdennader, T. Schneiter, Jie Zheng, H. Coignard, Martin Leverkus, Marie-Dominique Vignon-Pennamen, Werner Kempf, B. Cribier, Pascal Reygagne, Bungo Ohyama, Ying-Wei Chen, Druck Reinhardt Druck Basel, K.D. Mertz, Georg Stingl, Christine Bangert, Brigitte Peters, Hirohiko Sueki, K. Kerl, A. Fourtanier, Thomas Bieber, Olivier Join-Lambert, Peter Itin, Hayriye Sarıcaoğlu, Daniele Castiglia, C. Mainetti, A.C. Green, P. Giovanoli, Achim Guettner, P. McBride, Yoko Nagashima, G. Palmedo, I. Kolm, Erwin Kump, Han Tian, W. Kempf, Sara Pruneddu, Stephen K. Tyring, Anne-Sophie Le Guern, M. Schmid, Alexander Rufle, Hélène Guet-Revillet, Masumi Akita, Xavier Nassif, Alessandro Giunta, P. Itin, T. Hunziker, Anne Leflèche, Adam Ferguson, Hakan Turan, Wie Huang, Jean-Paul Ortonne, Sylvain Poirée, Sergio Chimenti, Tetsuya Tsuchida, Michael J. Cork, Florence Ribadeau-Dumas, M. Pfaltz, Sylvie Behillil, M. Pithon, Jens Ulrich, Sophia Weise-Riccardi, R.P. Braun, Zhen-Min Niu, L. Schärer, Francesca Cottoni, Peter Häusermann, Andreas W. Arnold, Harald Gollnick, Anna Pokrywka, M.T. Fernández Figueras, Paul Henri Consigny, Jean-Philippe Jais, J. Kamarashev, Daniela Göppner, Jing Zhang, Tamara Kopp, Patrick Berche, Alessandro Di Stefani, Sylvie Fraitag, Koji Iida, B. Burger, Wen-Tao Yuan, Jacques Thèze, Vincent Jullien, Jean Hatchuel, Thomas A. Luger, Giovanna Floriddia, A. Andrade, Rupert C. Ecker, Giovanna Zambruno, Xiao-Ying Chen, Bettina Burger, M.C.B. Hughes, Bruce Strober, Li-Wei Jin, Arife Oz, Olivier Lortholary, Aude Nassif, Takashi Hashimoto, Satz Mengensatzproduktion, L.E. French, Norito Ishii, H. Kutzner
Publikováno v:
Dermatology. 222:93-96
Publikováno v:
Dermatology. 222:10-14
Dystrophic epidermolysis bullosa (DEB) is a rare, clinically heterogeneous, blistering genodermatosis inherited as either autosomal dominant or recessive trait. All DEB forms are caused by mutations in the COL7A1 gene, which encodes for type VII coll
Publikováno v:
Journal of Clinical & Experimental Dermatology Research.
Clinical Findings A 68- year-old man presented with a skin thickening of the anterior neck started few months earlier. Upper respiratory and digestive tract had already been examined which proved normal. On clinical examination a hyper pigmented mass
Autor:
Sara, Pruneddu, Daniele, Castiglia, Giovanna, Floriddia, Francesca, Cottoni, Giovanna, Zambruno
Publikováno v:
Dermatology (Basel, Switzerland). 222(1)
Dystrophic epidermolysis bullosa (DEB) is a rare, clinically heterogeneous, blistering genodermatosis inherited as either autosomal dominant or recessive trait. All DEB forms are caused by mutations in the COL7A1 gene, which encodes for type VII coll
Autor:
Maria Vittoria, Masala, S, Scapaticci, Carla, Olivieri, Cesare, Pirodda, Maria Antonietta, Montesu, Maria Antonietta, Cuccuru, Sara, Pruneddu, Cesare, Danesino, Decio, Cerimele
Publikováno v:
European journal of dermatology : EJD. 17(3)
Werner syndrome (WS, MIM#277700) is a very rare autosomal recessive disorder. WS clinical signs include altered distribution of subcutaneous fat, juvenile bilateral cataracts, a mask-like face and bird-like nose, trophic ulcers of the feet, diabetes
Autor:
Pruneddu, Sara, Castiglia, Daniele, Floriddia, Giovanna, Cottoni, Francesca, Zambruno, Giovanna
Publikováno v:
Dermatology (10188665); Feb2011, Vol. 222 Issue 1, p10-14, 5p, 2 Color Photographs, 1 Graph