Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Sandra Lazaroski"'
Publikováno v:
Acta microbiologica et immunologica Hungarica. 60(2)
Patients with hyperimmunoglobulin E syndrome (HIES) and chronic granulomatous disease (CGD) have prominently increased immunoglobulin (Ig) E levels. We present a 9-year-old boy with medical history revealing recurrent pneumonia, suppurative lymphaden
Patients with hyperimmunoglobulin E syndrome (HIES) and chronic granulomatous disease (CGD) have prominently increased immunoglobulin (Ig) E levels. We present a 9-year-old boy with medical history revealing recurrent pneumonia, suppurative lymphaden
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::59042e2e93631784788894af342cba66
https://avesis.erciyes.edu.tr/publication/details/d43eb1f1-db89-4248-a9f3-5815828134bd/oai
https://avesis.erciyes.edu.tr/publication/details/d43eb1f1-db89-4248-a9f3-5815828134bd/oai
Autor:
Martina Beck, Josef Pfeilschifter, Meike Boosen, Wasiliki Tsalastra-Greul, Ingrid Fleming, Liliana Schaefer, Ellen Dreieicher, Karl-Friedrich Beck, Sandra Lazaroski
Publikováno v:
Journal of the American Society of Nephrology : JASN. 20(9)
Cytokines and nitric oxide (NO) stimulate rat mesangial cells to synthesize and secrete inflammatory mediators. To understand better the signaling pathways that contribute to this response, we exposed rat mesangial cells to the prototypic inflammator
Autor:
Ivana Stojanovic, Djordje Miljković, Sandra Lazaroski, Tamara Cvjetićanin, Stanislava Stosic-Grujicic
Publikováno v:
Immunology
Interleukin (IL)-17 is a pro-inflammatory cytokine produced by recently described T helper type 17 (Th17) cells, which have critical role in immunity to extracellular bacteria and the pathogenesis of several autoimmune disorders. IL-6 and transformin
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8ceba09ff7b962c5ca1d98d25e4d2bbf
http://ibiss-r.rcub.bg.ac.rs/123456789/1478
http://ibiss-r.rcub.bg.ac.rs/123456789/1478
Autor:
Karin R. Engelhardt, Bernhard Fleckenstein, Ekrem Unal, C. I. Edvard Smith, Dietmar Pfeifer, Mohamed-Ridha Barbouche, Meriem Ben-Ali, Hans J. Stauss, Mohamed Bejaoui, Mirzokhid Rakhmanov, Lamia Borchani, Monia Khemiri, Stuart M. Haslam, Zineb Jouhadi, Khadija Khadir, Imen Ben-Mustapha, Bodo Grimbacher, Karin E. Lundin, A. Charlotta Asplund, Manfred Fliegauf, Uwe Kölsch, Gang Wu, A. Sassi, Sandra Lazaroski, Turkan Patiroglu, Magdalena Dziadzio, Anne Dell, Mats Nilsson, Sellama Nadifi, Khairunnadiya Prayitno, Mehmet Akif Ozdemir, Fethi Mellouli, Andrea Maul-Pavicic, Manuela O. Gustafsson, Helene Kraus, Hermann Eibel, Lotte Moens, Hatice Eke Gungor, Thilo Jakob, Elin Falk-Sörqvist, Alejandro A. Schäffer, Sandra Schaffer, Rebecca Meier, Leila Ben-Khemis, Philipp Henneke
Publikováno v:
The Journal of allergy and clinical immunology, vol 133, iss 5
Journal of Allergy and Clinical Immunology
Journal of Allergy and Clinical Immunology, Elsevier, 2014, 133 (5), pp.1410-9, 1419.e1-13. ⟨10.1016/j.jaci.2014.02.025⟩
Journal of Allergy and Clinical Immunology
Journal of Allergy and Clinical Immunology, Elsevier, 2014, 133 (5), pp.1410-9, 1419.e1-13. ⟨10.1016/j.jaci.2014.02.025⟩
International audience; BACKGROUND: Recurrent bacterial and fungal infections, eczema, and increased serum IgE levels characterize patients with the hyper-IgE syndrome (HIES). Known genetic causes for HIES are mutations in signal transducer and activ