Zobrazeno 1 - 10
of 35
pro vyhledávání: '"Sandhya Annamaneni"'
Publikováno v:
Oman Journal of Ophthalmology, Vol 4, Iss 2, Pp 57-62 (2011)
Background : High myopia caused primarily due to abnormal emmetropization and excessive axial ocular elongation is associated with sight-threatening ocular pathology. Muscular dysfunction of ocular ciliary muscles due to altered intracellular calcium
Externí odkaz:
https://doaj.org/article/54250fdd8fd64828910b5c1f56ad9643
Autor:
Harshitha Yadav Payavula, Deepika Jamadandu, Salomy Velpula, Raghunadha Rao Digumarti, Vishnupriya Satti, Sandhya Annamaneni
Publikováno v:
Asian Pacific Journal of Cancer Prevention. 24:859-865
Autor:
Aleem Ahmed Khan, Sandhya Annamaneni, Shaik Iqbal Ahmed, Mazharuddin Ali Khan, Nagarapu Raju, Sandeep Kumar Vishwakarma, Chandrakala Lakkireddy, Avinash Bardia
Publikováno v:
Cell Mol Bioeng
INTRODUCTION: Human mesenchymal stem cells (hMSCs) holds great promise for managing several clinical conditions. However, the low engraftment efficiency and obscurity to harvest these cells without compromising the cellular viability, structural and
Autor:
null Praveen Kumar Neeli, null Shashikanta Sahoo, null Santosh Karnewar, null Gajalakshmi Singuru, null Sriravali Pulipaka, null Sandhya Annamaneni, null Srigiridhar Kotamraju
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::2d65f1e7cf918cf8ad7de04e828cbdd3
https://doi.org/10.1111/febs.16605/v2/response1
https://doi.org/10.1111/febs.16605/v2/response1
Autor:
Praveen Kumar Neeli, Shashikanta Sahoo, Santosh Karnewar, Gajalakshmi Singuru, Sriravali Pulipaka, Sandhya Annamaneni, Srigiridhar Kotamraju
Publikováno v:
The FEBS journalReferences.
DOT1L, a specific H3K79 methyltransferase, has a tumour-promoting role in various cancers, including triple-negative breast cancer (TNBC). However, the molecular mechanism by which the deregulated DOT1L promotes cancer progression is unclear. Herein,
Autor:
Shaik Iqbal Ahmed, Sandeep Kumar Vishwakarma, Syeda Maliha Fathima, Sandhya Annamaneni, Nagarapu Raju, Aleem Ahmed Khan, Avinash Bardia, Chandrakala Lakkireddy
Publikováno v:
Tissuecell. 73
Introduction The reconstruction/regeneration of human bone injuries/defects represents a crucial challenge due to the lack of suitable bio/immune compatible and implantable biological grafts. The available strategies represent implications of several
Publikováno v:
Indian Journal of Medical and Paediatric Oncology. 40:S70-S76
Context: Chronic myeloid leukemia (CML) is one of the most common hematological malignancies in all populations throughout the world. Even though the pathophysiology of CML was well explained in majority of the studies, the incidence of CML was shown
Publikováno v:
Gene. 701:113-120
We explored genetic susceptibility profile of the South Indian women with a large set of SNPs and tested if the lack of association of type 2 diabetes genes with PCOS, recently observed in a number of studies, holds true for this Indian population an
Significance of ATM Gene Polymorphisms in Chronic Myeloid Leukemia - a Case Control Study from India
Autor:
Manjula Gorre, Nageswara Rao Dunna, Sailaja Kagita, Santhoshirani Nanchari, Sarika Jarjapu, Sugunakar Vuree, Phanni Bhushann Meka, Anuradha Cingeetham, Raghunadharao Digumarti, Sandhya Annamaneni, Vishnupriya Satti, Prajitha Edathara Mohandas
Publikováno v:
Asian Pacific Journal of Cancer Prevention. 17:815-821
Background Development of chronic myeloid leukemia (CML) involves formation of double strand breaks (DSBs) which are initially sensed by the ataxia telangiectasia mutated (ATM) signal kinase to induce a DNA damage response (DDR). Mutations or single
Autor:
Satti, Bhushann Meka P, Srinivasulu Mukta, Sandhya Annamaneni, Triveni B, Sarika Jarjapu, Sandeep Kumar Vishwakarma, Nanchari, Anuradha Cingeetham
Publikováno v:
Tumor Biology. 37:6905-6912
BCL2 (B-cell leukemia/lymphoma 2) gene functions as antiapoptotic regulatory element and known to be associated with tumorigenesis. The SNP-938 (C>A) (rs2279115), located in the inhibitory P2 promoter of the BCL2 gene, influences differential binding