Zobrazeno 1 - 10
of 19
pro vyhledávání: '"Sana Kouba"'
Autor:
Sana Kouba, Nicolas Demaurex
Publikováno v:
Chronic Diseases and Translational Medicine, Vol 10, Iss 4, Pp 263-280 (2024)
Abstract Alterations in cellular calcium (Ca2+) signals have been causally associated with the development and progression of human cancers. Cellular Ca2+ signals are generated by channels, pumps, and exchangers that move Ca2+ ions across membranes a
Externí odkaz:
https://doaj.org/article/12e378082adb4fdab9f70b78d6768e72
Autor:
Debbie O'Reilly, Tim Downing, Sana Kouba, Marie Potier-Cartereau, Declan J. McKenna, Christophe Vandier, Paul Buchanan
Publikováno v:
Data in Brief, Vol 42, Iss , Pp 108143- (2022)
The data generated here in relates to the research article “CaV1.3 enhanced store operated calcium promotes resistance to androgen deprivation in prostate cancer”. A model of prostate cancer (PCa) progression to castration resistance was employed
Externí odkaz:
https://doaj.org/article/c960f9324a014268ae280a04722ca477
Autor:
Delphine Fontaine, Sandy Figiel, Romain Félix, Sana Kouba, Gaëlle Fromont, Karine Mahéo, Marie Potier-Cartereau, Aurélie Chantôme, Christophe Vandier
Publikováno v:
Journal of Lipid Research, Vol 61, Iss 6, Pp 840-858 (2020)
Ether lipids (ELs) are lipids characterized by the presence of either an ether linkage (alkyl lipids) or a vinyl ether linkage [i.e., plasmalogens (Pls)] at the sn1 position of the glycerol backbone, and they are enriched in PUFAs at the sn2 position
Externí odkaz:
https://doaj.org/article/16e50e30cc084062977e19ccaa8d3a36
Autor:
Mohamed Chamlali, Sana Kouba, Lise Rodat-Despoix, Luca Matteo Todesca, Zoltán Pethö, Albrecht Schwab, Halima Ouadid-Ahidouch
Publikováno v:
Cells, Vol 10, Iss 12, p 3487 (2021)
Orai3 calcium (Ca2+) channels are implicated in multiple breast cancer processes, such as proliferation and survival as well as resistance to chemotherapy. However, their involvement in the breast cancer cell migration processes remains vague. In the
Externí odkaz:
https://doaj.org/article/fec36aba3a24467da4ce2e62499c770d
Autor:
Céline Delierneux, Sana Kouba, Santhanam Shanmughapriya, Marie Potier-Cartereau, Mohamed Trebak, Nadine Hempel
Publikováno v:
Cells, Vol 9, Iss 2, p 432 (2020)
Calcium (Ca2+) uptake into the mitochondria shapes cellular Ca2+ signals and acts as a key effector for ATP generation. In addition, mitochondria-derived reactive oxygen species (mROS), produced as a consequence of ATP synthesis at the electron trans
Externí odkaz:
https://doaj.org/article/03e4fe28e2974a29832bc7f13fee354a
Autor:
Julie, Schnipper, Sana, Kouba, Frédéric, Hague, Alban, Girault, Marie-Sophie, Telliez, Stéphanie, Guénin, Ahmed, Ahidouch, Stine Falsig, Pedersen, Halima, Ouadid-Ahidouch
Publikováno v:
Cancers. 14(19)
Pancreatic ductal adenocarcinoma (PDAC) remains one of the most lethal malignancies, with a low overall survival rate of less than 10% and limited therapeutic options. Fluctuations in tumor microenvironment pH are a hallmark of PDAC development and p
Autor:
Julie Schnipper, Sana Kouba, Frédéric Hague, Alban Girault, Pierre Rybarczyk, Marie-Sophie Telliez, Stéphanie Guénin, Riad Tebbakha, Henri Sevestre, Ahmed Ahidouch, Stine Falsig Pedersen, Halima Ouadid-Ahidouch
Publikováno v:
International Journal of Molecular Sciences; Volume 23; Issue 14; Pages: 7923
Schnipper, J, Kouba, S, Hague, F, Girault, A, Rybarczyk, P, Telliez, M-S, Guenin, S, Tebbakha, R, Sevestre, H, Ahidouch, A, Pedersen, S F & Ouadid-Ahidouch, H 2022, ' The TRPC1 Channel Forms a PI3K/CaM Complex and Regulates Pancreatic Ductal Adenocarcinoma Cell Proliferation in a Ca 2+-Independent Manner ', International Journal of Molecular Sciences, vol. 23, no. 14, 7923 . https://doi.org/10.3390/ijms23147923
Schnipper, J, Kouba, S, Hague, F, Girault, A, Rybarczyk, P, Telliez, M-S, Guenin, S, Tebbakha, R, Sevestre, H, Ahidouch, A, Pedersen, S F & Ouadid-Ahidouch, H 2022, ' The TRPC1 Channel Forms a PI3K/CaM Complex and Regulates Pancreatic Ductal Adenocarcinoma Cell Proliferation in a Ca 2+-Independent Manner ', International Journal of Molecular Sciences, vol. 23, no. 14, 7923 . https://doi.org/10.3390/ijms23147923
Dysregulation of the transient receptor canonical ion channel (TRPC1) has been found in several cancer types, yet the underlying molecular mechanisms through which TRPC1 impacts pancreatic ductal adenocarcinoma (PDAC) cell proliferation are incomplet
Publikováno v:
International journal of molecular sciences. 23(18)
In the fight against cancer, therapeutic strategies using cisplatin are severely limited by the appearance of a resistant phenotype. While cisplatin is usually efficient at the beginning of the treatment, several patients endure resistance to this ag
Publikováno v:
International Journal of Molecular Sciences. 23:10687
In the fight against cancer, therapeutic strategies using cisplatin are severely limited by the appearance of a resistant phenotype. While cisplatin is usually efficient at the beginning of the treatment, several patients endure resistance to this ag
Autor:
Sana Kouba, Monique Mathé-Allainmat, Paul-Alain Jaffrès, Christophe Vandier, Muriel Pipelier, Aurélie Chantôme, Jacques Lebreton, Didier Dubreuil, Xuexin Zhang, Marie Potier-Cartereau, Mohamed Trebak, Romain Félix, Julien Braire
Publikováno v:
European Journal of Medicinal Chemistry
European Journal of Medicinal Chemistry, Elsevier, 2020, 186, pp.111854. ⟨10.1016/j.ejmech.2019.111854⟩
European Journal of Medicinal Chemistry, 2020, 186, pp.111854. ⟨10.1016/j.ejmech.2019.111854⟩
European Journal of Medicinal Chemistry, Elsevier, 2020, 186, pp.111854. ⟨10.1016/j.ejmech.2019.111854⟩
European Journal of Medicinal Chemistry, 2020, 186, pp.111854. ⟨10.1016/j.ejmech.2019.111854⟩
International audience; Small Conductance Calcium (Ca 2-)-activated potassium (K-) channels (SKCa) are now proved to be involved in many cancer cell behaviors such as proliferation or migration. The SIG channel isoform was particularly described in b
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4939da4e9cd31fe372dae955ef49c6f8
https://hal.univ-brest.fr/hal-02441673
https://hal.univ-brest.fr/hal-02441673