Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Samuel G. Adediran"'
Autor:
Johannes Tschöp, André Martignoni, Cora K. Ogle, Charles C. Caldwell, Greg Noel, Alice N. Neely, Maria D. Reid, Samuel G. Adediran, Jason D. Gardner
A dysfunctional immune system is known to be part of the pathophysiology after burn trauma. However, reports that support this have used a variety of methods, with numerous variables, to induce thermal injury. We hypothesized that, all other paramete
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7568bb1a524034ab7ff05036e398043d
https://opus.bibliothek.uni-augsburg.de/opus4/frontdoor/index/index/docId/81971
https://opus.bibliothek.uni-augsburg.de/opus4/frontdoor/index/index/docId/81971
Autor:
Holly S. Goetzman, Chad T. Robinson, Lisa G. England, Johannes Tschöp, Cindy M. Cave, Jonathan R. Dattilo, Samuel G. Adediran, Charles C. Caldwell, Derrick J. N. Dauplaise, Kevin R. Kasten
Publikováno v:
American Journal of Physiology-Regulatory, Integrative and Comparative Physiology. 299:R918-R925
Following burn injury, the host is susceptible to bacterial infections normally cleared by healthy patients. We hypothesized that during the systemic immune response that follows scald injury, the host's altered immune status increases infection susc
Publikováno v:
Shock (Augusta, Ga.). 34(4)
The complex immune response associated with sepsis results in a high rate of morbidity and mortality, despite substantial basic science and clinical advances. Most of the research has centered on the innate immune response, in contrast to the adaptiv
Publikováno v:
The FASEB Journal. 22
Autor:
Chad T. Robinson, Jay A. Johannigman, Joseph S. Solomkin, Holly S. Goetzman, Charles C. Caldwell, Kevin R. Kasten, Alison M. Rasper, Alex B. Lentsch, Maria R Reid, Samuel G. Adediran, Cindy M. Cave
Publikováno v:
BMC Immunology, Vol 11, Iss 1, p 4 (2010)
BMC Immunology
BMC Immunology
BackgroundThe immune response to trauma has traditionally been modeled to consist of the systemic inflammatory response syndrome (SIRS) followed by the compensatory anti-inflammatory response syndrome (CARS). We investigated these responses in a homo