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pro vyhledávání: '"Samantha M Adler"'
Autor:
Monica Norcini, Alexandra Sideris, Samantha M Adler, Lourdes A Martin Hernandez, Jin Zhang, Thomas J J Blanck, Esperanza Recio-Pinto
Publikováno v:
Frontiers in Molecular Neuroscience, Vol 9 (2016)
Following injury, primary sensory neurons undergo changes that drive central sensitization and contribute to the maintenance of persistent hypersensitivity. NR2B expression in the Dorsal root ganglia (DRG) has not been previously examined in neuropat
Externí odkaz:
https://doaj.org/article/e5e029344e1c47bbb7a0f67c188ae3b6
Publikováno v:
Neurobiology of Stress, Vol 13, Iss , Pp 100258- (2020)
Cognitive flexibility is a higher-order executive function that requires plasticity in neuronal circuits of the prefrontal cortex. Deficits in cognitive flexibility are prominent in a variety of psychiatric disorders, such as major depression, obsess
Externí odkaz:
https://doaj.org/article/e70badb895bc4f72ac015ace868f6327
Autor:
Samantha M. Adler, Claudia Schmauss
Publikováno v:
Neurobiology of Disease, Vol 94, Iss , Pp 1-9 (2016)
Studies showed that histone deacetylase (HDAC) inhibitors can reverse cognitive deficits found in neurodegenerative disorders and age-related memory decline. However, the role of HDACs in stress-induced cognitive deficits has not been investigated. I
Externí odkaz:
https://doaj.org/article/ee0e3883e9fd4743912b98c55224bdd3
Autor:
Milena Girotti, Elizabeth A. Fucich, David A. Morilak, Sarah E. Bulin, Denisse Paredes, Samantha M. Adler
Publikováno v:
Progress in Neuro-Psychopharmacology and Biological Psychiatry. 85:161-179
Prefrontal cortical executive functions comprise a number of cognitive capabilities necessary for goal directed behavior and adaptation to a changing environment. Executive dysfunction that leads to maladaptive behavior and is a symptom of psychiatri
Autor:
Claudia Schmauss, Samantha M. Adler
Publikováno v:
Neurobiology of Disease, Vol 94, Iss, Pp 1-9 (2016)
Studies showed that histone deacetylase (HDAC) inhibitors can reverse cognitive deficits found in neurodegenerative disorders and age-related memory decline. However, the role of HDACs in stress-induced cognitive deficits has not been investigated. I