Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Salvatore Cenzuales"'
Autor:
Francesco Ottaviani, Bruno Mario Cesana, Pasquale Capaccio, Salvatore Cenzuales, Valeria Cuccarini, Lorenzo Pignataro
Publikováno v:
The Journal of Laryngology & Otology. 119:371-376
Folate metabolism dysregulation may lead to abnormal cell proliferation andpredispose to carcinogenesis by inducing DNA hypomethylation. Folate pathways may be modified by polymorphisms in relevant genes, such as that for methylenetetrahydrofolate re
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8faf8bd862928a2172fb4bef7b28a803
https://doi.org/10.1258/0022215053945831
https://doi.org/10.1258/0022215053945831
Autor:
Alberto Mantovani, Martino Introna, Attilio Rivolta, Giuseppina Picardi, Andrea Basile, Cristian Matteucci, Nadia Polentarutti, Giuseppe Peri, Salvatore Cenzuales
Publikováno v:
European Journal of Immunology. 28:496-501
PTX3 is a prototypic long pentraxin expressed by various cell types, most prominently monocytes and endothelial cells, in response to interleukin-1 (IL-1), tumor necrosis factor (TNF) and bacterial products. In the present report, we show that interf
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::389e1058ec71fc98b4b8479610457019
https://doi.org/10.1002/(sici)1521-4141(199802)28:02<496::aid-immu496>3.0.co
https://doi.org/10.1002/(sici)1521-4141(199802)28:02<496::aid-immu496>3.0.co
Publikováno v:
Cytokine. 9:992-998
Monocyte chemotactic protein-3 (MCP-3) is a C-C chemokine which interacts with the CCR1, CCR2 (MCP-1) and CCR3 receptors and has a distinct spectrum of action, The present study was designed to assess whether mycobacterial components were able to ind
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a825c435bdff47f7cc4a3c3131c71c1f
https://doi.org/10.1006/cyto.1997.0242
https://doi.org/10.1006/cyto.1997.0242
Autor:
Salvatore Cenzuales, Valeria Cuccarini, Umberto Ambrosetti, Bruno Mario Cesana, Alessandro Bottero, Pasquale Capaccio, Lorenzo Pignataro, Enrico Fagnani, Francesco Ottaviani
Publikováno v:
Genetics in Medicine. 7:206-208
Purpose: Sudden hearing loss (SHL) can be caused by vascular disorders favoring impaired cochlear perfusion. A number of inherited prothrombotic risk factors have been considered in the pathogenesis of vascular impairment and the possible role of gen
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::f722993e308f2213f558d5c2f0e8a3a7
https://doi.org/10.1097/01.gim.0000157817.92509.45
https://doi.org/10.1097/01.gim.0000157817.92509.45
Autor:
Valeria Cuccarini, Umberto Ambrosetti, Bruno Mario Cesana, Pasquale Capaccio, Lorenzo Pignataro, Alessandro Bottero, Salvatore Cenzuales, Enrico Fagnani, Francesco Ottaviani
Sudden hearing loss (SHL) can be caused by vascular disorders favoring impaired cochlear perfusion. Several inherited prothrombotic risk factors have been considered in the pathogenesis of vascular impairment, and the possible role of genetic alterat
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0fdb7632bb518b4905c743fa7ba3ad8b
http://hdl.handle.net/11379/19972
http://hdl.handle.net/11379/19972
Publikováno v:
Cytokine. 6(2)
We investigated the influence of IL-4 on the interaction between Natural Killer (NK) cells and vascular endothelial cells (EC). Pretreatment of NK cells with IL-4 inhibited the adhesion of NK cells on resting or IL-1-activated EC. The inhibitory acti
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::17f83aee39dcdbb79efe19ff35dfe656
https://pubmed.ncbi.nlm.nih.gov/7913345
https://pubmed.ncbi.nlm.nih.gov/7913345
Autor:
Cinzia Calzarossa, Mauro Moroni, Edoardo Rossi, Salvatore Cenzuales, Alessia Stellari, Carlo Parravicini, Giovanna Bestetti, Massimo Galli, Antonella Lattuada, Mario Corbellino
Publikováno v:
Karolinska Institutet
Scopus-Elsevier
Scopus-Elsevier
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8f6f3d09bb5e6a2f1bf3e4a4831746db
http://www.ncbi.nlm.nih.gov/pubmed/14597993
http://www.ncbi.nlm.nih.gov/pubmed/14597993