Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Sai Krishna Athuluri Divakar"'
Autor:
Yolanda Garcia-Carmona, Adrian T. Ting, Lin Radigan, Sai Krishna Athuluri Divakar, Jose Chavez, Eric Meffre, Andrea Cerutti, Charlotte Cunningham-Rundles
Publikováno v:
Frontiers in Immunology, Vol 10 (2019)
Externí odkaz:
https://doaj.org/article/86652938ac8642a59c01677dab04cdb8
Autor:
Yolanda Garcia-Carmona, Adrian T. Ting, Lin Radigan, Sai Krishna Athuluri Divakar, Jose Chavez, Eric Meffre, Andrea Cerutti, Charlotte Cunningham-Rundles
Publikováno v:
Frontiers in Immunology, Vol 9 (2018)
TACI signals activate B cell proliferation, isotype switch and antibody production in both normal immunity and autoimmune states. In contrast to murine TACI, the human TACI gene undergoes alternative splicing to produce short and long isoforms (TACI-
Externí odkaz:
https://doaj.org/article/923d496975e9445f9bb0681751cdeacf
Publikováno v:
Molecular and Translational Medicine ISBN: 9783030215392
Chronic fibrotic liver disease caused by viral or metabolic etiologies is a high-risk condition for developing hepatocellular carcinoma (HCC). Even after complete HCC tumor resection or ablation, the carcinogenic tissue microenvironment in the remnan
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::02c18c920411b574295e3351fde9ee28
https://doi.org/10.1007/978-3-030-21540-8_1
https://doi.org/10.1007/978-3-030-21540-8_1
Autor:
Rodrigo Vasquez-Del Carpio, E. Premkumar Reddy, Jonathan R. Hart, Yogesh K. Gupta, Lynn Ueno, Stephen C. Cosenza, Kaushik Dutta, Peter K. Vogt, David J. Mulholland, M. V. Ramana Reddy, Chandan Guha, Sai Krishna Athuluri-Divakar, Indranil Basu, Stacey J. Baker, Aneel K. Aggarwal
Publikováno v:
Cell. 165:643-655
Oncogenic activation of RAS genes via point mutations occurs in 20%-30% of human cancers. The development of effective RAS inhibitors has been challenging, necessitating new approaches to inhibit this oncogenic protein. Functional studies have shown
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c493e4550e12f25b3cdb996fcd0828b9
https://doi.org/10.1016/j.cell.2016.03.045
https://doi.org/10.1016/j.cell.2016.03.045
Publikováno v:
Annals of the New York Academy of Sciences
Chronic fibrotic liver disease caused by viral or metabolic etiologies is a high-risk condition for developing hepatocellular carcinoma (HCC). Even after curative treatment of early-stage HCC tumor, the carcinogenic microenvironment persists in the r
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6f74402dbb6c580c3e575721086627a1