Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Sagita Mega Sekar, Kencana"'
Autor:
Sagita Mega Sekar Kencana, Nur Arfian, Ratih Yuniartha, Ramadhea Laila Afifa An-Nur Willya Saputri, Fauziyatul Munawaroh, Dwi Cahyani Ratna Sari
Publikováno v:
Iranian Journal of Medical Sciences, Vol 49, Iss 2, Pp 110-120 (2024)
Background: Chlorogenic acid (CGA) is known to have antifibrotic and hypoglycemic effects and may play a role in preventing diabetes-induced pulmonary fibrosis. This study aimed to determine the effect and optimum dose of CGA on diabetes-induced pulm
Externí odkaz:
https://doaj.org/article/db6341692c08475eb9dff1343877033a
Autor:
Ramadhea Laila Afifa An-Nur Willya SAPUTRI, Nur ARFIAN, Junaedy YUNUS, Fauziyatul MUNAWAROH, Sagita Mega Sekar KENCANA, Dwi Cahyani Ratna SARI
Publikováno v:
Turkish Journal of Medical Sciences; 2024, Vol. 54 Issue 4, p866-875, 10p
Autor:
Gunadi, Raedi Ardlo Luzman, Sagita Mega Sekar Kencana, Bhagas Dwi Arthana, Fauzan Ahmad, Ganjar Sulaksmono, Agitha Swandaru Rastaputra, Golda Puspa Arini, Ririd Tri Pitaka, Andi Dwihantoro, Akhmad Makhmudi
Publikováno v:
Frontiers in Pediatrics, Vol 9 (2021)
Background: Hirschsprung-associated enterocolitis (HAEC) is a major contributor in the mortality of Hirschsprung disease (HSCR) patients that can occur both preoperatively and post-operatively. Several cut-off values of HAEC score have been used, i.e
Externí odkaz:
https://doaj.org/article/1c89ef940f7e4804a2f7434b21608290
Autor:
Gunadi, Alvin Santoso Kalim, Estelita Liana, Aditya Rifqi Fauzi, Dian Nirmala Sirait, Dwiki Afandy, Sagita Mega Sekar Kencana, Eko Purnomo, Kristy Iskandar, Akhmad Makhmudi
Publikováno v:
BMC Pediatrics, Vol 19, Iss 1, Pp 1-5 (2019)
Abstract Background Recently, pathogenic alleles within ubiquitin N-recognin domain-containing E3 ligase 4 (UBR4) gene have been shown to be associated with Hirschsprung disease (HSCR). We determined the UBR4 expressions in Indonesian HSCR patients.
Externí odkaz:
https://doaj.org/article/2d45e3933d214c868c01319f0a9ed448
Autor:
Shiefa Annisa, Qisthi, Daniel Simada Pandapotan, Saragih, David Wijaya, Sutowo, Dian Nirmala, Sirait, Priscillia, Imelda, Sagita Mega Sekar, Kencana, Akhmad, Makhmudi, Gunadi
Publikováno v:
Kobe J Med Sci
Biliary atresia (BA) is a progressive obstruction and fibro-obliteration of the extrahepatic and intrahepatic biliary tract that causes cholestatic jaundice in infants, resulting in biliary cirrhosis and even death in the first year of life if the Ka
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid_dedup__::28389546ac71c55d6a133767d284c45c
https://hdl.handle.net/20.500.14094/81012555
https://hdl.handle.net/20.500.14094/81012555
Autor:
Akhmad Makhmudi, Dian Nirmala Sirait, Eko Purnomo, Estelita Liana, Aditya Rifqi Fauzi, Sagita Mega Sekar Kencana, Gunadi, Dwiki Afandy, Kristy Iskandar, Alvin Santoso Kalim
Publikováno v:
BMC Pediatrics
BMC Pediatrics, Vol 19, Iss 1, Pp 1-5 (2019)
BMC Pediatrics, Vol 19, Iss 1, Pp 1-5 (2019)
Background Recently, pathogenic alleles within ubiquitin N-recognin domain-containing E3 ligase 4 (UBR4) gene have been shown to be associated with Hirschsprung disease (HSCR). We determined the UBR4 expressions in Indonesian HSCR patients. Methods W
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ded0eafd3910b06c819b4dab99d619a1
http://europepmc.org/articles/PMC6907357
http://europepmc.org/articles/PMC6907357
Autor:
Devi Yuliana Suhardi, Edison Hery Susanto, Muhammad Mansyur Romi, Santosa Budiharjo, Bianda Dwida Pramudita, Dwi Cahyani Ratna Sari, Nur Arfian, Sagita Mega Sekar Kencana
Publikováno v:
2018 1st International Conference on Bioinformatics, Biotechnology, and Biomedical Engineering - Bioinformatics and Biomedical Engineering.
Introduction: Kidney fibrosis lead to myofibroblast formation with vascular remodeling and activation of Endothelin-1 (ET-1) and eNOS pathways. Vitamin D has reno-protective effect, however its role in ET-1 and eNOS pathways haven't been elucidated y
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::03fc14171dd9dfb8dfbf2386d3227433
https://doi.org/10.1109/biomic.2018.8610552
https://doi.org/10.1109/biomic.2018.8610552