Zobrazeno 1 - 10
of 24
pro vyhledávání: '"Safiyyah Ziyad"'
Autor:
Susan Bal, Jesus Berdeja, Myo Htut, Mehmet Kocoglu, Tara Gregory, Larry D. Anderson, Adriana Rossi, Daniel Egan, Luciano Costa, Lisa Kelly, Safiyyah Ziyad, Hongxiang Hu, Yanping Chen, Allison J. Kaeding, Michael Burgess, Kristen Hege, Omar Nadeem
Publikováno v:
HemaSphere, Vol 7, p e9863287 (2023)
Externí odkaz:
https://doaj.org/article/a86095477e5c4c2dae2c62f38d0b86d1
Autor:
Safiyyah Ziyad, Jesse D. Riordan, Ann M. Cavanaugh, Trent Su, Gloria E. Hernandez, Georg Hilfenhaus, Marco Morselli, Kristine Huynh, Kevin Wang, Jau-Nian Chen, Adam J. Dupuy, M. Luisa Iruela-Arispe
Publikováno v:
Cell Reports, Vol 22, Iss 5, Pp 1211-1224 (2018)
Summary: Given its role as the source of definitive hematopoietic cells, we sought to determine whether mutations initiated in the hemogenic endothelium would yield hematopoietic abnormalities or malignancies. Here, we find that endothelium-specific
Externí odkaz:
https://doaj.org/article/a74c22f7c9d148fc88f5c03dbb005087
Autor:
James E Korkola, Eric A Collisson, Laura M Heiser, Chris Oates, Nora Bayani, Sleiman Itani, Amanda Esch, Wallace Thompson, Obi L Griffith, Nicholas J Wang, Wen-Lin Kuo, Brian Cooper, Jessica Billig, Safiyyah Ziyad, Jenny L Hung, Lakshmi Jakkula, Heidi Feiler, Yiling Lu, Gordon B Mills, Paul T Spellman, Claire Tomlin, Sach Mukherjee, Joe W Gray
Publikováno v:
PLoS ONE, Vol 12, Iss 10, p e0186551 (2017)
[This corrects the article DOI: 10.1371/journal.pone.0133219.].
Externí odkaz:
https://doaj.org/article/f64d8adeba2f456294eef1b1123c9d8c
Autor:
James E Korkola, Eric A Collisson, Laura Heiser, Chris Oates, Nora Bayani, Sleiman Itani, Amanda Esch, Wallace Thompson, Obi L Griffith, Nicholas J Wang, Wen-Lin Kuo, Brian Cooper, Jessica Billig, Safiyyah Ziyad, Jenny L Hung, Lakshmi Jakkula, Heidi Feiler, Yiling Lu, Gordon B Mills, Paul T Spellman, Claire Tomlin, Sach Mukherjee, Joe W Gray
Publikováno v:
PLoS ONE, Vol 10, Iss 7, p e0133219 (2015)
We report here on experimental and theoretical efforts to determine how best to combine drugs that inhibit HER2 and AKT in HER2(+) breast cancers. We accomplished this by measuring cellular and molecular responses to lapatinib and the AKT inhibitors
Externí odkaz:
https://doaj.org/article/01e6b6cfbd714bc98c36f3440a8d060c
Autor:
Susan Bal, M. Hakan Kocoglu, Omar Nadeem, Myo Htut, Tara Gregory, Larry D. Anderson, Luciano J. Costa, Tonia J. Buchholz, Safiyyah Ziyad, Meng Li, Yanping Chen, Allison J. Kaeding, Michael R. Burgess, Kristen Hege, Jesus Berdeja
Publikováno v:
Blood. 140:883-885
Autor:
Marco Morselli, M. Luisa Iruela-Arispe, Kevin Wang, Georg Hilfenhaus, Gloria Hernandez, Ann M. Cavanaugh, Adam J. Dupuy, Jesse D. Riordan, Kristine Huynh, Jau-Nian Chen, Trent Su, Safiyyah Ziyad
Publikováno v:
Cell Reports, Vol 22, Iss 5, Pp 1211-1224 (2018)
Ziyad, Safiyyah; Riordan, Jesse D; Cavanaugh, Ann M; Su, Trent; Hernandez, Gloria E; Hilfenhaus, Georg; et al.(2018). A Forward Genetic Screen Targeting the Endothelium Reveals a Regulatory Role for the Lipid Kinase Pi4ka in Myelo-and Erythropoiesis. Cell Reports, 22(5), 1211-1224. doi: 10.1016/j.celrep.2018.01.017. UC Office of the President: Research Grants Program Office (RGPO). Retrieved from: http://www.escholarship.org/uc/item/4r45f0n3
Cell reports, vol 22, iss 5
Cell reports
Ziyad, Safiyyah; Riordan, Jesse D; Cavanaugh, Ann M; Su, Trent; Hernandez, Gloria E; Hilfenhaus, Georg; et al.(2018). A Forward Genetic Screen Targeting the Endothelium Reveals a Regulatory Role for the Lipid Kinase Pi4ka in Myelo-and Erythropoiesis. Cell Reports, 22(5), 1211-1224. doi: 10.1016/j.celrep.2018.01.017. UC Office of the President: Research Grants Program Office (RGPO). Retrieved from: http://www.escholarship.org/uc/item/4r45f0n3
Cell reports, vol 22, iss 5
Cell reports
SUMMARY Given its role as the source of definitive hematopoietic cells, we sought to determine whether mutations initiated in the hemogenic endothelium would yield hematopoietic abnormalities or malignancies. Here, we find that endothelium-specific t
Autor:
Esra Güç, Safiyyah Ziyad, Michele De Palma, Ryan D. Freshman, Mario Leonardo Squadrito, Julia J. Mack, Witold W. Kilarski, Huanhuan He, Caroline Baer, Carmen M. Warren, M. Luisa Iruela-Arispe, Austin I. McDonald, Melody A. Swartz
Publikováno v:
He, H; Mack, JJ; Güç, E; Warren, CM; Squadrito, ML; Kilarski, WW; et al.(2016). Perivascular Macrophages Limit Permeability. Arteriosclerosis, Thrombosis, and Vascular Biology, 36(11), 2203-2212. doi: 10.1161/ATVBAHA.116.307592. UCLA: Retrieved from: http://www.escholarship.org/uc/item/2mq001hp
Arteriosclerosis, thrombosis, and vascular biology, vol 36, iss 11
Arteriosclerosis thrombosis and vascular biology
Arteriosclerosis, thrombosis, and vascular biology, vol 36, iss 11
Arteriosclerosis thrombosis and vascular biology
Objective— Perivascular cells, including pericytes, macrophages, smooth muscle cells, and other specialized cell types, like podocytes, participate in various aspects of vascular function. However, aside from the well-established roles of smooth mu
Autor:
Sham Mailankody, Andrzej Jakubowiak, John Byon, Kelvin P. Lee, Julia Piasecki, William I. Bensinger, Safiyyah Ziyad, Myo Htut, Michelle L Blake, Todd Devries
Publikováno v:
Blood. 132:957-957
Introduction: B-cell maturation antigen (BCMA) is expressed on malignant plasma cells and is an attractive therapeutic target for multiple myeloma. BCMA CAR T-cells, antibody drug conjugates and bispecific T-cell engagers have demonstrated substantia
Autor:
M. Luisa Iruela-Arispe, Safiyyah Ziyad
Publikováno v:
Genes & Cancer. 2:1085-1096
Tumors have been recently recognized as aberrant organs composed of a complex mixture of highly interactive cells that in addition to the cancer cell include stroma (fibroblasts, adipocytes, and myofibroblasts), inflammatory (innate and adaptive immu
Autor:
Shamra Martin, Hung Vo, Jefferson Davis, Joe W. Gray, Kimberly Samayoa, Kenneth W. Wood, Sylvaine Cases, Heidi S. Feiler, Peter Bean, Wen Lin Kuo, Mamatha Reddy, Karen Thomsen, James W. Purcell, Safiyyah Ziyad, Jessica Billig
Publikováno v:
Clinical Cancer Research. 16:566-576
Purpose: Ispinesib (SB-715992) is a potent inhibitor of kinesin spindle protein, a kinesin motor protein essential for the formation of a bipolar mitotic spindle and cell cycle progression through mitosis. Clinical studies of ispinesib have shown a 9