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Autor:
Sadeleer, L.J. de, Verleden, S.E., Schupp, J.C., McDonough, J.E., Goos, T., Yserbyt, J., Bargagli, E., Rottoli, P., Kaminski, N., Prasse, Antje, Wuyts, W.A.
Background: Given the plethora of pathophysiologic mechanisms described in idiopathic pulmonary fibrosis (IPF), we hypothesize that the mechanisms driving fibrosis in IPF may be different from one patient to another. Research Question: Do IPF endotyp
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=od_______610::0e2bb67e63c175abfec7a1d537fee888
https://publica.fraunhofer.de/handle/publica/430208
https://publica.fraunhofer.de/handle/publica/430208