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Inhibition of CD226 Co-Stimulation Suppresses Diabetes Development in the NOD Mouse by Augmenting Tregs and Diminishing Effector T Cell Function.

Autor: Brown ME; Diabetes Institute, College of Medicine, University of Florida, Gainesville, FL 32610.; Department of Pathology, Immunology, and Laboratory Medicine, College of Medicine, University of Florida, Gainesville, FL 32610., Thirawatananond P; Diabetes Institute, College of Medicine, University of Florida, Gainesville, FL 32610.; Department of Pathology, Immunology, and Laboratory Medicine, College of Medicine, University of Florida, Gainesville, FL 32610., Peters LD; Diabetes Institute, College of Medicine, University of Florida, Gainesville, FL 32610.; Department of Pathology, Immunology, and Laboratory Medicine, College of Medicine, University of Florida, Gainesville, FL 32610., Kern EJ; Diabetes Institute, College of Medicine, University of Florida, Gainesville, FL 32610.; Department of Pathology, Immunology, and Laboratory Medicine, College of Medicine, University of Florida, Gainesville, FL 32610., Vijay S; Diabetes Institute, College of Medicine, University of Florida, Gainesville, FL 32610.; Department of Pathology, Immunology, and Laboratory Medicine, College of Medicine, University of Florida, Gainesville, FL 32610., Sachs LK; Diabetes Institute, College of Medicine, University of Florida, Gainesville, FL 32610.; Department of Pathology, Immunology, and Laboratory Medicine, College of Medicine, University of Florida, Gainesville, FL 32610., Posgai AL; Diabetes Institute, College of Medicine, University of Florida, Gainesville, FL 32610.; Department of Pathology, Immunology, and Laboratory Medicine, College of Medicine, University of Florida, Gainesville, FL 32610., Brusko MA; Diabetes Institute, College of Medicine, University of Florida, Gainesville, FL 32610.; Department of Pathology, Immunology, and Laboratory Medicine, College of Medicine, University of Florida, Gainesville, FL 32610., Shapiro MR; Diabetes Institute, College of Medicine, University of Florida, Gainesville, FL 32610.; Department of Pathology, Immunology, and Laboratory Medicine, College of Medicine, University of Florida, Gainesville, FL 32610., Mathews CE; Diabetes Institute, College of Medicine, University of Florida, Gainesville, FL 32610.; Department of Pathology, Immunology, and Laboratory Medicine, College of Medicine, University of Florida, Gainesville, FL 32610., Bacher R; Diabetes Institute, College of Medicine, University of Florida, Gainesville, FL 32610.; Department of Biostatistics, College of Public Health and Health Professions, University of Florida, Gainesville, FL 32610., Brusko TM; Diabetes Institute, College of Medicine, University of Florida, Gainesville, FL 32610.; Department of Pathology, Immunology, and Laboratory Medicine, College of Medicine, University of Florida, Gainesville, FL 32610.; Department of Pediatrics, College of Medicine, University of Florida, Gainesville, FL 32610.; Department of Biochemistry and Molecular Biology, College of Medicine, University of Florida, Gainesville, FL 32610.

Publikováno v: BioRxiv : the preprint server for biology [bioRxiv] 2024 Jul 19. Date of Electronic Publication: 2024 Jul 19.