Zobrazeno 1 - 10
of 30
pro vyhledávání: '"Sachihiko Yokoya"'
Autor:
Yasukazu Hozumi, Choichiro Tase, Yuxiang Zhang, Akio Wanaka, Seita Hagino, Kaoru Goto, Ken Iseki, Tetsuji Mori, Sachihiko Yokoya, Nobuko Sakai
Publikováno v:
Archives of Histology and Cytology. 74:1-7
Publikováno v:
The FASEB Journal. 21:4087-4094
Three-hydroxy-3-methyl-glutaryl-CoA (HMG-CoA) reductase inhibitors, known as statins, induce skeletal muscle injury including myalgia, myositis, and rhabdomyolysis. The mechanism of this myotoxicity remains unknown. This study examined the effect of
Autor:
Sachihiko Yokoya, Ken Iseki, Seita Hagino, Tsuyoshi Hikake, Shinichi Kikuchi, Akio Wanaka, Yuxiang Zhang, Tetsuji Mori, Nobuko Sakai
Publikováno v:
Neuroscience Letters. 349:29-32
Glypican-1, a heparan sulfate proteoglycan, is expressed in various tissues including developing and postnatal central nervous system. It serves as a receptor for heparin-binding molecules such as fibroblast growth factors (FGFs). We investigated whe
Autor:
Akio Wanaka, Sachihiko Yokoya, Yuxiang Zhang, Ken Iseki, Hiromi Takagi, Seita Hagino, Tetsuji Mori, Tsuyoshi Hikake
Publikováno v:
Anatomy and Embryology. 206:373-380
The transcription factor OASIS gene, which encodes for a CREB/ATF family member, is specifically expressed in the salivary gland, the cartilage and the tooth germs of the mouse embryo. In the present study, the expression patterns were compared betwe
Autor:
Tetsuji Mori, Mayumi Takeuchi, Ken Iseki, Akio Wanaka, Shinichi Kikuchi, Seita Hagino, Sachihiko Yokoya, Takuya Nikaido, Junko Takeda, Hiromi Takaki, Yuxiang Zhang
Publikováno v:
Molecular Brain Research. 108:129-138
We reported the expression patterns of a novel member of the CREB/ATF family, OASIS, in central nervous system (CNS) lesions and its transcriptional activity. OASIS gene expression was upregulated in the stab-injured spinal cord. Double labeling expe
Autor:
Masahiro Murakawa, Sachihiko Yokoya, Hiromi Takaki, Ken Iseki, Yuxiang Zhang, Tetsuji Mori, Mayumi Takeuchi, Seita Hagino, Akio Wanaka
Publikováno v:
Orthodontics & Craniofacial Research. 5:65-70
Structured Abstract Authors– Zhang Y, Mori T, Takaki H, Takeuchi M, Iseki K, Hagino S, Murakawa M, Yokoya S, Wanaka A. Objectives– To compare and contrast the gene expression of two LIM-homeobox type transcription factors, Lhx6 and L3/Lhx8, in se
Autor:
Atsushi Irisawa, Katsutoshi Obara, Akio Wanaka, Toshimitsu Suzuki, Masayuki Miyata, Tetsuji Mori, Sachihiko Yokoya, Reiji Kasukawa, Hidenori Kiyosawa, Teizo Fujita, Yukio Sato, Junko Takeda
Publikováno v:
Cellular Immunology. 204:11-18
Endogenous retroviral gene products have been recognized as being expressed in human cancerous tissues. However, these products have not been shown to be antigenic targets for T-cells, possibly due to immune tolerance. Since carcinogen-induced colon
Autor:
Yutaka Honma, Yoshihiro Tanno, Junko Takeda, Michiko Tojo, Sachihiko Yokoya, Teiji Yamamoto, Akio Wanaka, Kenya Kanazawa, Tetsuji Mori, Takuya Nikaido
Publikováno v:
Journal of Chemical Neuroanatomy. 17:99-107
Huntingtin-interacting protein-2 (Hip-2) was identified as a human protein specifically associated with huntingtin in vitro, a gene product affected in patients with Huntington disease (HD). It is a ubiquitin-conjugating enzyme identical to the previ
Autor:
Kenya Kanazawa, Fumio Kaneko, Yoshihiro Tanno, Yutaka Honma, Michiko Tojo, Hidenori Kiyosawa, Tetsuji Mori, Sachihiko Yokoya, Akio Wanaka
Publikováno v:
Pathology International. 49:687-694
In basal cell nevus syndrome (BCNS) patients, mutations of a gene, patched (ptc), which encodes a putative signal transducer of sonic hedgehog protein (SHH), were found and are thought to be one of the major causes of BCNS. The SHH signaling pathway
Autor:
Kenya Kanazawa, Takuya Nikaido, Tetsuji Mori, Tetsuro Tsukamoto, Michiko Tojo, Akio Wanaka, Yutaka Honma, Yoshihiro Tanno, Sachihiko Yokoya, Junko Takeda, Hidenori Kiyosawa
Publikováno v:
Molecular Brain Research. 69:93-103
Gliosis is a characteristic response of astrocytes to inflammation and trauma of the central nervous system (CNS). To study the mechanisms underlying gliosis, we performed differential display screening for genes specifically induced in long-term cul