Zobrazeno 1 - 10
of 20
pro vyhledávání: '"Sabry A. Shoeib"'
Publikováno v:
The Egyptian Journal of Internal Medicine, Vol 33, Iss 1, Pp 1-9 (2021)
Abstract Background Autoimmunity is used to cause by impairment of adaptive immunity alone, whereas autoinflammatory was originally defined as a consequence of unregulated innate immunity. So, the pathogenetic mechanisms of autoimmune diseases were w
Externí odkaz:
https://doaj.org/article/22aa8ca168bd4ca0ac9a4f535d96e2d9
Autor:
Mohamed Ahmed Abdel Hafez, Mahmoud Salah Abdel Salam, Sabry Abdullah Shoeib, Sara Gamal El Meligy, Rania Mohamed Afifi
Publikováno v:
The Egyptian Journal of Internal Medicine, Vol 36, Iss 1, Pp 1-9 (2024)
Abstract Background Diffuse large B-cell lymphoma (DLBCL) is the most common aggressive non-Hodgkin lymphoma (NHL), constituting 31% of all NHL. Malignancy and inflammation are closely linked. Inflammatory processes have been identified to play an im
Externí odkaz:
https://doaj.org/article/f856eb3b89184194a16c428ff1c4ea53
Publikováno v:
The Egyptian Journal of Internal Medicine, Vol 31, Iss 4, Pp 389-396 (2019)
Introduction Extracellular vesicles (EV) have emerged as important ‘nanoshuttles’ of information between cells, carrying proteins, genetic information, and bioactive lipids to modify the phenotype and function of recipient cells. EVs are potentia
Externí odkaz:
https://doaj.org/article/dc1650b978704517a638a2e00889c4b8
Publikováno v:
The Egyptian Journal of Hospital Medicine. 84:2599-2604
Background: Behcet’s syndrome (BS) is a systemic inflammatory vasculitis of unknown etiology, characinoid by recurrent oral, genital ulcers and uveitis. Its symptoms may affect patients’ quality of life. Objective: This study aimed to evaluate th
Publikováno v:
The Egyptian Journal of Hospital Medicine. 82:96-100
Background: Acute graft-versus-host disease (GVHD) following allogeneic hematopoietic stem cell transplant (AHscT) is an immune triggered process, leading to profound immune dysregulation and organ dysfunction. Three phases summarize the development
Autor:
Ayman Fathy Abd El-Halim, Sabry A Shoeib, Mohamad Ahmad Abd ElHafez, Essam A. Abd Elmohsen, Ashraf Dawod, Sameh Abo ElNasr Abd ElSalam ElBaz
Publikováno v:
The Egyptian Journal of Hospital Medicine. 80:820-826
Background: Sepsis is a complicated syndrome resulting from the inappropriate expression of host factors in response to infection and is a major cause of death in patients that are hospitalized in emergency departments and in critical care units. Obj
Autor:
Sabry A Shoeib, Ashraf Dawod, Aida Nazir, Essam A Abdelmohsen, Hanan M Bedair, Alaa Efat, Walaa Elgheriany
Publikováno v:
Clinical and Applied Thrombosis/Hemostasis
Clinical and Applied Thrombosis/Hemostasis, Vol 27 (2021)
Clinical and Applied Thrombosis/Hemostasis, Vol 27 (2021)
Immune thrombocytopenia (ITP) appears to be a heterogeneous disease. In some patients, autoimmunity may be associated with an inflammatory process, and in other patients, low platelets may interfere with other aspects of the coagulation system. Eithe
Autor:
Mohamad Ahmad Abd ElHafez, Alaa Efat, Hany F Eladly, Essam A. Abd Elmohsen, Reda A Ibrahim, Ayman F Arafa, Sabry A Shoeib, Ashraf Dawod, Aly M El-Kholy
Publikováno v:
International Journal of Immunopathology and Pharmacology
Background: Sepsis has been redefined recently as life-threatening organ dysfunction caused by dysregulated host responses to infection and septic shock. Soluble urokinase plasminogen activator receptor (SuPAR) and plasminogen activator inhibitor-1(P
Publikováno v:
Open Journal of Internal Medicine. 10:112-120
Introduction: Antiphospholipid antibody syndrome is a complex autoimmune disease that can affect all systems of the body and characterized by presence of antiphospholipid antibodies that cause vascular thrombosis and pregnancy complications, kidney i
Publikováno v:
The Egyptian Journal of Internal Medicine, Vol 33, Iss 1, Pp 1-9 (2021)
Background Autoimmunity is used to cause by impairment of adaptive immunity alone, whereas autoinflammatory was originally defined as a consequence of unregulated innate immunity. So, the pathogenetic mechanisms of autoimmune diseases were well-thoug