Zobrazeno 1 - 3
of 3
pro vyhledávání: '"Sabrina Oesteritz"'
Autor:
Sabrina Oesteritz, Wymke Ockenga, Waltraud Ackermann, Christopher Horst Lillig, José R. Godoy, Eva-Maria Hanschmann
Publikováno v:
Free Radical Biology and Medicine. 51:552-561
The disruption of redox control, i.e., oxidative stress, is one of the most destructive causes of ischemia-reperfusion (IR) injury. Thioredoxin (Trx) family proteins play a major role in the cellular response to oxidative stress. Here, we systematica
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e96b7d15ccc298f2ed79b66323ae9f45
https://doi.org/10.1016/j.freeradbiomed.2011.04.036
https://doi.org/10.1016/j.freeradbiomed.2011.04.036
Autor:
Sabrina Oesteritz, Maria Funke, José R. Godoy, Christopher Horst Lillig, Waltraud Ackermann, Hans-Peter Elsässer, Francisco Capani, Petra Haunhorst
Publikováno v:
Biochimica et Biophysica Acta (BBA) - General Subjects. 1810:2-92
Background Oxidoreductases of the thioredoxin family of proteins have been thoroughly studied in numerous cellular and animal models mimicking human diseases. Despite of their well documented role in various disease conditions, no systematic informat
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::066a16335e1a99c976b138723c43fed8
https://doi.org/10.1016/j.bbagen.2010.05.006
https://doi.org/10.1016/j.bbagen.2010.05.006
Autor:
José Rodrigo, Godoy, Maria, Funke, Waltraud, Ackermann, Petra, Haunhorst, Sabrina, Oesteritz, Francisco, Capani, Hans-Peter, Elsässer, Christopher Horst, Lillig
Publikováno v:
Biochimica et biophysica acta. 1810(1)
Oxidoreductases of the thioredoxin family of proteins have been thoroughly studied in numerous cellular and animal models mimicking human diseases. Despite of their well documented role in various disease conditions, no systematic information on the
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::30b0583ae755e24b6742835b09f98ee3
https://pubmed.ncbi.nlm.nih.gov/20682242
https://pubmed.ncbi.nlm.nih.gov/20682242