Zobrazeno 1 - 8
of 8
pro vyhledávání: '"Sabine Wolgast"'
Publikováno v:
Cell and Tissue Research. 355:437-446
The neurohormone melatonin is synthesized by the pineal gland under the stimulation of norepinephrine (NE). Its synthesis is inhibited by acetylcholine (ACh) and by insulin. Type 2 diabetic Goto Kakizaki (GK) rats have higher insulin and lower melato
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a4e172210b92394e2cd236a523fd9c45
https://doi.org/10.1007/s00441-013-1748-x
https://doi.org/10.1007/s00441-013-1748-x
Publikováno v:
Molecular and Cellular Endocrinology. 365:129-138
The retinoic-acid-related receptor family of orphan receptors (RORs) act as transcriptional activators or repressors. One of their functions involves integrated actions within circadian oscillators, particularly of the periphery. The present paper de
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ef49239c689ad755d18730fe9bc4c782
https://doi.org/10.1016/j.mce.2012.10.001
https://doi.org/10.1016/j.mce.2012.10.001
Publikováno v:
Journal of Pineal Research. 53:344-357
The pineal hormone melatonin exerts its influence on the insulin secretion of pancreatic islets by a variety of signalling pathways. The purpose of the present study was to analyse the impact of melatonin on the phosphorylated transcription factor cA
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::f5a303f0a1b4d555264ff136e42968c7
https://doi.org/10.1111/j.1600-079x.2012.01004.x
https://doi.org/10.1111/j.1600-079x.2012.01004.x
Publikováno v:
Journal of Pineal Research. 45:439-448
It is well-documented that melatonin influences insulin secretion. The effects are mediated by specific, high-affinity, pertussis-toxin-sensitive, G protein-coupled membrane receptors (MT1 as well MT2), which are present in both the pancreatic tissue
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e1dcbfa5a03fddfd2fd7b56988eaea84
https://doi.org/10.1111/j.1600-079x.2008.00612.x
https://doi.org/10.1111/j.1600-079x.2008.00612.x
Autor:
Ivonne, Bazwinsky-Wutschke, Sabine, Wolgast, Eckhard, Mühlbauer, Elke, Albrecht, Elmar, Peschke
Publikováno v:
Journal of pineal research. 53(4)
The pineal hormone melatonin exerts its influence on the insulin secretion of pancreatic islets by a variety of signalling pathways. The purpose of the present study was to analyse the impact of melatonin on the phosphorylated transcription factor cA
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::f3126849d81312b47543b9060fdf81f1
https://pubmed.ncbi.nlm.nih.gov/22616931
https://pubmed.ncbi.nlm.nih.gov/22616931
Publikováno v:
Histochemistry and cell biology. 134(2)
The present study dealt with the localization of different calcium-binding proteins (CaBPs) in the pancreatic tissue of non-diabetic and diabetic rats and in rat insulinoma beta-cells (INS-1). Transcripts of CaBPs displayed different expression level
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::04d22922a32176155df42349bc5f61e7
https://pubmed.ncbi.nlm.nih.gov/20607274
https://pubmed.ncbi.nlm.nih.gov/20607274
Publikováno v:
European journal of pharmacology. 606(1-3)
The transmission of circadian rhythms is mediated by specific promoter sequences binding a particular circadian clock factor. The pineal hormone melatonin acts via G-protein-coupled receptors to synchronise these clock-generated circadian rhythms. Th
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b54851eb17364a561593b5e66b9964df
https://pubmed.ncbi.nlm.nih.gov/19374844
https://pubmed.ncbi.nlm.nih.gov/19374844
Publikováno v:
Journal of pineal research. 39(3)
The effects of melatonin in mammalian cells are exerted via specific receptors or are related to its free radical scavenging activity. It has previously been reported that melatonin inhibits insulin secretion in the pancreatic islets of the rat and i
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::305d4bda1b4d995610b43374864d5fa1
https://pubmed.ncbi.nlm.nih.gov/16150114
https://pubmed.ncbi.nlm.nih.gov/16150114