Zobrazeno 1 - 10
of 44
pro vyhledávání: '"Saba Fekrvand"'
Autor:
Farhad Shahi, Reza Safaee, Saba Fekrvand, Fatemeh Fathi, Mohammad Reza Dabiri, Alireza Abdollahi, Hanieh Hosseini
Publikováno v:
International Journal of Hematology-Oncology and Stem Cell Research, Vol 17, Iss 2 (2023)
Background: Since 2019, Coronavirus has been a highly contagious disease. The COVID-19 outbreak was declared a pandemic by the World Health Organization in March 2020. Variable laboratory findings are reported in COVID-19 patients, among which elevat
Externí odkaz:
https://doaj.org/article/b549550115534de4b8b38f9e51597824
Publikováno v:
Frontiers in Immunology, Vol 13 (2022)
Common variable immunodeficiency (CVID) is a heterogeneous group of inborn errors of immunity characterized by reduced serum concentrations of different immunoglobulin isotypes. CVID is the most prevalent symptomatic antibody deficiency with a broad
Externí odkaz:
https://doaj.org/article/2d8bd57da6e04b58adc3ef2eb0ee2db5
Autor:
Shockrollah Farrokhi, Faezeh Abbasi-rad, Nafiseh Esmaeil, Roya Sherkat, Reza Yazdani, Sanaz Afshar-Ghasemlou, Saba Fekrvand, Mazdak Ganjalikhani-Hakemi
Publikováno v:
Iranian Journal of Allergy, Asthma and Immunology, Vol 19, Iss 4 (2020)
Common variable immunodeficiency (CVID) is a primary immune deficiency disorder characterized by a failure in B cell differentiation, impaired immunoglobulin production,and defect in response to vaccines. As a result of defective B cell maturation an
Externí odkaz:
https://doaj.org/article/2a6e6d111a824979844adc1b64a73c9e
Autor:
Reza Yazdani, Molood Safarirad, Saba Fekrvand, Ali Abbaszadeh Ganji, Hassan Abolhassani, Ahmad Vosughi Motlagh, Asghar Aghamohammadi
Publikováno v:
Endocrine, Metabolic & Immune Disorders - Drug Targets. 21:2099-2103
Kabuki syndrome is a rare congenital anomaly/mental retardation syndrome characterized by intellectual disability, developmental delay, short stature, facial dysmorphic features including ectropion of the lateral third of the lower eyelids and long p
Autor:
Asghar Aghamohammadi, Yasser Bagheri, Hassan Abolhassani, Seyed Alireza Mahdaviani, Mohammamd Nabavi, Afshin Shirkani, Reza Yazdani, Fereshte Salami, Gholamreza Azizi, Saba Fekrvand, Morteza Samadi, Sahar Shariati, Samaneh Delavari, Sepideh Shahkarami
Publikováno v:
Immunological Investigations. 51:381-394
Common variable immunodeficiency (CVID) is a primary immunodeficiency disease with a heterogeneous genetic background. Lipopolysaccharide-responsive beige-like anchor (LRBA), as well as cytotoxic T-lymphocyte-associated protein 4 (CTLA-4), have impor
Autor:
Farzad Nazari, Asghar Aghamohammadi, Sohail Mozdarani, Yasser Bagheri, Hassan Abolhassani, Saba Fekrvand, Mahsa Sohani, Gholamreza Hassanpour, Fatemeh Kiaee, Gholamreza Azizi, Hossein Mozdarani, Samaneh Delavari, Reza Yazdani
Publikováno v:
Immunological Investigations. 50:580-596
HIGM syndrome is a rare form of primary immunodeficiencies characterized by normal/increased amounts of serum IgM and decreased serum levels of other switched immunoglobulin classes. Since the affected patients are continuously infected with various
Autor:
Mojdeh Soltani, Mahnaz Rezaei, Saba Fekrvand, Mazdak Ganjalikhani‐Hakemi, Hassan Abolhassani, Reza Yazdani
Publikováno v:
Pediatric Allergy and Immunology. 33
Common variable immunodeficiency disorder (CVID) is a heterogeneous disorder and the most common symptomatic antibody deficiency disease characterized with hypogammaglobulinemia and a broad range of clinical manifestations. Multiple genetic, epigenet
Autor:
null Mojdeh Soltani, null Mahnaz Rezaei, null Saba Fekrvand, null Mazdak Ganjalikhani‐Hakemi, null Hassan Abolhassani, null Reza Yazdani
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::c15309886090930634c3c0a65de319c7
https://doi.org/10.1111/pai.13725/v2/response1
https://doi.org/10.1111/pai.13725/v2/response1
Autor:
Hassan Abolhassani, Asghar Aghamohammadi, Pilar Llobet Agulló, Hamid Ahanchian, Soheila Alyasin, Saba Arshi, Gholamreza Azizi, Mohamed-Ridha Barbouche, Mohammad Hassan Bemanian, Aziz Bousfiha, Zahra Chavoshzadeh, Taher Cheraghi, Romina Dieli Crimi, Charlotte Cunningham-Rundles, Abbas Dabbaghzadeh, Sepideh Darougar, Rainer Doffinger, Anne Durandy, Mohammad Ehlayel, Hermann Eibel, Mohammad Hossein Eslamian, Hossein Esmaeilzadeh, Teresa Espanol, Morteza Fallahpour, Saba Fekrvand, Andrew R Gennery, Javad Ghaffari, Negar Ghaffari, Sudhir Gupta, Lennart Hammarström, Marzieh Heydrzadeh, Arash Kalantari, Rasoul Nasiri Kalmarzi, Hirokazu Kanegane, Negar Khalighi, Abbas Khalili, Martin Lavin, Alireza Mahdaviani, Tooba Momen, Mohammad Nabavi, Tim Niehues, Hans D. Ochs, Peter Olbrich, Alessandro Plebani, Nima Rezaei, Farhad Seif, Mikko Seppänen, Mahnaz Sadeghi Shabestari, Alireza Shafiei, Mansoureh Shariat, Deepti Suri, Marzieh Tavakol, Mirjam van der Burg, Menno van Zelm, Ahmad Vosoughi Motlagh, Reza Yazdani
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::5b9c97f6b84cbcb6dd734523a60abefa
https://doi.org/10.1016/b978-0-12-821028-4.09989-9
https://doi.org/10.1016/b978-0-12-821028-4.09989-9
A phenocopy is defined as a clinical phenotype in an individual, noninherited, with environmental induction that is identical to the genetically determined phenotype of another. In parallel to immunity errors due to germline mutations of immune-relat
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::8fbaaca94f610fa1cf932d8cffc9d1ef
https://doi.org/10.1016/b978-0-12-821028-4.00009-9
https://doi.org/10.1016/b978-0-12-821028-4.00009-9