Zobrazeno 1 - 10
of 529
pro vyhledávání: '"SKP1A"'
Akademický článek
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Autor:
Lauterbach, Edward C.
Publikováno v:
In Progress in Neuropsychopharmacology & Biological Psychiatry 10 January 2013 40:236-239
Publikováno v:
In Parkinsonism and Related Disorders 2012 18 Supplement 1:S177-S179
Publikováno v:
In Parkinsonism and Related Disorders 2009 15 Supplement 3:S148-S151
Autor:
Edward C. Lauterbach
Publikováno v:
Progress in Neuro-Psychopharmacology and Biological Psychiatry. 40:236-239
Recently, the genes p19 S-phase kinase-associated protein 1A (SKP1), huntingtin interacting protein-2 (UBE2K), aldehyde dehydrogenase family 1 subfamily A1 (ALDH1A1), 19 S proteasomal protein PSMC4, and heat shock 70-kDa protein 8 (HSPA8) have been f
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::fc3db9ac5931e62f1c9643f6e8fcf570
https://doi.org/10.1016/j.pnpbp.2012.08.021
https://doi.org/10.1016/j.pnpbp.2012.08.021
Publikováno v:
Parkinsonism & Related Disorders. 15:S148-S151
Large-scale transcriptomics analysis of gene expression profile of sporadic Parkinson's disease (PD) substantia nigra (SN) has identified a number of differentially expressed genes participating in the neurotoxic cascade of DA neurons death, in parti
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ff0926ea034c29682ad94ed97434a411
https://doi.org/10.1016/s1353-8020(09)70803-x
https://doi.org/10.1016/s1353-8020(09)70803-x
Autor:
Edna Grünblatt, Gideon Rechavi, Moussa B.H. Youdim, Ninette Amariglio, Silvia Mandel, Peter Riederer, Jasmine Jacob Hirsch
Publikováno v:
Annals of the New York Academy of Sciences. 1053:356-375
Sporadic Parkinson's disease (PD) constitutes 99% of the disorder, while the remaining 1% of the cases is of familial (genetic) origin. The mutations reported to be associated with familial PD indicate impairment in protein processing and misfolding,
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0565bc080b4271735885463a8198e4d4
https://doi.org/10.1111/j.1749-6632.2005.tb00044.x
https://doi.org/10.1111/j.1749-6632.2005.tb00044.x
Akademický článek
Tento výsledek nelze pro nepřihlášené uživatele zobrazit.
K zobrazení výsledku je třeba se přihlásit.
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Publikováno v:
Parkinsonismrelated disorders. 18
Summary Aims To assess whether reduction of Skp1 will increase the vulnerability of dopaminergic neurons to genetic/environmental factors known to play a role in the pathological process of PD. Methods Short hairpin RNA lentiviruses infection of subs
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6c4375686b8ea406638a2a81094f9903
https://pubmed.ncbi.nlm.nih.gov/22166427
https://pubmed.ncbi.nlm.nih.gov/22166427
The aim of this study was to develop a new model of sporadic Parkinson disease (PD) based on silencing of the SKP1A gene, a component of the ubiquitin-proteasome/E3 ligase complex, Skp1, Cullin 1, F-box protein, which was found to be highly decreased
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::43c7ee3e40b971bfc470c8232beab4b0
https://europepmc.org/articles/PMC2781700/
https://europepmc.org/articles/PMC2781700/
