Zobrazeno 1 - 10
of 16
pro vyhledávání: '"SHAO-QING LEI"'
Autor:
Qian Sun, Qing-tao Meng, Ying Jiang, Hui-min Liu, Shao-qing Lei, Wa-ting Su, Wei-na Duan, Yang Wu, Zheng-yuan Xia, Zhong-yuan Xia
Publikováno v:
PLoS ONE, Vol 8, Iss 12, p e80859 (2013)
Ginsenoside Rb1 (RB1), the most clinically effective constituent of ginseng, possesses a variety of biological activities. The objectives of this study were to investigate the protective effects of RB1 and its underlying mechanism on renal injury ind
Externí odkaz:
https://doaj.org/article/5d0b8a1749904b86933365c27d6b9113
Autor:
Zhongyuan Xia, Yan Leng, Qing‑Tao Meng, Shao‑Qing Lei, Xiao‑Jing Wu, Qian Sun, Wei‑Na Duan, Min Liu
Publikováno v:
Molecular Medicine Reports
Previous studies have suggested that the Janus kinase (JAK)/signal transducers and activators of transcription (STAT) pathway is involved in hyperglycemia‑induced lung injury. The present study aimed to investigate the roles of suppressor of cytoki
Publikováno v:
Molecular Medicine Reports. 16:3648-3656
Although the mechanism remains unclear, ischemic post‑conditioning (IPO) is a promising approach to combat myocardial ischemia reperfusion (IR) injury; however, it has been proven ineffective in diabetes. The present study aimed to identify whether
Publikováno v:
Neuropsychiatric Disease and Treatment
Objective In this study, the neuroprotective mechanism of 2-arachidonoylglycerol 2-AG against non-caspase-dependent apoptosis in mice hippocampal neurons following MCAO was investigated. Method One hundred and fifty healthy clean male C57BL/6 mice we
Publikováno v:
Cell biology international. 41(1)
Long non-coding (lncRNA) MALAT1 can be increased by hypoxia or ischemic limbs. Also, downregulation of MALAT1 contributes to reduction of cardiomyocyte apoptosis. However, the functional involvement of MALAT1 in myocardial ischemia-reperfusion (I/R)
Publikováno v:
Oxidative Medicine & Cellular Longevity; 2/24/2019, p1-13, 13p
Publikováno v:
American journal of physiology. Renal physiology. 306(11)
Glomerular matrix accumulation is a hallmark of diabetic renal disease. Serine/threonine kinase PKC-β1 mediates glucose-induced Akt S473 phosphorylation, RhoA activation, and transforming growth factor (TGF)-β1 upregulation and finally leads to mat
Publikováno v:
Molecular Medicine Reports; Sep2017, Vol. 16 Issue 3, p3648-3656, 9p
Autor:
WEI‑NA DUAN, ZHONG‑YUAN XIA, MIN LIU, QIAN SUN, SHAO‑QING LEI, XIAO‑JING WU, QING‑TAO MENG, YAN LENG
Publikováno v:
Molecular Medicine Reports; Sep2017, Vol. 16 Issue 3, p2668-2674, 7p
Autor:
YANG WU, ZHONG-YUAN XIA, BO ZHAO, YAN LENG, JUAN DOU, QING-TAO MENG, SHAO-QING LEI, ZHI-ZE CHEN, JIE ZHU
Publikováno v:
International Journal of Molecular Medicine; Aug2017, Vol. 40 Issue 2, p389-399, 11p