Zobrazeno 1 - 10
of 1 023
pro vyhledávání: '"S. R. Price"'
Autor:
Schneider, Hans
Publikováno v:
Zeitschrift für Pflanzenkrankheiten, 1915 Feb . 24(8), 479-479.
Externí odkaz:
https://www.jstor.org/stable/43320148
Publikováno v:
Rivista di Patologia Vegetale, 1913 Aug 01. 6(6), 180-181.
Externí odkaz:
https://www.jstor.org/stable/42559111
Autor:
Ji, Lanxin1 (AUTHOR) lanxin.ji@nyulangone.org, Menu, Iris1 (AUTHOR), Majbri, Amyn1 (AUTHOR), Bhatia, Tanya1 (AUTHOR), Trentacosta, Christopher J.2 (AUTHOR), Thomason, Moriah E.1,3,4 (AUTHOR)
Publikováno v:
PLoS Biology. 11/19/2024, Vol. 22 Issue 11, p1-19. 19p.
Autor:
Gökgöz Özışık, Gülce1 (AUTHOR) gulcegokgozozisik@hitit.edu.tr, Yilmaz, Hayati2 (AUTHOR) hayati.yilmaz@maltepe.edu.tr
Publikováno v:
Journal of Clinical Medicine. Nov2024, Vol. 13 Issue 22, p6849. 9p.
Autor:
E. Löfberg, Jonas Bergström, A. Alvestrand, S. R. Price, A. Gutierrez, William E. Mitch, J. Wernerman, Björn Anderstam
Publikováno v:
European Journal of Clinical Investigation. 32:345-353
Background Treatment with glucocorticosteroids causes a negative nitrogen balance, but the kinetic mechanisms responsible for this catabolic effect are controversial. We investigated the effects of 60 mg day−1 prednisolone on protein synthesis and
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::425bed1a5d6e5d77e5e873c1ec233de4
https://doi.org/10.1046/j.1365-2362.2002.00993.x
https://doi.org/10.1046/j.1365-2362.2002.00993.x
Publikováno v:
Scopus-Elsevier
Abstract —Angiotensin II (Ang II) is known to act as a growth factor and may be involved in cardiac remodeling. We have shown that insulin-like growth factor-I (IGF-I) is an autocrine mediator of growth responses to Ang II in vascular smooth muscle
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c69c19edb6d727911198f967fd038a9e
https://doi.org/10.1161/01.hyp.34.5.1053
https://doi.org/10.1161/01.hyp.34.5.1053
Autor:
S. R. Price, X. Wang
Publikováno v:
Mineral and Electrolyte Metabolism. 25:224-227
Metabolic acidosis and glucocorticoids act in concert to stimulate branched-chain amino acid (BCAA) oxidation in adrenalectomized rats. In muscles of normal rats, metabolic acidosis increases the maximal activity of the rate-limiting enzyme, branched
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ecd7909b27eb15cc1d16fd528c15f077
https://doi.org/10.1159/000057452
https://doi.org/10.1159/000057452
Publikováno v:
American Journal of Physiology-Cell Physiology. 272:C2031-C2036
In muscles of rats with metabolic acidosis, branched-chain alpha-ketoacid dehydrogenase (BCKAD) activity is increased. Potential stimulatory signals include acidemia and/or glucocorticoids. It is unclear whether the signal(s) increases BCKAD activity
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9758641af89f93c4bd671fda66cd868e
https://doi.org/10.1152/ajpcell.1997.272.6.c2031
https://doi.org/10.1152/ajpcell.1997.272.6.c2031
Autor:
William E. Mitch, L S Phillips, Xiaonan H. Wang, X Ding, S. R. Price, B K England, C Jurkovitz, James L. Bailey
Publikováno v:
Journal of Clinical Investigation. 98:1703-1708
In normal subjects and diabetic patients, insulin suppresses whole body proteolysis suggesting that the loss of lean body mass and muscle wasting in insulinopenia is related to increased muscle protein degradation. To document how insulinopenia affec
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0d233e214f8a53cc60cd5ca634b19f32
https://doi.org/10.1172/jci118968
https://doi.org/10.1172/jci118968
Autor:
B. K. England, S. R. Price
Publikováno v:
Blood Purification. 13:147-152
Malnutrition and a loss of lean body mass frequently complicate chronic renal failure. Muscle wasting in uremia is caused by increased protein degradation, decreased protein synthesis and increased branched-chain amino acid oxidation. Acidosis and gl
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f3bbb3de9e03d87e2e90ab40223db621
https://doi.org/10.1159/000170197
https://doi.org/10.1159/000170197