Zobrazeno 1 - 4
of 4
pro vyhledávání: '"S. M. Deford"'
Autor:
Brian R. Pike, B. Bausano, P. Bramanti, Xiurong Zhao, Ronald L. Hayes, Francesca Antonia Arcadi, S. M. Deford, Filippo Flavio Angileri, Chiara Costa, Douglas K. Anderson, Arthur L. Day, Antonino Germanò
Publikováno v:
Scopus-Elsevier
Increases in intracellular calcium and subsequent activation of calcium-activated proteases (e.g., calpains) may play a critical role in central nervous system injury. Several studies have implicated calpain activation following subarachnoid hemorrha
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d99a67db99e4911fd19debb1a90c3e06
https://doi.org/10.1089/08977150260190474
https://doi.org/10.1089/08977150260190474
Autor:
S. M. DeFord, Raymond F. Genovese, Anita V. Moran, Rebecca Whipple, Ren-Wu Chen, Frank C. Tortella, Jitendra R. Dave, R. A. Connors
Publikováno v:
Cellular and molecular life sciences : CMLS. 64(21)
The objective of this study was to determine levels of DNA fragmentation in blood leukocytes and parietal cortex from guinea pigs following repeated low-level exposure to the chemical warfare nerve agent (CWNA) sarin. Guinea pigs were injected (s.c.)
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b8ad9cf2238ebbc5dcc6b23b79b175b2
https://pubmed.ncbi.nlm.nih.gov/17917701
https://pubmed.ncbi.nlm.nih.gov/17917701
Autor:
Xiurong Zhao, Douglas K. Anderson, N.C. Ringger, Esther Shohami, Ronald L. Hayes, Brian Bausano, S. M. Deford, Jennifer K. Newcomb-Fernandez, Brian R. Pike, Kevin K.W. Wang
Publikováno v:
Journal of neuroscience research. 64(2)
Primary septo-hippocampal cell cultures were incubated in varying concentrations of tumor necrosis factor (TNF-α; 0.3–500 ng/ml) to examine proteolysis of the cytoskeletal protein α-spectrin (240 kDa) to a signature 145 kDa fragment by calpain an
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::97670a9bc8b40ff215f34e6cbf8cff93
https://pubmed.ncbi.nlm.nih.gov/11288141
https://pubmed.ncbi.nlm.nih.gov/11288141
Publikováno v:
Brain research. 861(2)
Glutamate-mediated excitotoxicity has been shown to contribute to cellular dysfunction following traumatic brain injury (TBI). Increasing inhibitory function through stimulation of gamma-aminobutyric acid (GABA(A)) receptors may attenuate excitotoxic
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c46498cdce35f16f161633154a5eb1bd
https://pubmed.ncbi.nlm.nih.gov/10760494
https://pubmed.ncbi.nlm.nih.gov/10760494