Zobrazeno 1 - 10
of 32
pro vyhledávání: '"S. A. Sosnikhina"'
Autor:
Viktor G. Smirnov, E. I. Mikhailova, Natalia Tsvetkova, Natalia D. Tikhenko, A. V. Voylokov, S. P. Sosnikhina
Publikováno v:
Экологическая генетика, Vol 16, Iss 2, Pp 40-49 (2018)
The article provides information about the history and methods of development of Peterhof rye genetic collection, founded by V.S. Fedorov, Associate Professor of the Leningrad University. Isolation of self-compatible mutants, their crosses with self-
Autor:
S. P. Sosnikhina, Sergey V. Malyshev, A. V. Voylokov, Natalia Tsvetkova, T. V. Dolmatovich, N. A. Kartel
Publikováno v:
Russian Journal of Genetics. 49:511-516
The sy19 mutation, which impairs the homology of meiotic chromosome synapsis in rye, were mapped using a specially created F2 population by means of isozyme Acph1 locus and microsatellite (SSR) markers. The sy19 gene was localized in the chromosome 7
Autor:
S. P. Sosnikhina, Sergey V. Malyshev, N. A. Kartel, T. V. Dolmatovich, A. V. Voylokov, Natalia Tsvetkova
Publikováno v:
Russian Journal of Genetics. 49:411-416
The recessive spontaneous sy18 mutation with nonhomologous synapsis was mapped in rye. The sy18 gene was located in the centromeric region of chromosome 2R in relation to three rye SSR (simple sequence repeats) loci, i.e., Xrems1130, Xrems1203, and X
Publikováno v:
Russian Journal of Genetics. 46:1210-1213
The Peterhof Collection of spontaneous meiotic mutants of rye was used as a model to study the genetic control of meiosis key events in an organism with a large genome. A combination of methods, which included fluorescence in situ DNA-DNA hybridizati
Autor:
I. V. Iordanskaya, S. V. Golubtsov, Oxana Kolomiets, E. I. Mikhailova, Yu. F. Bogdanov, A. V. Voilokov, S. P. Sosnikhina
Publikováno v:
Russian Journal of Genetics. 46:682-688
A study was made of the expression and inheritance of the sy11 mutation, which alters homologous chromosome synapsis in meiotic prophase I of rye. The abnormal phenotype proved to be determined by a recessive allele of a single sy11 gene. Univalents
Autor:
I. V. Iordanskaya, S. P. Sosnikhina, A. V. Voylokov, Natalia Tsvetkova, E. I. Mikhailova, Oxana Kolomiets, Yu. F. Bogdanov, A. V. Lovtsyus
Publikováno v:
Russian Journal of Genetics. 45:1385-1393
Expression and inheritance of the sy18 mutation causing impairment of synapsis homology were studied. It was established that the abnormal phenotype is determined by a recessive allele of the sy18 gene. Univalents and multivalents are observed in hom
Autor:
S. P. Sosnikhina, A. V. Lovtsyus, Natalia Tsvetkova, Yu. F. Bogdanov, Oxana Kolomiets, O. A. Tikholiz, O. S. Sapronova, E. I. Mikhailova, Yu. S. Fedotova
Publikováno v:
Russian Journal of Genetics. 43:1193-1200
The cytological phenotype was studied in a desynaptic form isolated from a population of rye cultivar Vyatka. The primary defect of desynaptic plants was identified as nonhomologous (heterologous) chromosome synapsis, which was observed by electron m
Autor:
E. I. Mikhailova, Dylan Phillips, Glyn Jenkins, Alexandra V. Lovtsyus, S. P. Sosnikhina, R. N. Jones
Publikováno v:
Genetics. 174:1247-1258
Assembly of two orthologous proteins associated with meiotic chromosome axes in Arabidopsis thaliana (Asy1 and Zyp1) was studied immunologically at meiotic prophase of meiosis of wild-type rye (Secale cereale) and its synaptic mutant sy10, using anti
Autor:
S. P. Sosnikhina, Oxana Kolomiets, S. N. Priyatkina, Y F Bogdanov, V. G. Smirnov, E. I. Mikhailova, S.Y. Dadashev, O. A. Tikholiz
Publikováno v:
Cytogenetic and Genome Research. 109:215-220
Spontaneous meiotic mutations of winter rye Secale cereale L. (2n = 14) were revealed in inbred F2 progenies, which were obtained by self-pollination of F1 hybrids resulting from crosses of individual plants of cultivar Vyatka or weedy rye with plant
Publikováno v:
Russian Journal of Genetics. 39:288-294
Inheritance of two spontaneous meiosis-specific mutations with similar cytologic phenotype was studied. Both mutations were independently obtained from two rye populations (Vyatka variety and weedy rye). Both mutations are recessive, allelic, and mon