Zobrazeno 1 - 10
of 10
pro vyhledávání: '"S.‐C. G. Hui"'
Autor:
B. S. Qiu, S. C. G. Hui
Publikováno v:
Experientia. 52:66-69
Prazosin was injected i.v. at a dose of 50 micrograms/kg every 2 h for 8 h in conscious rats. Its hypotensive action significantly declined. A similar effect was also observed in rabbits pretreated with prazosin (40 micrograms/kg, i.v.) every 1 h for
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::aebd8fdd845bf4d37f51c778e678bb40
https://doi.org/10.1007/bf01922418
https://doi.org/10.1007/bf01922418
Autor:
S-C. G. Hui, C. W. Ogle
Publikováno v:
Archives Internationales de Physiologie, de Biochimie et de Biophysique. 101:43-46
The endoperoxide analogues U46619 and U44069 when injected intravenously (i.v.), into the femoral artery or directly into the aortic arch in chloralose-anaesthetised rats, decreased arterial blood pressure dose-dependently. Treatment i.v. 30 min befo
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::dfec117e26b49950821b9fc1fdeb68d6
https://doi.org/10.3109/13813459308998127
https://doi.org/10.3109/13813459308998127
Publikováno v:
Clinical and experimental pharmacologyphysiology. 19(10)
SUMMARY 1. The changes in plasma levels of thromboxane-B2 (TXB2) and 6-keto-prostaglandin-F1α(6-keto-PGF1α) were examined in rats given 5, 25, 50 or 100 μg/mL nicotine in drinking water for 10 days. 2. The effect of nicotine on prostacyclin (PGI2)
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::977342419d9643e3084e36c9b0f391fa
https://pubmed.ncbi.nlm.nih.gov/1424297
https://pubmed.ncbi.nlm.nih.gov/1424297
Autor:
S.‐C. G. Hui, C. W. Ogle
Publikováno v:
Journal of Pharmacy and Pharmacology. 43:592-593
Intravenous (i.v.) or intra-arterial injections of U46619, a thromboxane A2 (TxA2)-mimetic agent, into chloralose-anaesthetized rats dose-dependently decreased the arterial blood pressure. Indomethacin (8 mg kg−1) or atropine (1 mg kg−1), given i
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::fef8831ff0fafe6101714c8a2ed7d805
https://doi.org/10.1111/j.2042-7158.1991.tb03543.x
https://doi.org/10.1111/j.2042-7158.1991.tb03543.x
Publikováno v:
Clinical and Experimental Pharmacology and Physiology. 11:621-625
SUMMARY 1. In chloralose anaesthetized rats, intravenous administration of captopril, SQ 20881, SA 446 or MK 421 (0.5 mg/kg) potentiated the depressor responses to arachidonic acid 3 mg/kg given intravenously. 2. Same doses of the above angiotensin c
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::74436ad47ed4210518ad9cd7f6e5ba35
https://doi.org/10.1111/j.1440-1681.1984.tb00875.x
https://doi.org/10.1111/j.1440-1681.1984.tb00875.x
Publikováno v:
Pharmacological Research Communications. 16:495-511
Summary Morphine preference and tendency to relapse to morphine tolerance and dependence were studied in rats which were previously made morphine dependent. Tolerance to, and physical dependence on, morphine were initially produced by administration
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b71741b9e3b0319aff213224eacb4231
https://doi.org/10.1016/s0031-6989(84)80018-1
https://doi.org/10.1016/s0031-6989(84)80018-1
Publikováno v:
Clinical and Experimental Pharmacology and Physiology. 13:123-130
SUMMARY 1. The mechanisms underlying potentiation by captopril of the depressor responses to arachidonic acid were studied in chloralose-anaesthetized rats. 2. Captopril, in a dose (0.5 mg/kg, i.v.) which inhibited the pressor responses to angiotensi
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::53dffd8a29be8827a4130a570d881714
https://doi.org/10.1111/j.1440-1681.1986.tb00325.x
https://doi.org/10.1111/j.1440-1681.1986.tb00325.x
Autor:
S.-C. G. Hui, Clive W. Ogle
Publikováno v:
Clinical and Experimental Pharmacology and Physiology. 13:819-822
Captopril (1-5 mg/kg, i.v.) did not affect the vasodepressor responses to substance P (1-30 micrograms/kg, i.v.) in anaesthetized rats. Substance P (100 micrograms/kg, s.c.) produced significant algesia in mice; this was not potentiated by the smalle
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::14571ec3f4dfe23a72e2599f177add11
https://doi.org/10.1111/j.1440-1681.1986.tb02386.x
https://doi.org/10.1111/j.1440-1681.1986.tb02386.x
Publikováno v:
Clinical and experimental pharmacologyphysiology. 10(3)
Intravenous administration of captopril (0.1-0.3 mg/kg) to normotensive pithed rats, with or without unilateral nephrectomy, was followed by a sustained fall in arterial blood pressure. Concomitantly pressor responses to electrical stimulation of the
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4ce9bb461b4ba4694f4b755913d0a26a
https://pubmed.ncbi.nlm.nih.gov/6354532
https://pubmed.ncbi.nlm.nih.gov/6354532
Autor:
S‐C. G. Hui, A. L. A. Bourn
Publikováno v:
Clinical and experimental pharmacologyphysiology. 9(6)
SUMMARY 1. In chloralose anaesthetized rats intravenous administration of captopril (0.5 mg/kg) was followed by an approximately 100-fold decrease in sensitivity to the pressor actions of angiotensin I. Concomitantly there was a 100-fold increase in
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::28528f906fe68ca3c25c3c16bae38112
https://pubmed.ncbi.nlm.nih.gov/6819911
https://pubmed.ncbi.nlm.nih.gov/6819911