Zobrazeno 1 - 9
of 9
pro vyhledávání: '"S W, Christianson"'
Autor:
D. V. Serreze, E. H. Leiter, M. S. Hanson, S. W. Christianson, L. D. Shultz, R. M. Hesselton, D. L. Greiner
Publikováno v:
Diabetes. 44:1392-1398
Autor:
L D Shultz, P A Schweitzer, S W Christianson, B Gott, I B Schweitzer, B Tennent, S McKenna, L Mobraaten, T V Rajan, D L Greiner
Publikováno v:
The Journal of Immunology. 154:180-191
The scid mutation was backcrossed ten generations onto the NOD/Lt strain background, resulting in an immunodeficient stock (NOD/LtSz-scid/scid) with multiple defects in adaptive as well as nonadaptive immunologic function. NOD/LtSz-scid/scid mice lac
Publikováno v:
Diabetes. 42:44-55
Publikováno v:
Diabetes. 41:1672-1676
Autor:
S C, Pelsue, P A, Schweitzer, I B, Schweitzer, S W, Christianson, B, Gott, J P, Sundberg, W G, Beamer, L D, Shultz
Publikováno v:
European journal of immunology. 28(4)
The autosomal recessive mutation "flaky skin" (fsn) causes pleiotropic abnormalities in the immune and hematopoietic systems accompanied by pathologic changes in the skin. Homozygotes (fsn/fsn) showed increased size and histological alterations in th
Autor:
S W, Christianson, D L, Greiner, R A, Hesselton, J H, Leif, E J, Wagar, I B, Schweitzer, T V, Rajan, B, Gott, D C, Roopenian, L D, Shultz
Publikováno v:
Journal of immunology (Baltimore, Md. : 1950). 158(8)
Genetic crosses produced NOD/LtSz mice doubly homozygous for the severe combined immunodeficiency (scid) mutation and the beta2m (B2m) null allele. Both NOD/LtSz-scid/scid and NOD/LtSz-scid/scid B2m(null) mice lacked mature lymphocytes and serum Ig.
Autor:
P, Tapley, N K, Shevde, P A, Schweitzer, M, Gallina, S W, Christianson, I L, Lin, R B, Stein, L D, Shultz, J, Rosen, P, Lamb
Publikováno v:
Experimental hematology. 25(2)
The mouse mutation viable motheaten (me(v)) results in defects in the expression and catalytic activity of the cytoplasmic protein tyrosine phosphatase known as hematopoietic cell phosphatase (HCP). This reduction in HCP activity leads to the aberran
Publikováno v:
In vivo (Athens, Greece). 10(1)
Human cord blood (HuCB) can colonize a murine fetal thymus organ culture (FTOC) and generate phenotypically immature (CD4+ CD8+) and mature (CD4+ CD8-; CD4- CD8+) T cells. We have used this model system to demonstrate that the human T cells that deve
Autor:
L D, Shultz, P A, Schweitzer, S W, Christianson, B, Gott, I B, Schweitzer, B, Tennent, S, McKenna, L, Mobraaten, T V, Rajan, D L, Greiner
Publikováno v:
Journal of immunology (Baltimore, Md. : 1950). 154(1)
The scid mutation was backcrossed ten generations onto the NOD/Lt strain background, resulting in an immunodeficient stock (NOD/LtSz-scid/scid) with multiple defects in adaptive as well as nonadaptive immunologic function. NOD/LtSz-scid/scid mice lac