Zobrazeno 1 - 10
of 10
pro vyhledávání: '"S Santosh Reddy"'
Autor:
Vinayak Gupta, Poovitha Sundar, Nitish R. Mahapatra, Bhargavi Natarajan, S Santosh Reddy, Vikas Arige, Abrar A. Khan, Manoj Kumar Barthwal
Publikováno v:
Journal of Molecular and Cellular Cardiology. 158:140-152
3-Hydroxy-3-methyl glutaryl-coenzyme A reductase (Hmgcr) encodes the rate-limiting enzyme in the cholesterol biosynthesis pathway. The regulation of Hmgcr in rat models of genetic hypertension (viz. Spontaneously Hypertensive Rat [SHR] and its normot
Autor:
Bhargavi Natarajan, Vikas Arige, Nitish R. Mahapatra, Manoj Kumar Barthwal, Abrar A. Khan, S Santosh Reddy
Publikováno v:
Hypertension Research. 44:154-167
Kidneys have a high resting metabolic rate and low partial pressure of oxygen due to enhanced mitochondrial oxygen consumption and ATP production needed for active solute transport. Heightened mitochondrial activity leads to progressively increasing
Autor:
Vikas Arige, Nitish R. Mahapatra, Ananthamohan Kalyani, S Santosh Reddy, Abrar A. Khan, Heena Agarwal, Manoj Kumar Barthwal, Bhargavi Natarajan, Vinayak Gupta
Publikováno v:
Mol Cell Biol
Hypercholesterolemia is a strong predictor of cardiovascular diseases. The 3-hydroxy-3-methylglutaryl coenzyme A reductase gene (Hmgcr) coding for the rate-limiting enzyme in the cholesterol biosynthesis pathway is a crucial regulator of plasma chole
Autor:
S Santosh Reddy, Manoj Kumar Barthwal, Nitish R. Mahapatra, Sagar Bkc, Arige, Bhargavi Natarajan, Santhoshkumar R, Abrar A. Khan
Kidneys have a high resting metabolic rate and low tissue partial pressure of oxygen due to enhanced mitochondrial oxygen consumption and ATP production for active solute transport. Enhanced mitochondrial activity leads to progressive hypoxia from th
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::310490e0af1c4aeb64774fb6e258aa5e
https://doi.org/10.1101/816470
https://doi.org/10.1101/816470
Publikováno v:
Hypertension. 74
NADPH oxidase mediated ROS production has been recognized to be an important contributory mechanism for hypertension and left ventricular hypertrophy (LVH). However, the regulatory role of NADPH oxidase mediated superoxide production in Angiotensin I
Autor:
Madhu Dikshit, Poovitha Sundar, Manoj Kumar Barthwal, Vinayak Gupta, Vikas Arige, Abrar A. Khan, Nitish R. Mahapatra, S Santosh Reddy
3-Hydroxy-3-methyl glutaryl-coenzyme A reductase (Hmgcr) encoding the rate-limiting enzyme in the cholesterol biosynthesis pathway is a candidate gene for essential hypertension (EH). However, the regulation ofHmgcrin rat models of hypertension (viz.
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::120a20e328428f79497093af84012b4a
https://doi.org/10.1101/614669
https://doi.org/10.1101/614669
Autor:
Nitish R. Mahapatra, Ananthamohan Kalyani, S. Santosh Reddy, Anshu Agarwal, Vikas Arige, Abrar A. Khan, Bhargavi Natarajan, Manoj Kumar Barthwal, Vinayak Gupta
Publikováno v:
Journal of molecular biology. 431(6)
Monoamine oxidase B (MAO-B), a flavoenzyme located in the outer mitochondrial membrane, is involved in the catabolism of monoamines. Altered levels of MAO-B are associated with cardiovascular/neuronal diseases. However, molecular mechanisms of MAO-B
Autor:
Ananthamohan Kalyani, Manoj Kumar Barthwal, G. Bhanuprakash Reddy, Nitish R. Mahapatra, Heena Agarwal, Vinayak Gupta, S Santosh Reddy, Vikas Arige, Madhu Dikshit, Abrar A. Khan
Hypercholesterolemia is a strong predictor of cardiovascular diseases. 3-Hydroxy-3-methylglutaryl-coenzyme A reductase gene (Hmgcr) coding for the rate-limiting enzyme in the cholesterol biosynthesis pathway is a crucial regulator of plasma cholester
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2549524f5f8537fcbe72f94151efa6a4
https://doi.org/10.1101/383448
https://doi.org/10.1101/383448
Publikováno v:
Journal of Drug Delivery and Therapeutics. 4
Objective: To review multiple organ dysfunction syndrome with respect to: 1) clinical and preclinical measurement systems; 2) Interventions/ Model system used 3) pathophysiology and 4) Various therapeutic implications. Methods: The Medline, Pubmed, P
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