Zobrazeno 1 - 10
of 101
pro vyhledávání: '"S Rosen-Bronson"'
Autor:
A L King, S. J. Moodie, A. M. Dearlove, Harold Ellis, S. Rosen-Bronson, J. S. Fraser, Paul J. Ciclitira, David Curtis
Publikováno v:
Annals of Human Genetics. 65:377-386
Susceptibility to coeliac disease has a strong genetic component. The HLA associations have been well described but it is clear that other genes outside this region must also be involved in disease development. Two previous genome-wide linkage studie
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::a78b3aee538d27d2f866227954a91bb9
https://doi.org/10.1046/j.1469-1809.2001.6540377.x
https://doi.org/10.1046/j.1469-1809.2001.6540377.x
Autor:
P. M. BRETT, J. Y. YIANNAKOU, M.-A. MORRIS, S. ROSEN BRONSON, C. MATHEW, D. CURTIS, P. J. CICLITIRA
Publikováno v:
Annals of Human Genetics. 62:25-32
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::7b7cfee8f09d6cd91072516da4ecd053
https://doi.org/10.1017/s0003480098006642
https://doi.org/10.1017/s0003480098006642
Publikováno v:
Clinical and Experimental Immunology. 111:158-165
The nature of the immunopathogenic relationship underlying the very strong association of coeliac disease (CD) to the HLA-DQ (A1*0501, B1*0201) genotype is not known, but probably relates to binding of gluten-derived epitopes to the HLA-DQ (α1*0501,
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d40e5be9fb1cd37c670c1bd165770930
https://doi.org/10.1046/j.1365-2249.1998.00448.x
https://doi.org/10.1046/j.1365-2249.1998.00448.x
Publikováno v:
Gut. 39:818-823
BACKGROUND: Concentrations of pro-inflammatory cytokines are raised in the small intestine of patients with coeliac disease after ingestion of gluten but there are equivalent data on interleukin-4 (IL-4) and interleukin-10 (IL-10) producing cells. Th
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::70e816815fdb52a5097bb5195c4b9a54
https://doi.org/10.1136/gut.39.6.818
https://doi.org/10.1136/gut.39.6.818
Publikováno v:
The Journal of Immunology. 150:1314-1324
Microvariation within the DR1 Ag family has created two DR molecules which differ only at beta-chain residues 85 (Val/Ala) and 86 (Gly/Val). TCR utilized by human alloproliferative T lymphocyte clones which can distinguish between these microvariants
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::c732a58120c9feef1e14a9e8fe6e3822
https://doi.org/10.4049/jimmunol.150.4.1314
https://doi.org/10.4049/jimmunol.150.4.1314
Publikováno v:
Transplantation. 98:54-55
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::ef4e00718e711eeb31be88aecdd1471a
https://doi.org/10.1097/00007890-201407151-00176
https://doi.org/10.1097/00007890-201407151-00176
Publikováno v:
The Journal of Immunology. 146:4264-4270
The contributions to allorecognition of polymorphic amino acids in the HLA-DR7 beta 1 chain were analyzed by using mutant DR7 beta 1 chains with single amino acid substitutions at position 4, 11, 13, 25, 30, 37, 57, 60, 67, 70, 71, 74, or 78. Transfe
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::87fb298d0c4f255d675d426d858a2c0a
https://doi.org/10.4049/jimmunol.146.12.4264
https://doi.org/10.4049/jimmunol.146.12.4264
Autor:
J S Fraser, W. Engel, Harold Ellis, S Rosen-Bronson, E L Pollock, Paul J. Ciclitira, Herbert Wieser
Publikováno v:
Gut. 52(2)
Background: Coeliac disease (CD) is an enteropathy mediated by gluten specific T cells which secrete interferon γ (IFN-γ) when stimulated by gluten peptides presented by HLA-DQ2 or DQ8 molecules. Residues 62–75 of α2 gliadin have been proposed a
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8835e3fbf56ef08e9ee690032fa791d3
https://pubmed.ncbi.nlm.nih.gov/12524389
https://pubmed.ncbi.nlm.nih.gov/12524389
Autor:
Matthew Cooper, Jennifer Verbesey, O. Timofeeva, Monica Grafals, B. Javaid, S. Rosen-Bronson, D. Li, A. Gilbert
Publikováno v:
Transplantation. 98:513
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::9bcbc626f76a5dc935813c781d5c9c44
https://doi.org/10.1097/00007890-201407151-01716
https://doi.org/10.1097/00007890-201407151-01716
Autor:
Harold Ellis, M.-A. Morris, A. M. Dearlove, David Curtis, Paul J. Ciclitira, M. Rhodes, A L King, J. Y. Yiannakou, Christopher G. Mathew, Peter Brett, S. Rosen-Bronson
Publikováno v:
Annals of human genetics. 64(Pt 6)
The susceptibility to develop coeliac disease (CD) has a strong genetic component, which is not entirely explained by HLA associations. Two previous genome wide linkage studies have been performed to identify additional loci outside this region. Thes
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::acfcb9dcf617890235fd6f4e1b7abbfb
https://pubmed.ncbi.nlm.nih.gov/11281212
https://pubmed.ncbi.nlm.nih.gov/11281212