Zobrazeno 1 - 10
of 35
pro vyhledávání: '"S I, Baskin"'
Autor:
C K, Zoltani, S I, Baskin
Publikováno v:
Cellular and molecular biology (Noisy-le-Grand, France). 53(1)
Hypoxia-caused modulation of cardiac electrophysiology was modeled by computer simulation. Emphasis was on the effect of activation of anionic channels on the electrical state of the tissue. The model includes implicitly the effect of the presence of
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::b65f5b5d8689ce679d10d98ffda3db9b
https://pubmed.ncbi.nlm.nih.gov/17519115
https://pubmed.ncbi.nlm.nih.gov/17519115
Publikováno v:
Computers in Cardiology, 2004.
A high performance computational study of the cellular processes of ventriculur cardiac tissue exposed to cyanide (CN) is presented. The model used was based on the Luo-Rudy formalism with modifications. To account for the CN-caused changes, the mode
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::03ede03c729f9ab635acd6b2490b7809
https://doi.org/10.1109/cic.2004.1442927
https://doi.org/10.1109/cic.2004.1442927
A series of computer studies showing the effect of cyanide (CN) on the electrophysiology of cardiac tissue is presented. A mathematical model of the electrophysiology of cardiac tissue, with initial and boundary conditions based on experimental data
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::60163f7de6b378e71994f2fafe6f6366
https://doi.org/10.21236/ada432856
https://doi.org/10.21236/ada432856
Publikováno v:
Advances in experimental medicine and biology. 483
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::04f2b46bb1500e67a20cc08596f3fa69
https://pubmed.ncbi.nlm.nih.gov/11787588
https://pubmed.ncbi.nlm.nih.gov/11787588
Publikováno v:
Journal of applied toxicology : JAT. 20
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::c4df5b6cdd653297417558e48f25eaf4
https://pubmed.ncbi.nlm.nih.gov/11428618
https://pubmed.ncbi.nlm.nih.gov/11428618
Publikováno v:
Journal of applied toxicology : JAT. 20
Several anionic sulfur compounds are recognized as efficacious pretreatments for sulfur mustard (HD) poisoning. Our intent was to see if pretreatment compounds had a direct effect on DNA, a site where HD damage is thought to occur. A modification of
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::7d27f38cb3c8944f406145ef9b98cf47
https://pubmed.ncbi.nlm.nih.gov/11428639
https://pubmed.ncbi.nlm.nih.gov/11428639
Autor:
S I, Baskin, D W, Porter, G A, Rockwood, J A, Romano, H C, Patel, R C, Kiser, C M, Cook, A L, Ternay
Publikováno v:
Journal of applied toxicology : JAT. 19(3)
Antidotes for cyanide (CN) intoxication include the use of sulfane sulfur donors (SSDs), such as thiosulfate, which increase the conversion of CN to thiocyanate by the enzyme rhodanese. To develop pretreatments that might be useful against CN, SSDs w
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::ba07108335693f600b86ee23939f7814
https://pubmed.ncbi.nlm.nih.gov/10362268
https://pubmed.ncbi.nlm.nih.gov/10362268
Autor:
C N, Lieske, C R, Clark, L D, Zoeffel, R L, von Tersch, J R, Lowe, C D, Smith, C A, Broomfield, S I, Baskin, D M, Maxwell
Publikováno v:
Journal of applied toxicology : JAT. 16(2)
As part of our studies directed at new treatments for cyanide poisoning we examined the effect of temperature on both the non-catalyzed and the albumin-catalyzed reactions of cyanide with a colloidal suspension of elemental sulfur (CSES). Using satur
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::60a544b918b40c2fcfbcdff031866694
https://pubmed.ncbi.nlm.nih.gov/8935794
https://pubmed.ncbi.nlm.nih.gov/8935794
Autor:
D W, Porter, S I, Baskin
Publikováno v:
Journal of biochemical toxicology. 11(1)
3-Mercaptopyruvate sulfurtransferase catalyzes the transfer of sulfur from 3-mercaptopyruvate to several possible acceptor molecules, one of which is cyanide. Because the transsulfuration of cyanide is the primary in vivo mechanism of detoxification,
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::8e39d1ddbba29ba12f6588a186bfef89
https://pubmed.ncbi.nlm.nih.gov/8806051
https://pubmed.ncbi.nlm.nih.gov/8806051
Autor:
E U, Maduh, S I, Baskin
Publikováno v:
Research communications in molecular pathology and pharmacology. 86(2)
Detoxification of cyanide is catalyzed by a sulfurtransferase, rhodanese, a phosphoprotein regulated by unknown protein kinases. In this study, we determined if a Ca2+/phospholipid-modulated phosphotransferase, protein kinase C (PKC) could modify rho
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::44c8f28b0f4faac0d7faa85a00fd788c
https://pubmed.ncbi.nlm.nih.gov/7881866
https://pubmed.ncbi.nlm.nih.gov/7881866