Semaphorin 3F signaling actively retains neutrophils at sites of inflammation

Autor: Ailiang Zhang, Fiona Murphy, S Eamsamarng, Jenna L. Cash, Andrew J.W. Furley, Freek van Eeden, Tracie Plant, Moira K. B. Whyte, Emily R. Watts, Tyler Morrison, Leila Reyes, Felix Ellett, Jessie-May Morgan, Philip M. Elks, Patrícia Coelho, Stephen A. Renshaw, Ximena L. Raffo-Iraolagoitia, Ananda S. Mirchandani, Duncan Humphries, Manuel A. Sanchez-Garcia, Leo M. Carlin, Sarah R. Walmsley, Catherine A. Loynes

Publikováno v: The Journal of Clinical Investigation
Plant, T, Eamsamarng, S, Sanchez-Garcia, M A, Reyes, L, Renshaw, S A, Coelho, P, Mirchandani, A S, Morgan, J-M, Ellett, F E, Morrison, T, Humphries, D, Watts, E R, Murphy, F, Raffo-Iraolagoitia, X L, Zhang, A, Cash, J L, Loynes, C, Elks, P M, Van Eeden, F, Carlin, L M, Furley, A J W, Whyte, M K B & Walmsley, S R 2020, ' Semaphorin 3F signaling actively retains neutrophils at sites of inflammation ', Journal of Clinical Investigation, vol. 130, no. 6, pp. 3221-3237 . https://doi.org/10.1172/JCI130834

Neutrophilic inflammation is central to disease pathogenesis, for example, in chronic obstructive pulmonary disease, yet the mechanisms that retain neutrophils within tissues remain poorly understood. With emerging evidence that axon guidance factors

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