Zobrazeno 1 - 6
of 6
pro vyhledávání: '"S D, Konduri"'
Autor:
N, Yanamandra, S D, Konduri, S, Mohanam, D H, Dinh, W C, Olivero, M, Gujrati, G L, Nicolson, M, Obeyeseke, J S, Rao
Publikováno v:
Clinicalexperimental metastasis. 18(7)
Urokinase-type plasminogen activator receptors (uPARs) play an important role in tumor invasion by localizing degradative enzymes at the invasive zone. Our previous studies with human glioblastoma cell line SNB19 expressing AS-uPAR stable tranfectant
Publikováno v:
Clinicalexperimental metastasis. 18(4)
Human tissue factor pathway inhibitor-2 (TFPI-2), also known as placental protein (PP5) and matrix-associated serine protease inhibitor (MSPI), is a 32-kDa extracellular matrix (ECM) protein consisting of three tandomly arranged Kunitz-type domains t
Publikováno v:
Clinicalexperimental metastasis. 18(3)
Human tissue factor pathway inhibitor-2 (TFPI-2) is a Kunitz-type serine protease inhibitor that inhibits plasmin, trypsin, chymotrypsin, cathepsin G and plasma kallikrein but not urokinase (uPA) or tissue-type plasminogen activator and thrombin. Ear
Publikováno v:
Clinical cancer research : an official journal of the American Association for Cancer Research. 7(3)
Protease inhibitors regulate a variety of physiological and pathological processes including angiogenesis, embryo implantation, intravascular fibrinolysis, wound healing, and tumor invasion. Tissue factor pathway inhibitor (TFPI) 2 is a Mr 32,000 Kun
Publikováno v:
International journal of oncology. 18(1)
Human tissue factor pathway inhibitor-2 (TFPI-2) is a 32-kDa serine protease inhibitor that inhibits plasmin, trypsin, chymotrypsin, cathepsin G, and plasma kallikrein but not urokinase and tissue-type plasminogen activators or thrombin. After discov
Autor:
C, Nirmala, S L, Jasti, R, Sawaya, A P, Kyritsis, S D, Konduri, F, Ali-Osman, J S, Rao, S, Mohanam
Publikováno v:
International journal of cancer. 88(5)
Radiation-induced damage to the central nervous system (CNS) is believed to target glial or endothelial cells or both, although the pathophysiology of the process is poorly understood. We therefore used a coculture system, in which glioblastoma SNB19