Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Sükrü Anıl Doğan"'
Autor:
Mehmet Cihan Balci, Asuman Gedikbasi, Sukru Anil Dogan, Sevde Kahraman, Suzin Tatoryan, Sebnem Tekin Neijmann, Meryem Karaca, Fatmahan Atalar, Gulden Gokcay
Publikováno v:
Scientific Reports, Vol 14, Iss 1, Pp 1-8 (2024)
Abstract Cystathionine beta-synthase deficiency (CBSD) is the most prevalent inherited disorder of homocysteine metabolism in the transsulphuration pathway. Research have suggested oxidative stress and inflammation as candidate pathogenic mechanisms
Externí odkaz:
https://doaj.org/article/68c0e16ff6e647bb9b5a5e8c48f3f36e
Autor:
Sevgin Demirci, Evren Koban Baştanlar, Nihan Dilşad Dağtaş, Evangelia Pişkin, Atilla Engin, Füsun Ozer, Eren Yüncü, Sükrü Anıl Doğan, Inci Togan
Publikováno v:
PLoS ONE, Vol 8, Iss 12, p e81952 (2013)
In the present study, to contribute to the understanding of the evolutionary history of sheep, the mitochondrial (mt) DNA polymorphisms occurring in modern Turkish native domestic (n = 628), modern wild (Ovis gmelinii anatolica) (n = 30) and ancient
Externí odkaz:
https://doaj.org/article/4e937155767f411aabe8fc2bc117c9ad
Autor:
Samet Agca, Aylin Domaniku‐Waraich, Sevval Nur Bilgic, Melis Sucuoglu, Meric Dag, Sukru Anil Dogan, Serkan Kir
Publikováno v:
Journal of Cachexia, Sarcopenia and Muscle, Vol 15, Iss 5, Pp 1898-1914 (2024)
Abstract Background Tumour‐induced skeletal muscle wasting in the context of cancer cachexia is a condition with profound implications for patient survival. The loss of muscle mass is a significant clinical obstacle and is linked to reduced toleran
Externí odkaz:
https://doaj.org/article/d0200d5c1178411a990b0436bd6dd43a
Autor:
Gabriele Civiletto, Sukru Anil Dogan, Raffaele Cerutti, Gigliola Fagiolari, Maurizio Moggio, Costanza Lamperti, Cristiane Benincá, Carlo Viscomi, Massimo Zeviani
Publikováno v:
EMBO Molecular Medicine, Vol 10, Iss 11, Pp 1-15 (2018)
Abstract The mTOR inhibitor rapamycin ameliorates the clinical and biochemical phenotype of mouse, worm, and cellular models of mitochondrial disease, via an unclear mechanism. Here, we show that prolonged rapamycin treatment improved motor endurance
Externí odkaz:
https://doaj.org/article/4ceca83a73594e0b84ecb798f4bbcb33
Autor:
Alexandra Kukat, Sukru Anil Dogan, Daniel Edgar, Arnaud Mourier, Christoph Jacoby, Priyanka Maiti, Jan Mauer, Christina Becker, Katharina Senft, Rolf Wibom, Alexei P Kudin, Kjell Hultenby, Ulrich Flögel, Stephan Rosenkranz, Daniel Ricquier, Wolfram S Kunz, Aleksandra Trifunovic
Publikováno v:
PLoS Genetics, Vol 10, Iss 6, p e1004385 (2014)
Although mitochondrial dysfunction is often accompanied by excessive reactive oxygen species (ROS) production, we previously showed that an increase in random somatic mtDNA mutations does not result in increased oxidative stress. Normal levels of ROS
Externí odkaz:
https://doaj.org/article/874cce9099f1487993ef890ff62f85e0