Zobrazeno 1 - 10
of 46
pro vyhledávání: '"Ryoji Nishi"'
Autor:
Haruki Koike, Ryoji Nishi, Soma Furukawa, Naohiro Mouri, Yuki Fukami, Masahiro Iijima, Masahisa Katsuno
Publikováno v:
Allergology International, Vol 71, Iss 3, Pp 373-382 (2022)
Background: Although eosinophilic granulomatosis with polyangiitis (EGPA) has been considered as a single disease entity belonging to anti-neutrophil cytoplasmic antibody (ANCA)-associated vasculitis, several studies have suggested that in addition t
Externí odkaz:
https://doaj.org/article/ccab78674a7d4a7681e62a0f4a59e22d
Autor:
Kayo Takenaka, Takuya Takeichi, Ryoji Nishi, Masashi Suzuki, Haruki Koike, Masahisa Katsuno, Mariko Ogawa‐Momohara, Yoshinao Muro, Masashi Akiyama
Publikováno v:
Journal of Cutaneous Immunology and Allergy, Vol 4, Iss 2, Pp 34-36 (2021)
Abstract Systemic sclerosis (SSc) is a multi‐system autoimmune disease. Anti‐neutrophil cytoplasmic antibodies (ANCA) are autoantibodies directed against enzymes found within primary granules of neutrophils and lysosomes in monocytes. Although up
Externí odkaz:
https://doaj.org/article/33aaf62404b8437fbb91675f628deddf
Autor:
Haruki Koike, Ryoji Nishi, Satoru Yagi, Soma Furukawa, Yuki Fukami, Masahiro Iijima, Masahisa Katsuno
Publikováno v:
Advances in Therapy. 40:25-40
Eosinophilic granulomatosis with polyangiitis (EGPA), previously known as Churg-Strauss syndrome, is a systemic disorder characterized by asthma, eosinophilia, and vasculitis primarily affecting small vessels. Although this disease is classified as a
Publikováno v:
Muscle & Nerve. 66:175-182
The mechanism of complement-mediated neurological injury in vasculitic neuropathy associated with systemic lupus erythematosus (SLE) and rheumatoid arthritis (RA) is unknown. The current study aimed to investigate the local activation of the compleme
Autor:
Haruki Koike, Ryoji Nishi, Ken Ohyama, Saori Morozumi, Yuichi Kawagashira, Soma Furukawa, Naohiro Mouri, Yuki Fukami, Masahiro Iijima, Gen Sobue, Masahisa Katsuno
Publikováno v:
Neurology and Therapy. 11:21-38
Anti-neutrophil cytoplasmic antibody (ANCA)-associated vasculitis is a systemic disorder that frequently affects the peripheral nervous system and consists of three distinct conditions: microscopic polyangiitis (MPA), granulomatosis with polyangiitis
Autor:
Haruki Koike, Yuki Fukami, Masahiro Iijima, Ken Ohyama, Ryoji Nishi, Masahisa Katsuno, Gen Sobue
Publikováno v:
Neurology. 96:226-229
Eosinophilic granulomatosis with polyangiitis (EGPA) is one of the principal causes of vasculitic neuropathy.1 Recently, clinicopathologic differences among patients with EGPA based on their antineutrophil cytoplasmic antibody (ANCA) status have been
Autor:
Ryoji Nishi, Haruki Koike, Gen Sobue, Masahiro Iijima, Yuki Fukami, Masahisa Katsuno, Yuichi Kawagashira
Publikováno v:
Journal of Neurology, Neurosurgery & Psychiatry. 91:650-659
ObjectiveTo describe the pathological features of Guillain-Barré syndrome focusing on macrophage-associated myelin lesions.MethodsLongitudinal sections of sural nerve biopsy specimens from 11 patients with acute inflammatory demyelinating polyneurop
Autor:
Shohei Ikeda, Masahiro Iijima, Haruki Koike, Ryoji Nishi, Gen Sobue, Yuichi Kawagashira, Yuki Fukami, Masahisa Katsuno, Ken Ohyama
Publikováno v:
Neurology. 94:e1726-e1737
ObjectiveTo investigate the clinicopathologic features of eosinophilic granulomatosis with polyangiitis (EGPA)–associated neuropathy with a focus on the presence or absence of anti-neutrophil cytoplasmic antibodies (ANCAs).MethodsWe examined the cl
Autor:
Masahiro Iijima, Ryoji Nishi, Junichirou Hagita, Haruki Koike, Yuki Fukami, Hisayoshi Niwa, Masahisa Katsuno, Yuichi Kawagashira
Publikováno v:
Internal Medicine
We herein report the case of a 67-year-old man who presented with the acute onset of limb weakness. Brain magnetic resonance imaging revealed multiple abnormal-signal-intensity lesions. Steroids were administered, and the patient initially responded.
Autor:
Haruki Koike, Ryoji Nishi, Satoru Yagi, Soma Furukawa, Yuki Fukami, Masahiro Iijima, Masahisa Katsuno
Publikováno v:
Advances in Therapy. 40:718-718