Zobrazeno 1 - 10
of 73
pro vyhledávání: '"Rutberg, Mikael"'
Autor:
Chaudhari, Aditi, Håversen, Liliana, Mobini, Reza, Andersson, Linda, Ståhlman, Marcus, Lu, Emma, Rutberg, Mikael, Fogelstrand, Per, Ekroos, Kim, Mardinoglu, Adil, Levin, Malin, Perkins, Rosie, Borén, Jan
Publikováno v:
In BBA - Molecular and Cell Biology of Lipids November 2016 1861(11):1643-1651
Akademický článek
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Akademický článek
Tento výsledek nelze pro nepřihlášené uživatele zobrazit.
K zobrazení výsledku je třeba se přihlásit.
K zobrazení výsledku je třeba se přihlásit.
Autor:
Olofsson, Sven-Olof, Andersson, Linda, Håversen, Liliana, Olsson, Christina, Myhre, Susanna, Rutberg, Mikael, Mobini, Reza, Li, Lu, Lu, Emma, Borén, Jan, Boström, Pontus
Publikováno v:
In Prostaglandins, Leukotrienes and Essential Fatty Acids (PLEFA) November 2011 85(5):215-218
Autor:
Olofsson, Sven-Olof, Boström, Pontus, Andersson, Linda, Rutberg, Mikael, Perman, Jeanna, Borén, Jan
Publikováno v:
In BBA - Molecular and Cell Biology of Lipids 2009 1791(6):448-458
Autor:
Mobarak, Edouard, Håversen, Liliana, Manna, Moutusi, Rutberg, Mikael, Levin, Malin, Perkins, Rosie, Rog, Tomasz, Vattulainen, Ilpo, Borén, Jan
Publikováno v:
Scientific Reports, Vol 8, Iss 1, Pp 1-11 (2018)
Mobarak, E, Håversen, L, Manna, M, Rutberg, M, Levin, M, Perkins, R, Rog, T, Vattulainen, I & Borén, J 2018, ' Glucosylceramide modifies the LPS-induced inflammatory response in macrophages and the orientation of the LPS/TLR4 complex in silico ', Scientific Reports, vol. 8, 13600 . https://doi.org/10.1038/s41598-018-31926-0
Scientific Reports
Mobarak, E, Håversen, L, Manna, M, Rutberg, M, Levin, M, Perkins, R, Rog, T, Vattulainen, I & Borén, J 2018, ' Glucosylceramide modifies the LPS-induced inflammatory response in macrophages and the orientation of the LPS/TLR4 complex in silico ', Scientific Reports, vol. 8, 13600 . https://doi.org/10.1038/s41598-018-31926-0
Scientific Reports
Toll-like receptor 4 (TLR4) is activated by bacterial lipopolysaccharide (LPS), which drives the production of proinflammatory cytokines. Earlier studies have indicated that cholesterol- and glycosphingolipid-rich subregions of the plasma membrane (l
Autor:
Li, Lu, Stillemark-Billton, Pia, Beck, Caroline, Boström, Pontus, Andersson, Linda, Rutberg, Mikael, Ericsson, Johanna, Magnusson, Björn, Marchesan, Denis, Ljungberg, Anna, Borén, Jan, Olofsson, Sven-Olof
Publikováno v:
In Journal of Lipid Research January 2006 47(1):67-77
Autor:
Marchesan, Denis ‡, Rutberg, Mikael ‡, Andersson, Linda ‡, §, Asp, Lennart ‡, Larsson, Thomas §, Borén, Jan ‡, Johansson, Bengt R. ¶, Olofsson, Sven-Olof ‡, ∥
Publikováno v:
In Journal of Biological Chemistry 18 July 2003 278(29):27293-27300
Autor:
Håversen, Liliana, Sundelin, Jeanna Perman, Mardinoglu, Adil, Rutberg, Mikael, Ståhlman, Marcus, Wilhelmsson, Ulrika, Hultén, Lillemor Mattsson, Pekny, Milos, Fogelstrand, Per, Bentzon, Jacob Fog, Levin, Malin, Borén, Jan
Publikováno v:
Scientific Reports
Scientific Reports, Vol 8, Iss 1, Pp 1-13 (2018)
Repisalud
Instituto de Salud Carlos III (ISCIII)
Håversen, L, Sundelin, J P, Mardinoglu, A, Rutberg, M, Ståhlman, M, Wilhelmsson, U, Hultén, L M, Pekny, M, Fogelstrand, P, Bentzon, J F, Levin, M & Borén, J 2018, ' Vimentin deficiency in macrophages induces increased oxidative stress and vascular inflammation but attenuates atherosclerosis in mice ', Scientific Reports, vol. 8, no. 1, 16973 . https://doi.org/10.1038/s41598-018-34659-2
Scientific Reports, Vol 8, Iss 1, Pp 1-13 (2018)
Repisalud
Instituto de Salud Carlos III (ISCIII)
Håversen, L, Sundelin, J P, Mardinoglu, A, Rutberg, M, Ståhlman, M, Wilhelmsson, U, Hultén, L M, Pekny, M, Fogelstrand, P, Bentzon, J F, Levin, M & Borén, J 2018, ' Vimentin deficiency in macrophages induces increased oxidative stress and vascular inflammation but attenuates atherosclerosis in mice ', Scientific Reports, vol. 8, no. 1, 16973 . https://doi.org/10.1038/s41598-018-34659-2
The aim was to clarify the role of vimentin, an intermediate filament protein abundantly expressed in activated macrophages and foam cells, in macrophages during atherogenesis. Global gene expression, lipid uptake, ROS, and inflammation were analyzed