Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Ruiji Guo"'
Autor:
Samuel Knoedler, Sonja Broichhausen, Ruiji Guo, Ruoxuan Dai, Leonard Knoedler, Martin Kauke-Navarro, Fortunay Diatta, Bohdan Pomahac, Hans-Guenther Machens, Dongsheng Jiang, Yuval Rinkevich
Publikováno v:
Frontiers in Immunology, Vol 14 (2023)
Injuries to our skin trigger a cascade of spatially- and temporally-synchronized healing processes. During such endogenous wound repair, the role of fibroblasts is multifaceted, ranging from the activation and recruitment of innate immune cells throu
Externí odkaz:
https://doaj.org/article/7358472205ce401b9b620ced30e3b22b
Publikováno v:
Frontiers in Genetics, Vol 12 (2021)
Synpolydactyly (SPD) is a hereditary congenital limb malformation with distinct syndactyly designated as SPD1, SPD2, and SPD3. SPD1 is caused by mutations of HOXD13, which is a homeobox transcription factor crucial for limb development. More than 143
Externí odkaz:
https://doaj.org/article/cf549ef5b0154cc3a4caba7a29757c10
Publikováno v:
PLoS ONE, Vol 13, Iss 7, p e0200183 (2018)
BACKGROUND:Macrodactyly is a congenital malformation characterized by aggressive overgrowth of multiple tissues, including subcutaneous fat, nerves, and bones in digits or limbs. In type II macrodactyly, the peripheral nerve is enlarged; however, the
Externí odkaz:
https://doaj.org/article/8380091481d54592b2fd4abd868f7b16
Publikováno v:
Cold Spring Harbor perspectives in biology.
Wound healing disorders are a societal, clinical, and healthcare burden and understanding and treating them is a major challenge. A particularly important cell type in the wound healing processes is the fibroblast. Fibroblasts are not homogenous; how
Autor:
Knoedler, Samuel, Broichhausen, Sonja, Ruiji Guo, Ruoxuan Dai, Knoedler, Leonard, Kauke-Navarro, Martin, Diatta, Fortunay, Pomahac, Bohdan, Machens, Hans-Guenther, Dongsheng Jiang, Rinkevich, Yuval
Publikováno v:
Frontiers in Immunology; 2023, p1-16, 16p
Publikováno v:
Frontiers in Genetics, Vol 12 (2021)
Frontiers in Genetics
Frontiers in Genetics
Synpolydactyly (SPD) is a hereditary congenital limb malformation with distinct syndactyly designated as SPD1, SPD2, and SPD3. SPD1 is caused by mutations of HOXD13, which is a homeobox transcription factor crucial for limb development. More than 143
Publikováno v:
Annals of plastic surgery. 82(5)
GLI3 encodes a transcription factor in the sonic hedgehog signaling pathway, which is essential in regulating the human limb bud development, especially on the anteroposterior axis. Mutations in GLI3 have been confirmed to be associated with various