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Changes in the mitochondrial permeability transition pore in aging and age-associated diseases

Autor: Paradies G, Paradies V, Ruggiero FM, Petrosillo G.

Publikováno v: Mechanisms of ageing and development
134 (2013): 1–9.
info:cnr-pdr/source/autori:Paradies G, Paradies V, Ruggiero FM, Petrosillo G./titolo:Changes in the mitochondrial permeability transition pore in aging and age-associated diseases./doi:/rivista:Mechanisms of ageing and development (Print)/anno:2013/pagina_da:1/pagina_a:9/intervallo_pagine:1–9/volume:134

Aging is a biological process associated with impairment of mitochondrial bioenergetic function, increased oxidative stress, attenuated ability to respond to stresses and increased risk in contracting age-associated diseases. When mitochondria are su

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=cnr_________::8fd4230c94319d4f3ee4b17ba46929ec
https://publications.cnr.it/doc/271202

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Mitochondrial dysfunction in rat with nonalcoholic fatty liver Involvement of complex I, reactive oxygen species and cardiolipin

Autor: Petrosillo G, Portincasa P, Grattagliano I, Casanova G, Matera M, Ruggiero FM, Ferri D, Paradies G.

Publikováno v: Biochimica et biophysica acta. Bioenergetics 1767 (2007): 1260–1267.
info:cnr-pdr/source/autori:Petrosillo G; Portincasa P; Grattagliano I; Casanova G; Matera M; Ruggiero FM; Ferri D; Paradies G./titolo:Mitochondrial dysfunction in rat with nonalcoholic fatty liver Involvement of complex I, reactive oxygen species and cardiolipin/doi:/rivista:Biochimica et biophysica acta. Bioenergetics/anno:2007/pagina_da:1260/pagina_a:1267/intervallo_pagine:1260–1267/volume:1767

Mitochondrial dysfunction and oxidative stress play a central role in the pathophysiology of nonalcoholic fatty liver disease (NAFLD). This study aimed to elucidate the mechanism(s) responsible for mitochondrial dysfunction in nonalcoholic fatty live

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=cnr_________::02698a59a3601100b303280b191401e3
https://publications.cnr.it/doc/12838

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Decrease in mitochondrial complex I activity in ischemic/reperfused rat heart. Involvement of reactive oxygen species and cardiolipin

Autor: Paradies G, Petrosillo G, Pistolese M, Di Venosa N, Federici A, Ruggiero FM

Publikováno v: Circulation research 94 (2004): 53–59.
info:cnr-pdr/source/autori:Paradies G, Petrosillo G, Pistolese M, Di Venosa N, Federici A, Ruggiero FM/titolo:Decrease in mitochondrial complex I activity in ischemic%2Freperfused rat heart. Involvement of reactive oxygen species and cardiolipin/doi:/rivista:Circulation research/anno:2004/pagina_da:53/pagina_a:59/intervallo_pagine:53–59/volume:94

Reactive oxygen species (ROS) are considered an important factor in ischemia-reperfusion injury to cardiac myocytes. Mitochondrial respiration is an important source of ROS production and hence a potential contributor to cardiac reperfusion injury. I

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=cnr_________::196e0e4a6c027db277fa7a5f215a1f47
https://publications.cnr.it/doc/12768

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Decreased complex III activity in mitochondria isolated from rat heart subjected to ischemia and reperfusion: role of reactive oxygen species and cardiolipin

Autor: Petrosillo G, Ruggiero FM, Di Venosa N, Paradies G.

Publikováno v: The FASEB journal 17 (2003): 714–716.
info:cnr-pdr/source/autori:Petrosillo G, Ruggiero FM, Di Venosa N, Paradies G./titolo:Decreased complex III activity in mitochondria isolated from rat heart subjected to ischemia and reperfusion: role of reactive oxygen species and cardiolipin./doi:/rivista:The FASEB journal/anno:2003/pagina_da:714/pagina_a:716/intervallo_pagine:714–716/volume:17

Reactive oxygen species (ROS) are considered an important factor in ischemia-reperfusion injury to cardiac myocites. We have examined the effects of ischemia (30 min) and ischemia followed by reperfusion (15 min) of rat hearts on the activity of comp

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=cnr_________::c95871d2b77b8355cfdf780be2cefb57
http://www.cnr.it/prodotto/i/12695

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Role of reactive oxygen species and cardiolipin in the release of cytochrome c from mitochondria

Autor: Petrosillo G 1, Ruggiero FM 1, 2, Paradies G 1

Publikováno v: The FASEB journal 17 (2003): 2202–2208.
info:cnr-pdr/source/autori:Petrosillo G 1, Ruggiero FM 1, 2, Paradies G 1,2./titolo:Role of reactive oxygen species and cardiolipin in the release of cytochrome c from mitochondria./doi:/rivista:The FASEB journal/anno:2003/pagina_da:2202/pagina_a:2208/intervallo_pagine:2202–2208/volume:17

Several lines of evidence indicate that mitochondria-mediated reactive oxygen species (ROS) generation is a major source of oxidative stress in the cell. Release of cytochrome c from mitochondria is a central event in apoptosis induction and appears

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=cnr_________::d02cc32bc5b1f109261bda0f8f865ef8
https://publications.cnr.it/doc/12694

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