Zobrazeno 1 - 10
of 50
pro vyhledávání: '"Rou Ling Cho"'
Autor:
Sy-Jou Chen, Rou-Ling Cho, Skye Hsin-Hsien Yeh, Min-Chien Tsai, Yi-Ping Chuang, Chih-Feng Lien, Chuang-Hsin Chiu, Yi-Wei Yeh, Chin-Sheng Lin, Kuo-Hsing Ma
Publikováno v:
Lipids in Health and Disease, Vol 23, Iss 1, Pp 1-14 (2024)
Abstract Introduction Hypercholesterolemia is associated with increased inflammation and impaired serotonin neurotransmission, potentially contributing to depressive symptoms. However, the role of statins, particularly pitavastatin, in modulating ser
Externí odkaz:
https://doaj.org/article/f329d5b3fee247869baf7277017a95f5
Autor:
Ruey-Shyang Soong, Ravi K. Anchoori, Richard B. S. Roden, Rou-Ling Cho, Yi-Chan Chen, Sheng-Chieh Tseng, Yun-Li Huang, Po-Cheng Liao, Yu-Chiau Shyu
Publikováno v:
BMC Cancer, Vol 20, Iss 1, Pp 1-15 (2020)
Abstract Background According to GLOBOSCAN, hepatocellular carcinoma (HCC) claimed 782,000 lives in 2018. The tyrosine kinase inhibitor sofafenib is used to treat HCC, but new anticancer agents targeting different pathways are urgently needed to impr
Externí odkaz:
https://doaj.org/article/44a667359ad24cdaa8fc5e8ea4089343
Autor:
Chien-Chung Yang, Li-Der Hsiao, Chen-Yu Wang, Wei-Ning Lin, Ya-Fang Shih, Yi-Wen Chen, Rou-Ling Cho, Hui-Ching Tseng, Chuen-Mao Yang
Publikováno v:
Antioxidants, Vol 11, Iss 4, p 782 (2022)
Lung inflammation is a pivotal event in the pathogenesis of acute lung injury. Heme oxygenase-1 (HO-1) is a key antioxidant enzyme that could be induced by kaempferol (KPR) and exerts anti-inflammatory effects. However, the molecular mechanisms of KP
Externí odkaz:
https://doaj.org/article/636fba26b8de414d945f28abb87e73f9
Publikováno v:
Biomolecules, Vol 10, Iss 3, p 381 (2020)
Mevastatin (MVS) has been previously shown to induce heme oxygenase (HO)-1 expression through Nox/ROS-dependent PDGFRα/PI3K/Akt/Nrf2/ARE axis in human pulmonary alveolar epithelial cells (HPAEpiCs). However, alternative signaling pathways might invo
Externí odkaz:
https://doaj.org/article/a3d870fb0b2d4c9a92c6c49e1bc79557
Publikováno v:
Frontiers in Pharmacology, Vol 7 (2016)
Tumor necrosis factor-alpha (TNF-alpha) triggers activation of cytosolic phospholipase A2 (cPLA2) and then enhancing the synthesis of prostaglandin (PG) in inflammatory diseases. However, the detailed mechanisms of TNF-alpha induced cPLA2 expression
Externí odkaz:
https://doaj.org/article/89f562d975d145d99fc017d8255bfca0
Autor:
Ruey Shyang Soong, Po Cheng Liao, Yun Li Huang, Yu-Chiau Shyu, Rou Ling Cho, Yi Chan Chen, Sheng Chieh Tseng, Ravi K. Anchoori, Richard B.S. Roden
Publikováno v:
BMC Cancer, Vol 20, Iss 1, Pp 1-15 (2020)
BMC Cancer
BMC Cancer
Background According to GLOBOSCAN, hepatocellular carcinoma (HCC) claimed 782,000 lives in 2018. The tyrosine kinase inhibitor sofafenib is used to treat HCC, but new anticancer agents targeting different pathways are urgently needed to improve outco
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1132d2b000ebbd6a1a54bf1333098c3f
https://doi.org/10.21203/rs.2.18702/v2
https://doi.org/10.21203/rs.2.18702/v2
Publikováno v:
Biomolecules, Vol 10, Iss 3, p 381 (2020)
Biomolecules
Volume 10
Issue 3
Biomolecules
Volume 10
Issue 3
Mevastatin (MVS) has been previously shown to induce heme oxygenase (HO)-1 expression through Nox/ROS-dependent PDGFR&alpha
/PI3K/Akt/Nrf2/ARE axis in human pulmonary alveolar epithelial cells (HPAEpiCs). However, alternative signaling pathways
/PI3K/Akt/Nrf2/ARE axis in human pulmonary alveolar epithelial cells (HPAEpiCs). However, alternative signaling pathways
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1b690bebc7f2537dc05a46de17aba4f5
https://www.mdpi.com/2218-273X/10/3/381
https://www.mdpi.com/2218-273X/10/3/381
Autor:
Ruey-Shyang Soong, Anchoori, Ravi K., Roden, Richard B. S., Rou-Ling Cho, Yi-Chan Chen, Sheng-Chieh Tseng, Huang, Yun-Li, Po-Cheng Liao, Shyu, Yu-Chiau
Additional file 2: Figure S4. Original images to Fig. 1C. Figure S5. Original images to Fig. 1D. Figure S6. Original images to Fig. 2A. Figure S7. Original images to Fig. 6A. Figure S8. Original images to Fig. 6C. Figure S9. Original images to Fig. S
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7b93c8efbdc2fb9fc2a670ddc2986126
Autor:
Ruey-Shyang Soong, Anchoori, Ravi K., Roden, Richard B. S., Rou-Ling Cho, Yi-Chan Chen, Sheng-Chieh Tseng, Huang, Yun-Li, Po-Cheng Liao, Shyu, Yu-Chiau
Additional file 1: Figure S1. RA190 treatment HepG2 cell lines did not activate the autophagy pathway. Figure S2. Immunostaining of NF-κB and IκB after treating with RA190. Figure S3. RA190 treatment of HepG2 cells produced significant accumulation
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::317dbc03a6f914411cd7fe90feadd29f
Autor:
Chien-Chung Yang, Rou-Ling Cho, Li-Der Hsiao, Chuen-Mao Yang, Hui-Ching Tseng, Chih-Chung Lin
Publikováno v:
British Journal of Pharmacology. 175:3928-3946
Background and purpose Haem oxygenase-1 (HO-1) is induced by thiazolidinediones including rosiglitazone and exerts anti-inflammatory effects in various models. However, the molecular mechanisms underlying rosiglitazone-induced HO-1 expression remain
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::71bebac1f04301bee7350b7df5bc89b8
https://doi.org/10.1111/bph.14465
https://doi.org/10.1111/bph.14465